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This man started experiencing visual hallucinations at the age of 80. Visual hallucinations gradually became more frequent and occurred many times during the day. At the time of the visit, he repeatedly described seeing bilateral visual hallucinations with vivid details. The hallucinations reported by this patient were well formed, ranging from simple flashes or colored background to more complex with the appearance of common faces, objects and bodies of people, or landscapes. The hallucinations varied in size and color, and were binocular, covering the entire visual field. However, animations (i.e. scenes in motion) were only present in the right visual field. The visual hallucinations were always perceived as pleasant; they generally occurred with the eyes open and did not disappear when closing the eyes and were never accompanied by abnormal perception in any other sensory modality. The patient was fully aware of their unreal nature but he was not able to consciously control their occurrence or content. Based on his clinical history and the diagnostic exams he underwent, a diagnosis of CBS was made by the neurologist. Indeed, the patient fulfilled the four diagnostic criteria for CBS: (1) hallucinations must be complex, repetitive, and persistent; (2) awareness that the hallucinations are not real; (3) no additional delusions; and (4) absence of additional hallucinations in the other senses .
| 4.097656
| 0.96875
|
sec[1]/sec[0]/sec[0]/p[1]
|
en
| 0.999997
|
31318888
|
https://doi.org/10.1371/journal.pone.0219656
|
[
"hallucinations",
"visual",
"this",
"occurred",
"complex",
"field",
"eyes",
"their",
"additional",
"experiencing"
] |
[
{
"code": "MB27.2Z",
"title": "Hallucinations, unspecified"
},
{
"code": "MB27.2Y",
"title": "Other specified hallucinations"
},
{
"code": "6E61.0",
"title": "Secondary psychotic syndrome, with hallucinations"
},
{
"code": "MB27.27",
"title": "Visual hallucinations"
},
{
"code": "MB27.20",
"title": "Auditory hallucinations"
},
{
"code": "9E1Z",
"title": "Diseases of the visual system, unspecified"
},
{
"code": "MC1Y",
"title": "Other specified symptoms or signs involving the visual system"
},
{
"code": "9D9Z",
"title": "Vision impairment, unspecified"
},
{
"code": "9D90.2",
"title": "Moderate vision impairment"
},
{
"code": "QA00.6Z",
"title": "Examination of eyes or vision, unspecified"
}
] |
=== ICD-11 CODES FOUND ===
|
[] |
MB27.2Z
|
Hallucinations, unspecified
|
Patient 4 was a 41‐year‐old male with a history of schizophrenia which was currently treated with escitalopram, a selective serotonin reuptake inhibitor, not known to have any effect on phototoxicity. He presented himself three days after sudden onset of vision loss and central scotoma in both eyes. On the day before onset of symptoms, he participated in a street parade. He denied sun gazing or drug abuse, but reconstruction of the events was difficult. At first consultation, BVA was 20/123 in both eyes. Anterior segment examination was normal. Dilated ophthalmoscopic examination showed a clearly yellow area at the fovea with a reddish centre in both eyes ( Figure 5(a) ). B‐scan OCT demonstrated disruption of the RPE and the EZ and a wide hyperreflective band reaching from the outer nuclear layer to the outer plexiform layer ( Figure 5(b) ), similar to patient 1. En face OCT at the level of the EZ showed a hyporeflective defect surrounded by a hyperreflective ring ( Figure 5(c) ), which evolved into a central alteration with granular appearances and expanding in size before regressing to a clearly delineated EZ loss. BVA first dropped to 20/200 in both eyes 4 days after the incident, improved again to 20/100 in both eyes at 10 days, to 20/80 in both eyes at 2 months and was 20/50 in right and 20/60 in left eye at last follow-up. Initial presentation and evolution let presume a solar maculopathy.
| 4.011719
| 0.978516
|
sec[2]/sec[0]/p[3]
|
en
| 0.999997
|
31885977
|
https://doi.org/10.1155/2019/3849871
|
[
"both",
"eyes",
"which",
"onset",
"loss",
"clearly",
"hyperreflective",
"outer",
"layer",
"schizophrenia"
] |
[
{
"code": "LB99.6",
"title": "Acheiria"
},
{
"code": "MB51.Z",
"title": "Diplegia of upper extremities, unspecified"
},
{
"code": "LB9A.4",
"title": "Apodia"
},
{
"code": "LB51",
"title": "Anorchia or microorchidia"
},
{
"code": "9D90.2",
"title": "Moderate vision impairment"
},
{
"code": "9E1Z",
"title": "Diseases of the visual system, unspecified"
},
{
"code": "QF01.Y",
"title": "Other specified acquired absence of organs"
},
{
"code": "LA10.Y",
"title": "Other specified structural developmental anomalies of ocular globes"
},
{
"code": "LA10.0",
"title": "Microphthalmos"
},
{
"code": "NA06.8D",
"title": "Ocular laceration without prolapse or loss of intraocular tissue, unilateral"
}
] |
=== ICD-11 CODES FOUND ===
|
[] |
LB99.6
|
Acheiria
|
The steroid-sparing agent, hydroxychloroquine 200 mg QD was administered to treat SLE since the first pediatric rheumatic outpatient clinic follow-up. Over 2 months, moon face developed and her body weight increased from 55 to 66 kg, and another steroid-sparing agent azathioprine 50 mg/day was administered after 6 weeks of steroid treatment in parallel with the decreasing ESR and remission of the facial malar rash. The prednisolone was gradually decreased to a maintenance dose of 5 mg/day in 3 months. An 80% reduction in transaminase levels was achieved by 4 weeks of treatment, while the liver function test results were completely normalized in 2 months. The patient's condition remained stable during the last 3 years of outpatient clinic follow-up without relapse. No renal or central nervous system involvement of the SLE was noticed. However, after a 3-year course of treatment, remission of the AIH was not achieved (ANA titer, 1:80). The liver function test results remained in the normal range (AST, 14 U/L; ALT, 7 U/L). Other rheumatic laboratory workups showed a normal complete blood cell count, elevated ESR (40 mm/h), anti-ds DNA Ab (<40.5 WHO unit/mL), normal complement levels (C3 120.0 and C4 15.5 mg/dL). Abdominal sonography showed no evidence of liver fibrosis or cirrhosis. Her maintenance medication included prednisolone 5 mg QOD, azathioprine 50 mg QOD, and hydroxychloroquine 200 mg QOD.
| 4.007813
| 0.971191
|
sec[1]/p[4]
|
en
| 0.999996
|
31428592
|
https://doi.org/10.3389/fped.2019.00310
|
[
"steroid",
"liver",
"sparing",
"agent",
"hydroxychloroquine",
"rheumatic",
"outpatient",
"clinic",
"azathioprine",
"remission"
] |
[
{
"code": "6C4H.Y",
"title": "Other specified disorders due to use of non-psychoactive substances"
},
{
"code": "MA12.4",
"title": "Finding of steroid agent in blood"
},
{
"code": "6C4H.1Z",
"title": "Harmful pattern of use of non-psychoactive substances, unspecified"
},
{
"code": "MF99",
"title": "Elevated urine levels of drugs, medicaments and biological substances"
},
{
"code": "2F32.Y",
"title": "Other specified benign neoplasm of ovary"
},
{
"code": "DB9Z",
"title": "Diseases of liver, unspecified"
},
{
"code": "DB97.Z",
"title": "Inflammatory liver disease, unspecified"
},
{
"code": "DB99.7",
"title": "Hepatic failure without mention whether acute or chronic"
},
{
"code": "LB20.0Y",
"title": "Other specified structural developmental anomalies of liver"
},
{
"code": "LB20.0Z",
"title": "Structural developmental anomalies of liver, unspecified"
}
] |
=== ICD-11 CODES FOUND ===
|
[] |
6C4H.Y
|
Other specified disorders due to use of non-psychoactive substances
|
Regarding past medical history, radiologic, endoscopic, and pathologic data, the patient was initially diagnosed with a malignant nonepithelial lower rectal tumor of the unknown origin and staged as mrT3a mrN0 cM0. Being aware of primary colorectal sarcomas, their recurrence and progression patterns and a threatening rate of R1 and R2 resections, an institutional multidisciplinary board suggested curative surgery as a primary treatment option. Total mesorectal excision (TME) with complete mesocolic excision and central vascular ligation (CME/CVL) carried by an open approach was selected as proper extent of surgery. Standardized surgical technique in this case included complete left flexure mobilization, ligation of the inferior mesenteric artery at its origin near the aorta and inferior mesenteric vein just below the pancreatic tail with consecutive total mesorectal excision up to the level of the pelvic diaphragm . Accurate TME procedure had been exacerbated by severe fibrotic changes of surrounding pelvic tissues due to postoperative changes after iliac lymphadenectomy for cervical cancer and radiation-induced fibrosis as well. Regarding the significant fibrotic transformation of the anterior rectal wall with adjacent tissues, an intraoperative decision was made to avoid reconstruction aware of an estimated high risk of anastomotic leakage. Closure of the pelvic peritoneum was avoided as well.
| 4.039063
| 0.967285
|
sec[2]/p[0]
|
en
| 0.999998
|
31885968
|
https://doi.org/10.1155/2019/1610653
|
[
"excision",
"pelvic",
"regarding",
"rectal",
"origin",
"aware",
"total",
"mesorectal",
"complete",
"ligation"
] |
[
{
"code": "FC00.3",
"title": "Acquired deformity of pelvis"
},
{
"code": "GA34.Y",
"title": "Other specified female pelvic pain associated with genital organs or menstrual cycle"
},
{
"code": "LB30.7",
"title": "Ectopic or pelvic kidney"
},
{
"code": "MD82",
"title": "Intra-abdominal or pelvic swelling, mass or lump"
},
{
"code": "GA05.Z",
"title": "Female pelvic inflammatory diseases, unspecified"
},
{
"code": "QA15.1",
"title": "Counselling related to sexual behaviour and orientation or sexual relationships of the person"
},
{
"code": "QA15.2",
"title": "Counselling related to sexual behaviour and orientation or sexual relationships of third party"
},
{
"code": "QA15.0",
"title": "Counselling related to sexual attitudes"
},
{
"code": "DB30.4",
"title": "Stenosis of the rectum"
},
{
"code": "DB31.0&XA4KU2",
"title": "Rectal fistula"
}
] |
=== ICD-11 CODES FOUND ===
|
[] |
FC00.3
|
Acquired deformity of pelvis
|
A 54-year-old Peruvian man was suffering from constant fatigue, cough, dyspnea, and fever for a couple of months. In emergency service, a computed tomography (CT) scan revealed an anterosuperior mediastinal tumor (12.8 cm × 10 cm × 7 cm) and pericardial effusion . He underwent an exploratory sternotomy, where it was found that the tumor with a cerebroid aspect occupied two-thirds of the anterior mediastinum and infiltrated the pericardial cavity and the right pleura (T3N0M0, G1, stage IIIa). Surgical resection was not feasible. However, a pericardiocentesis (800 cc, hematic) and biopsy were performed. The pathology report described it as a thymic NET consistent with a typical carcinoid . The diagnosis was corroborated with immunohistochemistry markers (positivity of synaptophysin and cytokeratin; negativity of TTF1 and Ki-67 10%–15%). Given the rarity of this type of tumor, the patient was referred to the best-specialized cancer center in Peru (Instituto Nacional de Enfermedades Neoplásicas). A second CT scan was performed, revealing that the tumor was in intimate contact with great vessels and was infiltrating the SVC with 85% of its lumen obstructed. This obstruction corresponded with the symptoms that the patient suffered from the beginning (SVC syndrome). The patient underwent a second surgery, where again the resection was not possible. The pathology report confirmed the previous diagnosis.
| 3.927734
| 0.982422
|
sec[1]/p[0]
|
en
| 0.999997
|
32000435
|
https://doi.org/10.1097/MD.0000000000018990
|
[
"tumor",
"that",
"scan",
"pericardial",
"resection",
"pathology",
"this",
"peruvian",
"suffering",
"constant"
] |
[
{
"code": "2F9Z",
"title": "Neoplasms of unknown behaviour of unspecified site"
},
{
"code": "ME61",
"title": "Subcutaneous swelling, mass or lump of uncertain or unspecified nature"
},
{
"code": "2E6Z",
"title": "Carcinoma in situ of unspecified site"
},
{
"code": "2F91.1",
"title": "Neoplasms of unknown behaviour of trachea, bronchus or lung"
},
{
"code": "2F92",
"title": "Neoplasms of unknown behaviour of skin"
},
{
"code": "8A80.Z",
"title": "Migraine, unspecified"
},
{
"code": "QA76",
"title": "Medication or substance that is known to be an allergen without injury or harm"
},
{
"code": "PL13.6",
"title": "Medication or substance that is known to be an allergen, as mode of injury or harm"
},
{
"code": "9C40.A0",
"title": "Papilloedema"
},
{
"code": "PA6Z",
"title": "Unintentional fall from unspecified height"
}
] |
=== ICD-11 CODES FOUND ===
|
[] |
2F9Z
|
Neoplasms of unknown behaviour of unspecified site
|
His MRI showed an abnormal symmetrical signal in the bilateral basal ganglia, which was considered to be indicative of DEACMP . Of note, the bilateral cerebral peduncles also showed abnormal symmetrical signals . The patient’s EEG indicated that Fp1, Fp2, F4, F8 leads in the frontal lobe area had a mixture of wide sharp, sharp, and three-phase waves . Based on the above clinical manifestations and examination results, the patient was diagnosed with DEACMP. We recommend that the patient be treated with hyperbaric oxygen therapy (HBO). The relatives decided to return the patient to the local hospital to continue rehabilitation. At hospital discharge, the patient had blurred consciousness, indifferent expression, poor understanding, poor memory, poor orientation of characters and space, slow response, and low speech. His tongue was slightly to the right. Proximity muscle strength of right lower limb was grade 4+, and distal muscle strength was grade 3-. Fig. 1 A bilateral symmetrical anomaly signal of the basal ganglia was captured in fluid attenuated inversion recovery images ( a ). Bilateral cerebral peduncles showed symmetrical high signals in DWI ( b ), but low signals in ADC Fig. 2 The EEG indicated a diffuse distribution of Delta waves. The Fp1, Fp2, F4 in the frontal lobe area had a mixture of wide sharp, sharp, and three-phase waves. No clinical events occurred during the monitoring process
| 3.914063
| 0.979004
|
sec[1]/sec[0]/p[3]
|
en
| 0.999997
|
32183727
|
https://doi.org/10.1186/s12883-020-01677-5
|
[
"symmetrical",
"sharp",
"signals",
"waves",
"poor",
"signal",
"basal",
"ganglia",
"deacmp",
"cerebral"
] |
[
{
"code": "KA20.11",
"title": "Symmetrical intrauterine growth restriction"
},
{
"code": "EC23.Y",
"title": "Other specified genetic disorders of skin pigmentation"
},
{
"code": "EF02.1",
"title": "Subcutaneous lipomatosis"
},
{
"code": "KA20.00",
"title": "Small for gestational age, symmetrical"
},
{
"code": "EK5Y",
"title": "Other specified skin disorders provoked by external factors"
},
{
"code": "4A43.3",
"title": "Mixed connective tissue disease"
},
{
"code": "PA83.1",
"title": "Unintentionally cut or pierced by sharp glass"
},
{
"code": "PG9Z&XE4BU",
"title": "Contact with sharp object, undetermined intent"
},
{
"code": "PG9Z",
"title": "Cut or pierced by other or unspecified sharp object, undetermined intent"
},
{
"code": "PE3Z",
"title": "Assault by being cut or pierced by other or unspecified sharp object"
}
] |
=== ICD-11 CODES FOUND ===
|
[] |
KA20.11
|
Symmetrical intrauterine growth restriction
|
Air leak syndrome is the overarching diagnosis for one or more pathologic air collections present outside of the respiratory tract. Seven classic presentations in neonates include pulmonary interstitial emphysema (PIE), pneumothorax, pneumomediastinum, pneumopericardium, pneumoperitoneum, subcutaneous emphysema, and systemic air embolism. Air leak occurs in approximately 10% of neonates with MAS for many presumed reasons including ball-valve effect of the meconium, ventilation-perfusion mismatch, surfactant inactivation, and parenchymal injury. While pneumoperitoneum is pathognomonic of perforation of an abdominal viscus , it rarely can result from air leak (incidence unknown) and, in such cases, is referred to as benign pneumoperitoneum (BPPT). Management of pneumoperitoneum resulting from a perforated viscus versus BPPT is quite different, the former representing a surgical emergency and the latter allowing the potential for conservative, nonoperative management. In a ventilated neonate with pneumoperitoneum, existing clinical evidence is ambiguous at delineating which cases may be amenable to initial observation alone. For clinicians caring for ill neonates, increased awareness of BPPT as a clinical entity might circumvent unnecessary surgeries and their inherent complications. We report a case of a term neonate with meconium aspiration syndrome (MAS) and air leak syndrome who developed BPPT.
| 4.265625
| 0.902832
|
sec[0]/p[0]
|
en
| 0.999995
|
31956461
|
https://doi.org/10.1155/2019/4238601
|
[
"pneumoperitoneum",
"leak",
"bppt",
"neonates",
"emphysema",
"meconium",
"viscus",
"cases",
"neonate",
"overarching"
] |
[
{
"code": "DC51.Y",
"title": "Other specified disorders of peritoneum or retroperitoneum"
},
{
"code": "KB27.4",
"title": "Pneumoperitoneum, originating in the perinatal period, due to primary pulmonary air leak syndromes"
},
{
"code": "CB21.Z",
"title": "Pneumothorax, unspecified"
},
{
"code": "MF50.5",
"title": "Extravasation of urine"
},
{
"code": "NE81.3",
"title": "Postsurgical leak"
},
{
"code": "GB0Y",
"title": "Other specified diseases of the male genital system"
},
{
"code": "CB60",
"title": "Tracheostomy malfunction"
},
{
"code": "5A00.00",
"title": "Permanent congenital hypothyroidism with diffuse goitre"
},
{
"code": "KB45",
"title": "Neonatal hypertension"
},
{
"code": "KB2D",
"title": "Respiratory failure of newborn"
}
] |
=== ICD-11 CODES FOUND ===
|
[] |
DC51.Y
|
Other specified disorders of peritoneum or retroperitoneum
|
ECG showed anterior wall myocardial infarction ( Figure 1(a) ). The patient was immediately transferred to the cardiac catheterization laboratory and received successful emergency angioplasty/drug-eluting- (DE) stenting of the subtotally occluded left anterior descending artery ( Figure 1(b) ). Transthoracic echocardiography showed left ventricular hypertrophy, moderately reduced systolic left ventricular function (LVEF 40%) with anterior, septal, anteroseptal, inferior-apical, and apical hypo- and akinesia. The hemodynamically stable patient was monitored at the chest pain unit. CRP apheresis using the CRP adsorber (PentraSorb® CRP) within C-reactive Protein Apheresis in Acute Myocardial Infarction (CAMI-1) trial was performed 34 h and 58 h after the onset of symptoms. In each apheresis session, 6000 ml plasma was treated via peripheral venous access. Plasma CRP levels declined from 28.77 mg/l to 12.58 mg/l during the first apheresis session and from 24.17 mg/l to 11.55 mg/l during the second session, respectively (Figures 2(a) and 2(b) ). Figure 2 also shows cardiac enzyme progress over 72 h. Elevated creatinine kinase (CK), CK-MB, and troponin levels at admission documented acute STEMI. CRP levels, however, were normal at admission and, as a result of myocardial necrosis, increased with time . CRP apheresis efficiently counteracted acute phase CRP elevation and reduced peak CRP plasma levels.
| 4.082031
| 0.782227
|
sec[1]/p[1]
|
en
| 0.999996
|
30533227
|
https://doi.org/10.1155/2018/4767105
|
[
"apheresis",
"myocardial",
"session",
"plasma",
"infarction",
"cardiac",
"ventricular",
"apical",
"wall",
"immediately"
] |
[
{
"code": "QB99",
"title": "Apheresis"
},
{
"code": "BD1Z",
"title": "Heart failure, unspecified"
},
{
"code": "BA41.Z",
"title": "Acute myocardial infarction, unspecified"
},
{
"code": "BA52.Z",
"title": "Coronary atherosclerosis, unspecified site"
},
{
"code": "LA8Z",
"title": "Structural developmental anomaly of heart or great vessels, unspecified"
},
{
"code": "BA6Z",
"title": "Ischaemic heart diseases, unspecified"
},
{
"code": "QB9Y",
"title": "Other specified contact with health services for nonsurgical interventions not involving devices"
},
{
"code": "QB96",
"title": "Contact with health services for radiotherapy session"
},
{
"code": "QB97",
"title": "Contact with health services for chemotherapy session for neoplasm"
},
{
"code": "2A83.2",
"title": "Solitary plasmacytoma"
}
] |
=== ICD-11 CODES FOUND ===
|
[] |
QB99
|
Apheresis
|
Because the results of biochemical tests indicated renal damage, plain abdominal computed tomography (CT) was performed. An irregular tumor of approximately 130 mm × 120 mm × 80 mm in size was observed in the left upper quadrant, which corresponds to the transverse colon. No intestinal distention was observed on the oral side of the tumor. Moreover, the tumor was in close contact with the pancreatic body and tail, and the gastric corpus greater curvature, suggesting invasion into nearby organs. Also, mildly enlarged lymph nodes were observed at the periphery of the tumor . Fig. 1 Abdominal computed tomography imaging. A plain abdominal computed tomography (CT) image on admission shows an irregular tumor in the left upper quadrant corresponding to the transverse colon. The tumor was in close contact with the pancreatic body and tail, and the gastric corpus greater curvature. Mildly enlarged lymph nodes were observed at the periphery of the tumorous lesion (1 a and 1 b ). The second plain abdominal CT conducted 36 days after the first CT showed that the tumor had regressed spontaneously and the swelling of lymph nodes around the tumorous lesion had disappeared (1 c and 1 d ). CT conducted 48 days after the first visit revealed intestinal distention on the oral side of the narrowed area in the transverse colon, and the patient underwent decompression by transanal ileus tube insertion (1 e and 1 f )
| 3.849609
| 0.962402
|
sec[1]/p[2]
|
en
| 0.999997
|
31931725
|
https://doi.org/10.1186/s12876-020-1162-2
|
[
"tumor",
"abdominal",
"plain",
"computed",
"tomography",
"transverse",
"colon",
"lymph",
"nodes",
"irregular"
] |
[
{
"code": "2F9Z",
"title": "Neoplasms of unknown behaviour of unspecified site"
},
{
"code": "ME61",
"title": "Subcutaneous swelling, mass or lump of uncertain or unspecified nature"
},
{
"code": "2E6Z",
"title": "Carcinoma in situ of unspecified site"
},
{
"code": "2F91.1",
"title": "Neoplasms of unknown behaviour of trachea, bronchus or lung"
},
{
"code": "2F92",
"title": "Neoplasms of unknown behaviour of skin"
},
{
"code": "MD81.3",
"title": "Acute abdomen"
},
{
"code": "JA01.0",
"title": "Abdominal pregnancy"
},
{
"code": "ME04.Z",
"title": "Ascites, unspecified"
},
{
"code": "NB51.0&XA3KX0",
"title": "Laceration without foreign body of abdominal wall"
},
{
"code": "NB9Y",
"title": "Other specified injuries to the abdomen, lower back, lumbar spine or pelvis"
}
] |
=== ICD-11 CODES FOUND ===
|
[] |
2F9Z
|
Neoplasms of unknown behaviour of unspecified site
|
Her mother contacted a pediatrician and scheduled an appointment for further evaluation the next day. Six hours prior to the office visit the following day, the patient mentioned feeling faint while walking down the stairs at home, with no loss of consciousness, which quickly resolved without any interventions. The patient’s mother also complained that the patient had a low-grade fever two days prior to the initial visit to the ED and denied having a history of asthma. On review of systems, she continued to endorse a persistent, non-productive cough, chest pressure, and shortness of breath, and had developed a sore throat. All other systems were non-contributory. Vital signs, in the supine position, were as follows: temperature of 98.0o F, pulse rate of 82 beats per minute, respiratory rate of 16 breaths per minute, and blood pressure of 116/72 mmHg. S1/S2 heart sounds were present but without murmurs. Pulmonary examination demonstrated clear breath sounds bilaterally. Five minutes later in the sitting position, pulse rate was 84 beats per minute and blood pressure was 112/70 mmHg. The patient was started on empiric antibiotic azithromycin for the treatment of suspected atypical community-acquired pneumonia (CAP) and was also prescribed an albuterol inhaler for suspected bronchospasm caused by cough syncope. At that time, she had blood taken to test for TSH, T4, and Mycoplasma pneumoniae IgM Ab.
| 3.626953
| 0.984375
|
sec[1]/p[14]
|
en
| 0.999995
|
32089974
|
https://doi.org/10.7759/cureus.6669
|
[
"pressure",
"minute",
"blood",
"mother",
"visit",
"without",
"that",
"systems",
"cough",
"breath"
] |
[
{
"code": "EH90.Z",
"title": "Pressure ulcer of unspecified grade"
},
{
"code": "MB23.L",
"title": "Pressured speech"
},
{
"code": "MD30.Z",
"title": "Chest pain, unspecified"
},
{
"code": "CB22.Y",
"title": "Other specified diseases of mediastinum, not elsewhere classified"
},
{
"code": "BA2Z",
"title": "Hypotension, unspecified"
},
{
"code": "ED5Y",
"title": "Other specified disorders of epidermal keratinisation"
},
{
"code": "KD30.0",
"title": "Birth depression with 5 minute Apgar score 0-3"
},
{
"code": "KD30.1",
"title": "Birth depression with 5 minute Apgar score 4-6"
},
{
"code": "KB21.0",
"title": "Severe birth asphyxia"
},
{
"code": "KB21.1",
"title": "Mild and moderate birth asphyxia"
}
] |
=== ICD-11 CODES FOUND ===
|
[] |
EH90.Z
|
Pressure ulcer of unspecified grade
|
Multiple laboratory and imaging studies were completed. Urine mucopolysaccharide screening negative. Normal CBC, LFTs, chemistry, creatinine kinase, TSH, lactate and ammonia levels. Normal pyruvate, carnitine, plasma and urine amino acids. Normal ceruloplasmin and copper levels. Initial very long‐chain fatty acid analysis showed moderate elevation of C26 and C26/C22 ratio with minimal elevation in C24 and C22; however, repeat analysis was normal. Normal CSF neopterin, tetrahydrobiopterin, homovanillic acid, 3‐O‐methyldopa, and 5‐hydroxyindoleacetic acid with otherwise normal CSF cell counts, protein, and glucose. Negative spinocerebellar ataxia type 2 and 3 genetic testing, as well as negative PARK2 testing (early‐onset Parkinsonism). Dopa‐responsive dystonia evaluation reported variant of unknown significance that was likely benign in the TH sequencing variant: IVS12 + 9 C>T. Normal glutaryl‐coA dehydrogenase enzyme activity in fibroblasts. Microarray completed at 6 years old was negative. MRI at 1 year reported nonspecific white matter changes, a small arachnoid cyst in the posterior fossa, J‐shaped sella, and mild tonsillar ectopy. Repeat MRI with MR spectroscopy completed at 2 years of age showed nonspecific white matter changes without spectroscopic abnormality. CXR and skeletal surveys were normal. Multiple EEGs were completed and were normal. Hearing and vision screening were also normal.
| 3.914063
| 0.93457
|
sec[1]/p[2]
|
en
| 0.999996
|
30455893
|
https://doi.org/10.1002/ccr3.1671
|
[
"acid",
"multiple",
"urine",
"screening",
"elevation",
"repeat",
"testing",
"variant",
"nonspecific",
"white"
] |
[
{
"code": "5C73.Z",
"title": "Acidosis, unspecified"
},
{
"code": "DA22.Z",
"title": "Gastro-oesophageal reflux disease, unspecified"
},
{
"code": "DA41.2",
"title": "Acid hypersecretion"
},
{
"code": "5B5C.Z",
"title": "Vitamin B3 deficiency, unspecified"
},
{
"code": "5C64.3",
"title": "Disorders of phosphorus metabolism or phosphatases"
},
{
"code": "6B64",
"title": "Dissociative identity disorder"
},
{
"code": "JA80.Z",
"title": "Maternal care related to unspecified multiple gestation"
},
{
"code": "QA46.Z",
"title": "Outcome of delivery, unspecified"
},
{
"code": "8A40.Z",
"title": "Multiple sclerosis, unspecified"
},
{
"code": "ND31",
"title": "Open wounds involving multiple body regions"
}
] |
=== ICD-11 CODES FOUND ===
|
[] |
5C73.Z
|
Acidosis, unspecified
|
A 53-year-old African-American woman with ESRD was transferred from dialysis clinic to the emergency room (ER) for evaluation of non-radiating and dull epigastric pain for two weeks associated with fever and chills during hemodialysis (HD). Three months ago, she was hospitalized and treated for Streptococcus pneumoniae and Enterobacter cloacae bacteremia. A year ago she was treated for S. maltophilia bacteremia secondary to an infected dialysis catheter. Past medical history was also significant for hypertension, atherosclerotic vascular disease pending elective coronary artery bypass graft (CABG) surgery, and sudden cardiac arrest followed by ICD placement. With the current presentation, both blood and catheter cultures obtained at the dialysis clinic were positive for S. maltophilia , prompting her subsequent arrival to the emergency room. She presented with a continuation of fever and chills, as well as tachycardia and episodic hypotension. She was noted to have mild epigastric tenderness. There was no surrounding erythema, discharge, or tenderness noted around the tunneled dialysis catheter on the right anterior chest. Initial workup showed elevated troponin and procalcitonin. Chest X-ray findings were suggestive for left lower lobe pneumonia. Within the ER, the patient's hypotension resolved with fluid resuscitation and intravenous levofloxacin therapy was started with blood cultures drawn.
| 3.783203
| 0.983887
|
sec[1]/p[0]
|
en
| 0.999998
|
31065471
|
https://doi.org/10.7759/cureus.4165
|
[
"dialysis",
"catheter",
"clinic",
"emergency",
"room",
"epigastric",
"fever",
"chills",
"treated",
"bacteremia"
] |
[
{
"code": "QB94.Z",
"title": "Care involving dialysis, unspecified"
},
{
"code": "QB94.1",
"title": "Care involving extracorporeal dialysis"
},
{
"code": "9B73.0",
"title": "Retinal detachment with retinal break"
},
{
"code": "QB94.2",
"title": "Care involving peritoneal dialysis"
},
{
"code": "QB94.Y",
"title": "Care involving other specified dialysis"
},
{
"code": "QB62.Z",
"title": "Attention to artificial openings, unspecified"
},
{
"code": "QB30.5",
"title": "Fitting or adjustment of urinary device"
},
{
"code": "PK93.10",
"title": "Gastroenterology or urology devices associated with injury or harm, urinary catheter"
},
{
"code": "PK90.1",
"title": "Anaesthesiology devices associated with injury or harm, therapeutic, nonsurgical or rehabilitative devices"
},
{
"code": "PK91.2Y",
"title": "Other specified cardiovascular devices associated with injury or harm, prosthetic or other implants, materials or accessory devices"
}
] |
=== ICD-11 CODES FOUND ===
|
[] |
QB94.Z
|
Care involving dialysis, unspecified
|
The patient underwent endoscope-assisted evacuation of ASDH under general anesthesia. We selected the site for craniotomy at the thickest part of the hematoma using neuronavigation. Then, a straight skin incision of 5 cm was made, and a 4-cm-diameter small parietal craniotomy was performed. The hematoma was jelly-like and had partially formed a capsule. The hematoma was aspirated with a suction tube while being observed with a 2.7-mm 0° and 30° rigid endoscope . The rigid endoscope was held in the left hand, the suction cannula was held in the right hand, and the suction cannula was exchanged for bipolar forceps, as needed. The hematoma was removed using a suction cannula under the guidance of a rigid endoscope. When bleeding from a vessel on the brain surface was observed, a suction cannula was placed at the bleeding point, and coagulation was performed using monopolar electrocoagulation with a suction cannula. The bridging vein, which we identified as the source of bleeding, was coagulated using bipolar forceps. It was covered with absorbable Fibrillar™ hemostat (Surgicel®; Ethicon, Tokyo, Japan) for bleeding points. Finally, the subdural space was irrigated with artificial cerebrospinal fluid(Artcereb, Otsuka Pharmaceutical Factory, Inc., Tokushima, Japan). The dura mater was tightly closed and the bone was repositioned and stabilized using a fixture device, followed by closure of the scalp.
| 3.914063
| 0.95752
|
sec[1]/p[2]
|
en
| 0.999996
|
39099992
|
https://doi.org/10.7759/cureus.63817
|
[
"suction",
"using",
"cannula",
"endoscope",
"hematoma",
"bleeding",
"rigid",
"craniotomy",
"held",
"hand"
] |
[
{
"code": "QE11.Z",
"title": "Hazardous drug use, unspecified"
},
{
"code": "6C4Z",
"title": "Disorders due to substance use, unspecified"
},
{
"code": "QE11.3",
"title": "Hazardous use of cocaine"
},
{
"code": "QE11.2",
"title": "Hazardous use of sedatives, hypnotics or anxiolytics"
},
{
"code": "QE11.1",
"title": "Hazardous use of cannabis"
},
{
"code": "DA25.3Y/PK80.32",
"title": "Oesophageal ulcer due to endoscopic procedures"
},
{
"code": "DA42.8Z/PK80.32",
"title": "Gastritis due to endoscopic procedures"
},
{
"code": "DA51.5Y/PK80.32",
"title": "Duodenitis due to endoscopic procedures"
},
{
"code": "PL11.4",
"title": "Failure of sterile precautions, as mode of injury or harm"
},
{
"code": "PK80.92",
"title": "Vascular procedure associated with injury or harm, endoscopic approach"
}
] |
=== ICD-11 CODES FOUND ===
|
[] |
QE11.Z
|
Hazardous drug use, unspecified
|
Results: Twelve children (boys=7) received anakinra, dose 2-6 mg/kg, max 300mg/ day. All had received IVIG and pulse methylprednisolone (IVMP) 30 mg/kg/24h, 3-6 doses. Eight were admitted to ICU. In 5/8 anakinra was initiated concomitantly with IVIG and IVMP due to rapid deterioration of cardiovascular function, necessitating support with inotropes; in 3/8 due to non response to IVIG and IVMP. Three infants (all boys, aged 5-7 months) received anakinra for 6 months due to coronary involvement, 2 with Coronary Artery Aneurysms with improvement in a 1-month period, one with medium CA dilation with complete recovery. All other children were aged 5 to 14 years; 1 received anakinra for MAS resistant to 6 IVMP pulses, 8 for cardiovascular collapse. Median time to response in maximum anakinra dose was 2 days. All children had gastrointestinal involvement and four gallbladder edema; 2 developed pancreatitis that resolved after one month of treatment. In 7/9 older children duration of treatment with anakinra was 2 months and of steroids 1 month. A 13-year-old girl and an 11-year-old girl received anakinra for 6 months due to recalcitrant pericarditis and fever with hyperinflammation recurrence after URI, respectively. Except for the 2 infants with CAA all children recovered completely without sequelae. Cardiac MRI was performed 6 months after initial hospitalization and revealed normal cardiac function.
| 4.039063
| 0.63623
|
sec[0]/sec[242]/sec[0]/sec[0]/sec[0]/sec[0]/p[4]
|
en
| 0.999994
|
39252016
|
https://doi.org/10.1186/s12969-024-01004-z
|
[
"anakinra",
"children",
"ivmp",
"ivig",
"boys",
"cardiovascular",
"function",
"response",
"infants",
"coronary"
] |
[
{
"code": "5B51&XS25",
"title": "Marasmus in children"
},
{
"code": "5B7Z",
"title": "Unspecified undernutrition"
},
{
"code": "5B51",
"title": "Wasting in infants, children or adolescents"
},
{
"code": "5B53",
"title": "Stunting in infants, children or adolescents"
},
{
"code": "DD93.Y",
"title": "Other functional digestive disorders of infants, neonates or toddlers"
},
{
"code": "BE2Y",
"title": "Other specified diseases of the circulatory system"
},
{
"code": "BE2Z",
"title": "Diseases of the circulatory system, unspecified"
},
{
"code": "BA00.Z",
"title": "Essential hypertension, unspecified"
},
{
"code": "BD1Z",
"title": "Heart failure, unspecified"
},
{
"code": "5C56.0Y",
"title": "Other specified sphingolipidosis"
}
] |
=== ICD-11 CODES FOUND ===
|
[] |
5B51&XS25
|
Marasmus in children
|
• Ventilator-associated pneumonia (VAP): Carbapenem-resistant Acinetobacter baumannii (CRAB) ( Table 2 ) was isolated from the Bronchoalveolar lavage (BAL) fluid. Meropenem was discontinued, and sulbactam was added to the Polymyxin B regimen. Subsequently, the patient underwent a tracheostomy on day 7. Fig. 1 a showing bilateral infiltrates during the initiation of treatment. With aggressive respiratory care and prone positioning, the patient gradually improved over the following days. Table 2 Antibiotic susceptibility of the Acinetobacter baumannii complex isolate recovered from this patient. Table 2 Antibiotics Interpretation MIC (mu/ml) Tigecycline Sensitive NA Ceftriaxone/Sulbactam/EDTA Sensitive NA Colistin Intermediate 1 Piperacillin/Tazobactam Resistant ≥ 128 Ceftazidime Resistant ≥ 64 Cefoperazone/Sulbactam Resistant ≥ 64 Cefepime Resistant ≥ 32 Aztreonam Resistant ≥ 64 Imipenem Resistant ≥ 16 Meropenem Resistant ≥ 16 Amikacin Resistant ≥ 64 Gentamicin Resistant ≥ 16 Ciprofloxacin Resistant ≥ 4 Levofloxacin Resistant ≥ 8 Minocycline Resistant 16 Trimethoprim/Sulfamethoxazole Resistant ≥ 320 Ampicillin/Sulbactam Resistant NA Method: Conventional Aerobic Culture. Semiquantitative/standard Loop. Susceptibility by Disc Diffusion/Automated MIC. Fig. 1 a) Chest X ray showing bilateral infiltrates (day 7 after admission); b) Resolution of pneumonia after treatment (day 21 of admission). Fig. 1
| 3.947266
| 0.93457
|
sec[3]/sec[4]/p[0]
|
en
| 0.999994
|
40115001
|
https://doi.org/10.1016/j.toxrep.2025.101968
|
[
"resistant",
"sulbactam",
"pneumonia",
"acinetobacter",
"baumannii",
"meropenem",
"infiltrates",
"susceptibility",
"sensitive",
"ventilator"
] |
[
{
"code": "MG55.0",
"title": "Artemisinin resistant Plasmodium falciparum"
},
{
"code": "5A74.Y",
"title": "Other specified adrenocortical insufficiency"
},
{
"code": "5A44",
"title": "Insulin-resistance syndromes"
},
{
"code": "LD2A.4",
"title": "46,XY disorder of sex development due to androgen resistance"
},
{
"code": "LB45.1",
"title": "46,XX gonadal dysgenesis"
},
{
"code": "CA40.Z",
"title": "Pneumonia, organism unspecified"
},
{
"code": "CA40.Y",
"title": "Other specified pneumonia"
},
{
"code": "KB24",
"title": "Congenital pneumonia"
},
{
"code": "CA40.1Z",
"title": "Viral pneumonia, unspecified"
},
{
"code": "CA40.0Z",
"title": "Bacterial pneumonia, unspecified"
}
] |
=== ICD-11 CODES FOUND ===
|
[] |
MG55.0
|
Artemisinin resistant Plasmodium falciparum
|
On the morning of postoperative day one, the patient’s blood pressure was 133/65 mmHg, and magnesium sulfate was discontinued 24 hours following delivery. However, labetalol 200 mg two times a day was continued as blood pressures ranged from 114-155/55-86 bpm throughout the day. At this point, she had received a total amount of 3,340 mL intravenous fluids, which were also discontinued at this time. Her intake and output of fluids are shown in Table 4 . The patient reported dyspnea with nasal congestion and coughing that improved with sitting up. Inspiratory stridor was heard, but the lungs were clear to auscultation. She had 1+ pitting edema of the lower extremities. On 4 L/min of supplemental oxygen via nasal cannula, oxygen saturations were 89-90%. By the late afternoon, her dyspnea was worsening, and she was requiring up to 10 L/min of supplemental oxygen. At this time, wet lung sounds were heard on auscultation. Labs showed increased leukocytosis but she remained afebrile (Table 2 ). Testing for COVID-19, influenza, and respiratory syncytial virus were negative. A computed tomography angiography (CTA), as shown in Figure 1 , was ordered and showed pulmonary edema with possible pneumonia and pulmonary hypertension. Intravenous furosemide was started, and her urine output was monitored. Other interventions to improve breathing included pulmonary hygiene and the use of an incentive spirometer.
| 3.697266
| 0.980469
|
sec[1]/p[3]
|
en
| 0.999996
|
39224732
|
https://doi.org/10.7759/cureus.66065
|
[
"this",
"oxygen",
"pulmonary",
"blood",
"discontinued",
"intravenous",
"fluids",
"time",
"output",
"shown"
] |
[
{
"code": "4A01.03",
"title": "Transient hypogammaglobulinaemia of infancy"
},
{
"code": "MD11.1",
"title": "Asphyxia"
},
{
"code": "NE60",
"title": "Harmful effects of drugs, medicaments or biological substances, not elsewhere classified"
},
{
"code": "3A51.7",
"title": "High affinity haemoglobin"
},
{
"code": "NF05",
"title": "Asphyxiation"
},
{
"code": "PB08",
"title": "Unintentional threat to breathing from low oxygen environment"
},
{
"code": "CB40.Y",
"title": "Other specified diseases of the respiratory system"
},
{
"code": "LA75.1",
"title": "Agenesis of lung"
},
{
"code": "CA40.Z",
"title": "Pneumonia, organism unspecified"
},
{
"code": "CB41",
"title": "Respiratory failure"
}
] |
=== ICD-11 CODES FOUND ===
|
[] |
4A01.03
|
Transient hypogammaglobulinaemia of infancy
|
A 52-year-old female with a BMI of 21.7 kg/m 2 and ASA II classification had a thoracic epidural placed at the thoracic 10-11 level immediately after her explorative laparotomy with a total abdominal hysterectomy and bilateral salpingo-oophorectomy surgery for pain control. She had a past medical history of uterine cancer, idiopathic thrombocytopenic purpura, and anxiety. She was allergic to penicillin. She was on a patient-controlled epidural analgesia with 0.06% bupivacaine, oral acetaminophen, and intravenous hydromorphone for pain control. On examination, her vital signs and laboratory values were within normal limits. On the first and second days after surgery, she had inadequate pain control, and during the examination, reported an inconsistent loss of sensations only over the left thoracoabdominal region, which did not improve despite all the readjustment maneuvers. Hence, an ultrasound evaluation of the catheter was done with the patient in a sitting position and along the parasagittal oblique view, which revealed that the catheter tip was not in the epidural space . It was subsequently replaced at the thoracic eight-nine level with satisfactory blockade and pain control. It was used for the next couple of days with the previous regimen until the patient was able to transition to oral acetaminophen and oxycodone. She was later discharged home the day after the catheter was discontinued.
| 3.748047
| 0.98291
|
sec[1]/p[7]
|
en
| 0.999996
|
39564044
|
https://doi.org/10.7759/cureus.71916
|
[
"pain",
"control",
"thoracic",
"epidural",
"catheter",
"oral",
"acetaminophen",
"which",
"classification",
"immediately"
] |
[
{
"code": "MG3Z",
"title": "Pain, unspecified"
},
{
"code": "8E43.Z",
"title": "Pain disorders, unspecified"
},
{
"code": "MG31.Z",
"title": "Acute pain, unspecified"
},
{
"code": "MG30.Z",
"title": "Chronic pain, unspecified"
},
{
"code": "FB56.2",
"title": "Myalgia"
},
{
"code": "6C01.Z",
"title": "Encopresis, unspecified"
},
{
"code": "6C00.Z",
"title": "Enuresis, unspecified"
},
{
"code": "MF50.2Z",
"title": "Urinary incontinence, unspecified"
},
{
"code": "5A14",
"title": "Diabetes mellitus, type unspecified"
},
{
"code": "6C0Z",
"title": "Elimination disorders, unspecified"
}
] |
=== ICD-11 CODES FOUND ===
|
[] |
MG3Z
|
Pain, unspecified
|
The first treatment cycle (administered activity: 7.4GBq) was well-tolerated with only decreased appetite, mild nausea, and fatigue. Following the first cycle, his PSA value slightly increased to 48.23 ng/mL. Six weeks later, he presented to the emergency department (ED) with increased nausea and vomiting. During admission, his symptoms were successfully managed with supportive treatment, as laboratory results and an ultrasound showed no abnormalities. He received a second cycle (administered activity: 7.4GBq) without any symptoms. Post-therapy SPECT scan revealed no new metastases or progression in existing lesions. His PSA value was 61.63 ng/mL after the second cycle. Three weeks later, the patient returned to the ED with symptoms of vomiting and back pain. In the patient’s history, there was no record of previous pancreatitis, gallstones, alcohol intake, high blood lipid levels, or other identifiable risk factors for pancreatitis. Blood tests revealed slightly elevated bilirubin and liver enzyme levels. An outside CT scan identified a mass-like lesion adjacent to the pancreatic head, previously noted as PSMA-avid lymph nodes on pre-therapy [ 68 Ga] Ga-PSMA-11 PET/CT and contrast-enhanced CT imaging, which was causing biliary duct obstruction and ductal dilation. The obstruction was resolved with an ERCP procedure and common bile duct (CBD) stent placement, leading to symptomatic improvement.
| 3.916016
| 0.970215
|
sec[1]/p[2]
|
en
| 0.999998
|
39435291
|
https://doi.org/10.3389/fonc.2024.1442293
|
[
"cycle",
"activity",
"nausea",
"slightly",
"vomiting",
"scan",
"pancreatitis",
"blood",
"psma",
"duct"
] |
[
{
"code": "6A80.5",
"title": "Rapid cycling"
},
{
"code": "PA03",
"title": "Unintentional land transport traffic event injuring a motor cyclist"
},
{
"code": "5C50.AZ",
"title": "Disorders of urea cycle metabolism, unspecified"
},
{
"code": "PA23",
"title": "Unintentional land transport event unknown whether traffic or nontraffic injuring a motor cyclist"
},
{
"code": "PA02",
"title": "Unintentional land transport traffic event injuring a pedal cyclist"
},
{
"code": "QF2Z",
"title": "Difficulty or need for assistance with unspecified activity"
},
{
"code": "6A05.Z",
"title": "Attention deficit hyperactivity disorder, presentation unspecified"
},
{
"code": "QF23",
"title": "Difficulty or need for assistance with mobility"
},
{
"code": "QF28",
"title": "Difficulty or need for assistance with work activities"
},
{
"code": "CA40.Z",
"title": "Pneumonia, organism unspecified"
}
] |
=== ICD-11 CODES FOUND ===
|
[] |
6A80.5
|
Rapid cycling
|
Here, we present 2 siblings born to healthy, consanguineous first-cousin parents from Sudan . In addition, there was one spontaneous termination of pregnancy for which information was not available. The proband (P1) is a male who presented with ataxia and severe global developmental delay. The pregnancy and delivery history were uneventful. At 3 years of age, he was hypotonic and areflexic, and lacked head control. He was unable to crawl or speak. By 8 years of age, he had dysarthria, nystagmus, and dysmetria, with greater interest in his surroundings, and ambulation with unilateral support. Hearing and vision remained intact. Further examination revealed microcephaly, spasticity, and hyperreflexia of both upper and lower limbs, in addition to feet deformity. Brain MRI showed a mildly thinned corpus callosum and mildly dilated lateral ventricles , but the cerebellum was intact. The diffusion-weighted imaging (DWI) sequences showed bilateral symmetric hyperintensities in the posterior limbs of the internal capsules . His younger sister (P2) presented also with delayed milestones. At 4 years old, she had dysarthria, and she could walk with support. Microcephaly, nystagmus, and spasticity of upper and lower limbs were present, but there was no ataxia. MRI of the brain showed a thinned corpus callosum and bilateral symmetric DWI hyperintense signals in the posterior limbs of the internal capsules .
| 4.03125
| 0.963379
|
sec[2]/sec[0]/p[0]
|
en
| 0.999996
|
39066872
|
https://doi.org/10.1007/s10048-024-00773-9
|
[
"limbs",
"present",
"addition",
"pregnancy",
"ataxia",
"dysarthria",
"nystagmus",
"support",
"intact",
"microcephaly"
] |
[
{
"code": "ND56.1",
"title": "Open wound of unspecified body region"
},
{
"code": "LB9Z",
"title": "Structural developmental anomalies of the skeleton, unspecified"
},
{
"code": "FB56.6",
"title": "Other specified soft tissue disorders"
},
{
"code": "5B51&XS25",
"title": "Severe wasting in infants, children or adolescents"
},
{
"code": "ND55",
"title": "Other injuries of leg, level unspecified"
},
{
"code": "JA82.2",
"title": "Maternal care for transverse or oblique lie"
},
{
"code": "JA82.1",
"title": "Maternal care for breech presentation"
},
{
"code": "JA82.Z",
"title": "Maternal care for malpresentation of fetus, unspecified"
},
{
"code": "JA82.6",
"title": "Maternal care for compound presentation"
},
{
"code": "JA82.5",
"title": "Maternal care for multiple gestation with malpresentation of one fetus or more"
}
] |
=== ICD-11 CODES FOUND ===
|
[] |
ND56.1
|
Open wound of unspecified body region
|
His serum creatine kinase level was significantly elevated at 17705 U/L (normal: 39–192 U/L). Other laboratory results were as follows: troponin I 0.299 ug/L (normal: 0–0.06 ug/L), lactate dehydrogenase 650 U/L (normal: 120–246 U/L), alanine aminotransferase 160 U/L (normal: <49 U/L), aspartate aminotransferase 297 U/L (normal: <49 U/L). Blood cell counts, thyroid function, parathyroid hormone, renal function, bone density, and spinal X-ray yielded no abnormal results. The electrocardiogram showed sinus arrhythmia and rightward deviation of the electrical axis. The echocardiography demonstrated a normal heart function and structure. The magnetic resonance imaging of the thigh muscles of both lower limbs suggested partial fatty infiltration of the gluteus maximus muscle bilaterally , with no significant fatty replacement of his bilateral thigh muscle. Simultaneously, we communicated with the parents of the patient and recommended that the child undergo a comprehensive magnetic resonance imaging of the brain. However, the parents opted to refuse due to financial constraints and the child’s lack of cooperation, which would necessitate the use of sedatives. Therefore, the child’s clinical symptoms, physical examination, magnetic resonance imaging findings of the thigh muscles of both lower limbs, and the markedly increased creatine kinase levels together indicated to us that the boy might have BMD.
| 3.984375
| 0.964844
|
sec[1]/sec[2]/p[0]
|
en
| 0.999999
|
39659883
|
https://doi.org/10.3389/fnins.2024.1459582
|
[
"function",
"magnetic",
"resonance",
"imaging",
"thigh",
"creatine",
"kinase",
"aminotransferase",
"muscles",
"both"
] |
[
{
"code": "DD91.Z",
"title": "Irritable bowel syndrome or functional bowel disorders, unspecified"
},
{
"code": "9E1Z",
"title": "Diseases of the visual system, unspecified"
},
{
"code": "6B60.Z",
"title": "Dissociative neurological symptom disorder, with unspecified symptoms"
},
{
"code": "6B60.8Y",
"title": "Dissociative neurological symptom disorder, with other specified movement disturbance"
},
{
"code": "6B60.3",
"title": "Dissociative neurological symptom disorder, with other sensory disturbance"
},
{
"code": "NA06.80",
"title": "Retained intraocular magnetic foreign body, unilateral"
},
{
"code": "NA06.80&XA03X9",
"title": "Retained magnetic foreign body in ciliary body, unilateral"
},
{
"code": "NA06.80&XA4HU2",
"title": "Retained magnetic foreign body in vitreous, unilateral"
},
{
"code": "NA06.80&XA3GW7",
"title": "Retained magnetic foreign body in iris, unilateral"
},
{
"code": "NA06.80&XA13U9",
"title": "Retained magnetic foreign body in lens, unilateral"
}
] |
=== ICD-11 CODES FOUND ===
|
[] |
DD91.Z
|
Irritable bowel syndrome or functional bowel disorders, unspecified
|
This case shows a possible association between an incomplete double ureter, arching testicular arteries and an accessory testicular artery. To our knowledge, this report is the first to identify and describe the coexistence of these anomalies and their possible clinical implications. Both double ureter and these arterial anomalies can cause urological symptoms and therefore must be distinguished. Furthermore, arching testicular arteries may cause nutcracker syndrome and should be especially considered as a possible cause when work-up reveals no compression by the SMA. This possible etiology of nutcracker syndrome has been suggested in at least several articles but has not been studied in depth or proven to exist. This case report highlights the possible association between three examples of CAKUT and therefore, unlike other articles, proposes that one look twice after finding an anomaly in order to detect other variations and correctly identify the root cause of the patient’s symptoms. Our report contributes to the limited literature and highlights the importance of reporting these anomalies when encountered during autopsies or pedagogical cadaveric dissection. A comprehensive understanding and identification of variations in testicular vessels and renal structures, along with considering potentially associated complications, are essential for accurate diagnosis and safe surgical interventions.
| 4.113281
| 0.945313
|
sec[3]/p[0]
|
en
| 0.999997
|
39310392
|
https://doi.org/10.7759/cureus.67613
|
[
"possible",
"this",
"testicular",
"cause",
"these",
"anomalies",
"association",
"double",
"ureter",
"arching"
] |
[
{
"code": "QA86",
"title": "Problem with isolation protocol without injury or harm"
},
{
"code": "6B41",
"title": "Complex post traumatic stress disorder"
},
{
"code": "JA8Z",
"title": "Maternal care related to unspecified fetus, amniotic cavity or possible delivery problems"
},
{
"code": "JA8Y",
"title": "Maternal care related to other specified fetus, amniotic cavity or possible delivery problems"
},
{
"code": "4A01.03",
"title": "Transient hypogammaglobulinaemia of infancy"
},
{
"code": "GB0Y&XA4947",
"title": "Disease or disorder of testis, unspecified"
},
{
"code": "GB02.1&XA4947",
"title": "Inflammation of testis"
},
{
"code": "LB51",
"title": "Anorchia or microorchidia"
},
{
"code": "GB03",
"title": "Atrophy of testis"
},
{
"code": "2F97&XA4947",
"title": "Neoplasms of unknown behaviour of testis"
}
] |
=== ICD-11 CODES FOUND ===
|
[] |
QA86
|
Problem with isolation protocol without injury or harm
|
Given the clear diagnosis of ADHD in the child, she was treated with methylphenidate, starting at a dose of 18 mg/day, which was gradually increased to 36 mg/day based on her attentional performance, while also undergoing physical and language rehabilitation training. Additionally, the pediatrician advised the parents to engage the child in reading and retelling stories at home daily. The parents were fully involved in the treatment process. During the first year of treatment, the parents reported a positive impact, and the child's teacher noted an improvement in her attention span and the speed of completing homework. However, in the second year of treatment, both parents and the teacher reported that the child had difficulty concentrating again and exhibited learning difficulties, prompting an increase in the methylphenidate dose to 45 mg/day. This led to difficulties in falling asleep at night and poor sleep quality, so the dose was reduced back to 36 mg/day ( Table 1 ). After two years of regular treatment, the child still met the diagnostic criteria for ADHD. Therefore, this suggests that methylphenidate was not effective in treating the child, and given her comorbid conditions, the possibility of inherited metabolic diseases should be considered. To further investigate the etiology and guide treatment, after obtaining informed consent from the parents, we conducted whole exome sequencing.
| 4.007813
| 0.932129
|
sec[4]/p[0]
|
en
| 0.999997
|
PMC11703840
|
https://doi.org/10.3389/fped.2024.1500152
|
[
"parents",
"methylphenidate",
"adhd",
"teacher",
"that",
"difficulties",
"this",
"clear",
"treated",
"starting"
] |
[
{
"code": "QE50.4",
"title": "Relationship with parents, in-laws or other family members"
},
{
"code": "QE52.1",
"title": "Loss of love relationship in childhood"
},
{
"code": "QE9Y",
"title": "Other specified problems associated with upbringing"
},
{
"code": "QE52.0",
"title": "Caregiver-child relationship problem"
},
{
"code": "QE95",
"title": "Inappropriate parental pressure or other abnormal qualities of upbringing"
},
{
"code": "PB22&XM1NX2",
"title": "Unintentional exposure to or harmful effects of methylphenidate"
},
{
"code": "PC92&XM1NX2",
"title": "Intentional self-harm by exposure to or harmful effects of methylphenidate"
},
{
"code": "PE82&XM1NX2",
"title": "Assault by exposure to or harmful effects of methylphenidate"
},
{
"code": "NE60",
"title": "Harmful effects of drugs, medicaments or biological substances, not elsewhere classified"
},
{
"code": "PH42&XM1NX2",
"title": "Harmful effects of or exposure to noxious substances, drugs, medicaments or biological substances, psychostimulants, methylphenidate, undetermined intent"
}
] |
=== ICD-11 CODES FOUND ===
|
[] |
QE50.4
|
Relationship with parents, in-laws or other family members
|
During his hospital stay, pulmonary consolidation involving the right upper lobe, with pleural effusion and enlarged mediastinal lymph nodes, was detected, associated with an increase in inflammatory markers. Suspecting an infectious origin, systemic empirical antibiotic therapy (levofloxacin, later switched to piperacillin/tazobactam) was administered. A few days later, the patient developed disseminated hemorrhagic edematous cutaneous lesions with vesiculation and necrotic trend toward punched-out ulcerations, primarily on the head and, to a lesser extent, the trunk and limbs, where they appeared more scattered and with an umbilicated morphology. Two days after the onset of cutaneous manifestations, the patient was transferred to the intensive care unit (ICU) due to a deteriorating general condition, with respiratory failure and fever requiring invasive ventilation. The cutaneous manifestations worsened, with extensive skin necrosis, particularly in the nasal area, covered by granulation tissue and hemorrhagic and bullous lesions . Erosive lesions involved the tongue, oral, and nasal mucosa with significant crusting of the lips. A biopsy was taken from the right arm for histological examination, cultures, and direct immunofluorescence (DIF). Systemic therapy with antifungal and antiviral agents was initiated, and antibiotic therapy was continued. However, the patient continued to deteriorate.
| 3.931641
| 0.979004
|
sec[1]/p[1]
|
en
| 0.999997
|
39444817
|
https://doi.org/10.3389/fmed.2024.1468712
|
[
"cutaneous",
"lesions",
"systemic",
"antibiotic",
"hemorrhagic",
"manifestations",
"nasal",
"stay",
"pulmonary",
"consolidation"
] |
[
{
"code": "ME60.Z",
"title": "Skin lesion of unspecified nature"
},
{
"code": "EB90.40",
"title": "Dystrophic calcification of the skin of uncertain or unspecified aetiology"
},
{
"code": "ND56.0",
"title": "Superficial injury of unspecified body region"
},
{
"code": "ME60.3",
"title": "Keratosis of skin of uncertain or unspecified nature"
},
{
"code": "EB90.1",
"title": "Cutaneous mucinosis"
},
{
"code": "FA5Z",
"title": "Arthropathies, unspecified"
},
{
"code": "FC0Z",
"title": "Diseases of the musculoskeletal system or connective tissue, unspecified"
},
{
"code": "MD41",
"title": "Clinical findings on diagnostic imaging of lung"
},
{
"code": "GC2Z&XA6KU8",
"title": "Disease of kidney, not elsewhere classified"
},
{
"code": "4A42.Z",
"title": "Systemic sclerosis, unspecified"
}
] |
=== ICD-11 CODES FOUND ===
|
[] |
ME60.Z
|
Skin lesion of unspecified nature
|
The absence of insulinopenia and episodes of ketosis did not allow classification as idiopathic type 1 diabetes . Type 2 diabetes was unlikely due to age diagnosis, lack of weight, and family history of diabetes. Three monogenic diabetes that are associated with liver disease could be mentioned. The first is lipoatrophic diabetes which can lead to end-stage liver disease secondary to nonalcoholic steatohepatitis . However, our patient did not have a paucity of fat or insulin resistance to suggest lipoatrophic diabetes. The association between Maturity-onset diabetes of the young type 3 (MODY-3) and liver adenomatosis has been reported . However, liver adenomatosis does not lead directly to end-stage liver disease. The association between MODY-5 and an increased liver enzyme has been reported . Candidates for genetic testing for MODY include nonobese subjects with hyperglycemia, no evidence of Î 2 -cell autoimmunity, preserved Î 2 -cell function, and a strong family history of similar-type diabetes among first-degree relatives . Although this patient had many criteria in favor of, the probability of MODY seemed low in the absence of a family history of similar diabetes in first-degree relatives and high insulin requirements. Because his clinical profile did not fit with one of the known MODY types, genetic testing was not performed given the cost and logistic constraints associated with testing.
| 4.132813
| 0.63916
|
sec[2]/p[1]
|
en
| 0.999996
|
PMC12032616
|
https://doi.org/10.11604/pamj.2025.50.16.46217
|
[
"diabetes",
"liver",
"mody",
"type",
"family",
"testing",
"absence",
"associated",
"lipoatrophic",
"lead"
] |
[
{
"code": "5A14",
"title": "Diabetes mellitus, type unspecified"
},
{
"code": "JA63.2",
"title": "Diabetes mellitus arising in pregnancy"
},
{
"code": "5A13.4",
"title": "Diabetes mellitus due to drug or chemical"
},
{
"code": "LD2H.Y",
"title": "Other specified syndromic genetic deafness"
},
{
"code": "5C64.3",
"title": "Disorders of phosphorus metabolism or phosphatases"
},
{
"code": "DB9Z",
"title": "Diseases of liver, unspecified"
},
{
"code": "DB97.Z",
"title": "Inflammatory liver disease, unspecified"
},
{
"code": "DB99.7",
"title": "Hepatic failure without mention whether acute or chronic"
},
{
"code": "LB20.0Y",
"title": "Other specified structural developmental anomalies of liver"
},
{
"code": "LB20.0Z",
"title": "Structural developmental anomalies of liver, unspecified"
}
] |
=== ICD-11 CODES FOUND ===
|
[] |
5A14
|
Diabetes mellitus, type unspecified
|
During following weeks there was a significant, life-threatening deterioration in the patient’s clinical condition (NEU – 0.0 10 3 /μl, Hb – 9.1 g/dl). Despite a lack of confirmation of the genetic diagnosis, due to the rapid deterioration of the patient’s condition, the medical council decided to implement non-standard treatment. Therefore, immunoablative treatment using anti-thymocyte globulins (ATG) (according to the SAA program) was implemented. A good response was achieved (the number of granulocytes increased to 74 x 10 3 /μl), but after 2 weeks the symptoms recurred. On Day 24 after ATG administration, the white blood cells (WBC) count dropped to 0.67 x 10 3 /μl. Due to the initial good response to ATG treatment, it was decided to start immunosuppressive treatment in accordance with the program for the treatment of HLH - dexamethasone, etoposide and cyclosporine. Improvement was achieved (WBC – 6.27 x 10 3 /μl) after 4 weeks of treatment. Due to persistent hypogammaglobulinemia, the patient received IVIG infusions. Then, for 11 months, the treatment according to the HLH 2004 regimen was continued, but it was complicated by the symptoms of renal failure (urea – 86 mg/dl), elevated N-terminal pro b-type natriuretic peptide – 34 130 pg/ml without clinical symptoms of heart failure and cytomegalovirus infection treated with ganciclovir. After 2 weeks of treatment, the virus was undetectable.
| 3.962891
| 0.972168
|
sec[1]/p[3]
|
en
| 0.999998
|
39624103
|
https://doi.org/10.3389/fimmu.2024.1484467
|
[
"deterioration",
"decided",
"according",
"program",
"good",
"response",
"failure",
"life",
"threatening",
"despite"
] |
[
{
"code": "PL13.8",
"title": "Expired or deteriorated medication or substance, as mode of injury or harm"
},
{
"code": "6A2Z",
"title": "Schizophrenia or other primary psychotic disorders, unspecified"
},
{
"code": "BE2Y",
"title": "Other specified diseases of the circulatory system"
},
{
"code": "MG20.Z",
"title": "Cachexia, unspecified"
},
{
"code": "MG22",
"title": "Fatigue"
},
{
"code": "QA47.Z",
"title": "Liveborn infants according to place of birth, unspecified"
},
{
"code": "EE12.1",
"title": "Onychomycosis"
},
{
"code": "QA00.5",
"title": "Examination for normal comparison or control in clinical research programme"
},
{
"code": "4B40.2",
"title": "Good syndrome"
},
{
"code": "6B24.0",
"title": "Hoarding disorder with fair to good insight"
}
] |
=== ICD-11 CODES FOUND ===
|
[] |
PL13.8
|
Expired or deteriorated medication or substance, as mode of injury or harm
|
Pathogenic variants in the WDR81 gene located on chromosome 17p13.3 are associated with cerebellar ataxia, impaired intellectual development, and disequilibrium syndrome-2 (CAMRQ2). This disorder is characterized by congenital cerebellar ataxia, intellectual disability, and gait abnormalities . This disorder is extremely rare, with fewer than 20 previously published cases in the literature . All cases of CAMRQ2 that have been reported are consanguineous with autosomal recessive inheritance . The WDR81 gene has been found to show some similarity with genes including NSMAF (neutral sphingomyelinase activation associated factor), NBEA (neurobeachin), and LYST (lysosomal trafficking regulator) . Previously published cases describe other varying features, including thoracic kyphosis and scoliosis, short stature, absent or limited speech, intention tremor, coarse facial features, hirsutism, strabismus, wide and short neck, and small hands and feet . Findings on brain MRI of previously published cases include cerebellar atrophy, generalized brain atrophy, and hypoplasia of the corpus callosum . All previously published cases have occurred in consanguineous families, and the majority of affected individuals ambulated with a quadrupedal gait . Here, we describe a case report of a more mildly affected female with bipedal gait from a nonconsanguineous family, expanding the phenotype of this rare disorder.
| 4.390625
| 0.730957
|
sec[0]/p[0]
|
en
| 0.999998
|
40013199
|
https://doi.org/10.7759/cureus.78066
|
[
"cases",
"previously",
"published",
"cerebellar",
"this",
"gait",
"gene",
"associated",
"ataxia",
"intellectual"
] |
[
{
"code": "JB20.Z",
"title": "Single spontaneous delivery, unspecified"
},
{
"code": "QA48.0",
"title": "Care or examination immediately after delivery"
},
{
"code": "6A02.5",
"title": "Autism spectrum disorder with disorder of intellectual development and with absence of functional language"
},
{
"code": "6A02.3",
"title": "Autism spectrum disorder with disorder of intellectual development and with impaired functional language"
},
{
"code": "6A02.Y",
"title": "Other specified autism spectrum disorder"
},
{
"code": "6A02.2",
"title": "Autism spectrum disorder without disorder of intellectual development and with impaired functional language"
},
{
"code": "6A02.1",
"title": "Autism spectrum disorder with disorder of intellectual development and with mild or no impairment of functional language"
},
{
"code": "LA06.Z",
"title": "Cerebellar structural developmental anomalies, unspecified"
},
{
"code": "8E7Y",
"title": "Other specified diseases of the nervous system"
},
{
"code": "NA07.3Y&XA1CW2",
"title": "Laceration of cerebellum"
}
] |
=== ICD-11 CODES FOUND ===
|
[] |
JB20.Z
|
Single spontaneous delivery, unspecified
|
The laboratory findings revealed several significant abnormalities. The hemoglobin level was 11.3 g/dL (normal range: 12.0-16.0 g/dL). The platelet count was within normal limits at 182,000/μL (normal range: 150,000-450,000/μL). However, both aspartate aminotransferase (ASAT) and alanine aminotransferase (ALAT) were markedly elevated. ASAT was 8 times above the normal range, while ALAT was 4 times elevated (normal range: ASAT 10-40 U/L, ALAT 7-56 U/L). The prothrombin time was reduced to 55%, below the normal range of 70%-100%. Potassium levels were found to be low at 3.2 mmol/L (normal range: 3.5-5.0 mmol/L), and creatinine was significantly elevated at 10.82 mg/dL (normal range: 0.6-1.2 mg/dL). Urea levels were 0.43 g/dL (normal range: 0.7-1.2 g/dL). Arterial blood gas analysis revealed a pH of 7.54, indicative of metabolic alkalosis (normal range: 7.35-7.45). The partial pressure of oxygen (PaO2) was elevated at 148 mmHg, exceeding the normal range of 75-100 mmHg. The partial pressure of carbon dioxide (PaCO2) was low at 6.1 mmHg (normal range: 35-45 mmHg), suggesting respiratory alkalosis. The bicarbonate (HCO3-) level was low at 6.1 mmol/L (normal range: 22-28 mmol/L), and lactate levels were elevated at 7.9 mmol/L, indicating hyperlactatemia (normal range: 0.5-2.2 mmol/L). Additionally, proteinuria was present, with a level of 900 mg/mL, significantly above the normal range of <150 mg/mL.
| 4.277344
| 0.553711
|
sec[1]/p[1]
|
en
| 0.999996
|
39850765
|
https://doi.org/10.1016/j.radcr.2024.12.018
|
[
"range",
"mmol",
"mmhg",
"asat",
"alat",
"aminotransferase",
"times",
"significantly",
"alkalosis",
"partial"
] |
[
{
"code": "QA00.6Y",
"title": "Other specified examination of eyes or vision"
},
{
"code": "4B00.0Z",
"title": "Neutropaenia, unspecified"
},
{
"code": "3B63.1Z",
"title": "Acquired thrombocytosis, unspecified"
},
{
"code": "MA14.1C",
"title": "Raised antibody titre"
},
{
"code": "BD11.1",
"title": "Left ventricular failure with mid range ejection fraction"
},
{
"code": "GB42.1",
"title": "Albuminuria, Grade A3"
},
{
"code": "GB42.0",
"title": "Albuminuria, Grade A2"
},
{
"code": "MA18.0Y",
"title": "Other specified elevated blood glucose level"
},
{
"code": "5C50.9",
"title": "Disorders of ornithine metabolism"
},
{
"code": "5C50.12",
"title": "Tyrosinaemia type 2"
}
] |
=== ICD-11 CODES FOUND ===
|
[] |
QA00.6Y
|
Other specified examination of eyes or vision
|
A 46-year-old man (BSA 1.92 m 2 ; BMI 24.5 Kg/m 2 ) without previous cardiovascular events, who was affected by anti-Mi2 dermatomyositis and lymphocytic interstitial pneumonia (LIP) with chronic respiratory failure and was treated with 50 mg of azathioprine daily, 25 mg prednisone daily, 700 mg of intravenous (IV) rituximab once weekly, and oxygen therapy (1 to 6 L per minute), was admitted to the Emergency Department (ED) of our institution due to ongoing dyspnoea, chest pain, and general malaise. At the hospital admission, the patient’s arterial oxygen saturation (SaO2) in ambient air was 66%, their blood pressure was 110/70 mmHg, their heart rate was 102 b.p.m., and their body temperature was 36.3 °C. A blood gas analysis showed hypoxemia (PaO2 = 39.6 mmHg) and hypocapnia (PaCO2 = 27.2 mmHg), pH = 7.3, and a lactate level of 7.7 mmol/L (normal range: 0.36–1.25 mmol/L). Blood tests revealed a serum haemoglobin amount of 12.8 g/dL; a serum white blood cell count of 13,900 × 10 6 /L ; a serum Neutrophil–Lymphocyte Ratio (NLR) of 18.6; a serum creatinine level of 1.57 mg/dL; a serum troponin I level of 0.42 ng/mL (normal range 0.00–0.04 ng/mL); a serum C-reactive protein (CRP) amount of 58 mg/L (normal range: 0–5 mg/L); a serum D-dimer of 4215 microg/L (normal range: 1–500 microg/L); and a serum N-terminal pro-B-type natriuretic peptide (NT-proBNP) level of 6474 pg/mL (normal range: <125 pg/mL).
| 4.019531
| 0.97168
|
sec[1]/p[0]
|
en
| 0.999998
|
PMC11942914
|
https://doi.org/10.3390/jcm14062035
|
[
"serum",
"range",
"blood",
"their",
"mmhg",
"daily",
"oxygen",
"mmol",
"microg",
"without"
] |
[
{
"code": "NE80.3",
"title": "Other serum reactions"
},
{
"code": "5D0Y",
"title": "Other specified metabolic disorders"
},
{
"code": "5B91.0",
"title": "Hypercalcaemia"
},
{
"code": "4A84.Y",
"title": "Other specified anaphylaxis"
},
{
"code": "5C50.F2",
"title": "Homocarnosinosis"
},
{
"code": "QA00.6Y",
"title": "Other specified examination of eyes or vision"
},
{
"code": "4B00.0Z",
"title": "Neutropaenia, unspecified"
},
{
"code": "3B63.1Z",
"title": "Acquired thrombocytosis, unspecified"
},
{
"code": "MA14.1C",
"title": "Raised antibody titre"
},
{
"code": "BD11.1",
"title": "Left ventricular failure with mid range ejection fraction"
}
] |
=== ICD-11 CODES FOUND ===
|
[] |
NE80.3
|
Other serum reactions
|
Drug-induced urinary calculi are rare. Previous studies reported that ceftriaxone, a ceftriaxone drug, can cause urinary calculi. The mechanism may involve the binding of ceftriaxone to calcium ions, leading to the formation of crystals that closely adhere to the surface of renal tubule cells, thereby blocking renal tubules, damaging cells, and interfering with calcium reabsorption. The increased calcium level in the urine continues to bind to ceftriaxone and form more crystals. Although ceftazidime causes urinary calculi, only 1 study reported that calculi disappeared after the insertion of a double J-type ureteral stent during surgery and the cessation of intravenous injection of ceftazidime and potassium sodium hydrogen citrate particles to alkalize the urine. The patient underwent laparoscopic appendectomy due to acute appendicitis. Preoperative CT revealed no urinary calculi, and the patient only received only anti-infection treatment with ceftazidime without any other drug injection or drug administration, The patient had no metabolic disorders, malnutrition or other diseases. Therefore, we believe that the calculi in the right kidney and ureter of the patient were caused by ceftazidime. In addition, we refer to the treatment plan of ceftriaxone causing urinary calculi and recommend ceftazidime drug discontinuation, spasmolytic treatment, and hydration treatment before calculus discharge.
| 4.082031
| 0.749023
|
sec[2]/p[0]
|
en
| 0.999998
|
40258730
|
https://doi.org/10.1097/MD.0000000000042221
|
[
"calculi",
"drug",
"urinary",
"ceftriaxone",
"ceftazidime",
"that",
"calcium",
"crystals",
"renal",
"cells"
] |
[
{
"code": "GB70.Z",
"title": "Calculus of upper urinary tract, unspecified"
},
{
"code": "GB71.0",
"title": "Calculus in bladder"
},
{
"code": "CB40.Y",
"title": "Other specified diseases of the respiratory system"
},
{
"code": "GB70.0Z",
"title": "Calculus of kidney, unspecified"
},
{
"code": "DC11.3",
"title": "Calculus of gallbladder or cystic duct without cholecystitis or cholangitis"
},
{
"code": "QE11.Z",
"title": "Hazardous drug use, unspecified"
},
{
"code": "6C4G.2Z",
"title": "Unknown or unspecified psychoactive substance dependence, substance and state of remission unspecified"
},
{
"code": "NE60",
"title": "Harmful effects of drugs, medicaments or biological substances, not elsewhere classified"
},
{
"code": "4A85.0Z",
"title": "Drug hypersensitivity of unspecified type"
},
{
"code": "6C4G.3",
"title": "Intoxication due to unknown or unspecified psychoactive substance"
}
] |
=== ICD-11 CODES FOUND ===
|
[] |
GB70.Z
|
Calculus of upper urinary tract, unspecified
|
Metastasis of the tumor to the calvarium can occur in several cancers, most frequently originating from the prostate, breast, thyroid, and lung. Bladder carcinoma typically spreads to the bone marrow, liver, and lungs. However, skull metastasis from urothelial carcinoma of the urinary bladder (UCB) is rare, occurring in less than 1.0% of patients with this condition. When distant metastasis occurs, the prognosis becomes poor. A solid mass causing lytic lesions in the skull indicates metastasis and can present with a range of imaging findings. Metastasis to the skull can lead to numerous clinical symptoms, like local swelling, ulceration of skin, cosmetic issues, pain, bleeding, dural sinus compression, neurological deficits, and sinus thrombosis. Metastasis to the bone significantly deteriorates the prognosis for cancer patients due to the progression of the primary cancer or organ infiltration. If metastases to the cranium invade or overlie the dural venous sinus, resecting them surgically can offer effective palliation for symptomatic calvarial metastasis. Additionally, focal palliative radiotherapy can help relieve symptoms and prevent recurrence . One of the key goals in staging the tumor is to determine whether it has invaded the muscle, as this significantly affects the prognosis and management. We present a patient who is a middle-aged woman with calvarial metastasis from bladder cancer.
| 4.140625
| 0.937012
|
sec[0]/p[1]
|
en
| 0.999999
|
PMC11282353
|
https://doi.org/10.7759/cureus.63171
|
[
"metastasis",
"bladder",
"skull",
"prognosis",
"sinus",
"cancer",
"tumor",
"carcinoma",
"bone",
"patients"
] |
[
{
"code": "2E2Z",
"title": "Malignant neoplasm metastasis, unspecified"
},
{
"code": "2E03",
"title": "Malignant neoplasm metastasis in bone or bone marrow"
},
{
"code": "2E08",
"title": "Metastatic malignant neoplasm involving skin"
},
{
"code": "2E0Y&XA25Q2",
"title": "Malignant neoplasm metastasis in pelvic viscera"
},
{
"code": "2D8Y&XA1WN1",
"title": "Malignant neoplasm metastasis in oral cavity"
},
{
"code": "GC01.Z",
"title": "Disorder of bladder, unspecified"
},
{
"code": "GC00.1",
"title": "Infectious cystitis"
},
{
"code": "GC01.0",
"title": "Bladder neck obstruction"
},
{
"code": "GC01.Y",
"title": "Other specified disorders of bladder"
},
{
"code": "QF01.Y",
"title": "Other specified acquired absence of organs"
}
] |
=== ICD-11 CODES FOUND ===
|
[] |
2E2Z
|
Malignant neoplasm metastasis, unspecified
|
A 3-month-old male infant, 5.35 kg, was diagnosed with severe MS and MR via transthoracic echocardiography . Preoperative echocardiography revealed right heart and left atrial enlargement, with the left ventricle compressed into a “D” shape. The mitral valve is significantly thickened, with hypertrophic and underdeveloped papillary muscles and thickened, shortened chordae tendineae. The valve leaflets are stiff, with restricted opening (orifice area −1.0 cm 2 ) and poor closure. The tricuspid valve is also thickened, with thickened and shortened sub-valvular chordae tendineae and poor closure. Color Doppler flow imaging (CDFI) revealed accelerated diastolic blood flow through the mitral valve with a peak velocity of 2.7 m/s, a pressure gradient of 29 mmHg, and a mean pressure gradient of 13 mmHg. Significant regurgitation signals are observed on the left atrial side during systole for the mitral valve and on the right atrial side during systole for the tricuspid valve. To address these conditions, the patient underwent a complex mitral valvuloplasty procedure under anesthesia . The mitral valve repair procedure includes several key steps: firstly, the abnormal chordae tendineae are excised, followed by commissurotomy and papillary muscle release. Subsequently, artificial chordae tendineae are placed, and repair is performed using the commissure magic stitch technique ( Supplementary Video S1 ).
| 4.144531
| 0.92627
|
sec[1]/p[0]
|
en
| 0.999996
|
39114560
|
https://doi.org/10.3389/fcvm.2024.1425900
|
[
"valve",
"mitral",
"thickened",
"chordae",
"tendineae",
"atrial",
"echocardiography",
"papillary",
"shortened",
"poor"
] |
[
{
"code": "GB61.Z",
"title": "Chronic kidney disease, stage unspecified"
},
{
"code": "BC00",
"title": "Multiple valve disease"
},
{
"code": "BB9Z",
"title": "Pulmonary valve disease, unspecified"
},
{
"code": "BB6Z",
"title": "Mitral valve disease, unspecified"
},
{
"code": "LA8Z",
"title": "Structural developmental anomaly of heart or great vessels, unspecified"
},
{
"code": "BB60.Z",
"title": "Mitral valve stenosis, unspecified"
},
{
"code": "LA89.2",
"title": "Mitral atresia"
},
{
"code": "LA87.11",
"title": "Congenital mitral valvar stenosis"
},
{
"code": "LA87.10",
"title": "Congenital mitral regurgitation"
},
{
"code": "FB86.11",
"title": "Hypertrophy of bone"
}
] |
=== ICD-11 CODES FOUND ===
|
[] |
GB61.Z
|
Chronic kidney disease, stage unspecified
|
In September of 2022, she began seeing a pain management specialist at the Florida Spine Institute for chronic pain due to fibromyalgia and rheumatoid arthritis that had been worsening despite her treatment regimen at that time. During this visit, the doctor first suggested ketamine treatment for her pain as well as her depression. In October, she began taking 30 mg ketamine capsules daily, which was then increased to three times daily in December. On December 19, 2022, she began a 10-day IV ketamine infusion per protocol as a treatment for her depression. She reported that this was her first time ever trying ketamine treatment and immediately saw hopeful results. These infusions were administered according to the pain management doctor's prescribed protocol, using a starting dose of 150 mg of ketamine alongside 10 mg of lidocaine, 4 mg of ondansetron, and 25 mg of promethazine to prevent nausea and vomiting. She successfully completed this treatment with no adverse effects and reported improved mood and energy by the end of the first round of infusion. By her third treatment, she reported great improvement in her depression, resulting in increased socialization and activity compared to her usual state. In January of 2023, she continued using ketamine hydrochloride (HCL) powder and was prescribed 30 mg ketamine lozenges three times daily. She continued follow-up infusions for two days per week.
| 3.611328
| 0.974609
|
sec[1]/p[1]
|
en
| 0.999997
|
PMC11342591
|
https://doi.org/10.7759/cureus.65261
|
[
"ketamine",
"pain",
"that",
"this",
"depression",
"daily",
"time",
"doctor",
"three",
"times"
] |
[
{
"code": "NE60",
"title": "Harmful effects of drugs, medicaments or biological substances, not elsewhere classified"
},
{
"code": "PB21&XM7C11",
"title": "Unintentional exposure to or harmful effects of ketamine"
},
{
"code": "PC91&XM7C11",
"title": "Intentional self-harm by exposure to or harmful effects of ketamine"
},
{
"code": "6C4D.2Z",
"title": "Dissociative drug dependence including ketamine or PCP, unspecified"
},
{
"code": "6C4D.3",
"title": "Dissociative drug intoxication including Ketamine or PCP"
},
{
"code": "MG3Z",
"title": "Pain, unspecified"
},
{
"code": "8E43.Z",
"title": "Pain disorders, unspecified"
},
{
"code": "MG31.Z",
"title": "Acute pain, unspecified"
},
{
"code": "MG30.Z",
"title": "Chronic pain, unspecified"
},
{
"code": "FB56.2",
"title": "Myalgia"
}
] |
=== ICD-11 CODES FOUND ===
|
[] |
NE60
|
Harmful effects of drugs, medicaments or biological substances, not elsewhere classified
|
A 59-year-old female was seen for recurrent allergic reactions. Her history included anxiety, carpal tunnel syndrome, type 2 diabetes mellitus with diabetic polyneuropathy, essential hypertension, morbid obesity, and opioid use disorder. Her reactions began with palmar and plantar itch, followed by a cascade of variable symptoms, namely, hand edema, generalized urticaria, facial and tongue swelling, palpitations, a lightheaded sensation and presyncope, nausea, vomiting, and diarrhea. These symptoms resolved after about 1 hour and were followed by a day of generalized fatigue. Diphenhydramine had afforded modest relief of her acute symptoms. She reported 8 to 9 episodes during the prior year. The findings of her baseline physical examination were normal, as were her levels of serum tryptase and urinary excretion of MC mediator metabolites ( Table II ). Administration of cetirizine, 10 mg orally per day, was started, and she was instructed to take prednisone, 40 mg, as well as diphenhydramine and famotidine, at the next onset of symptoms. Table II Patient 2 baseline and acute serum tryptase and urinary mast cell mediator metabolite values Mediator (reference range) Baseline value Acute value Acute/baseline Tryptase (ng/mL), (<11.5) 8.6 29.9 3.5 N-MH (μg/g of Cr), (30-200) 76 86 1.1 (2,3-dinor)-11β-PGF 2α pg/mg of Cr, (<1,802) 948 15,246 16.1 LTE 4 (pg/mg of Cr), (≤104) 81 534 6.6 Cr , Creatinine.
| 4.035156
| 0.96875
|
sec[1]/sec[1]/sec[1]/p[0]
|
en
| 0.999997
|
39906893
|
https://doi.org/10.1016/j.jacig.2024.100399
|
[
"baseline",
"tryptase",
"mediator",
"reactions",
"diphenhydramine",
"serum",
"urinary",
"recurrent",
"allergic",
"included"
] |
[
{
"code": "4A85.00",
"title": "Drug-induced liver hypersensitivity disease"
},
{
"code": "8C60.Y",
"title": "Other specified myasthenia gravis"
},
{
"code": "LA8Y",
"title": "Other specified structural developmental anomaly of heart or great vessels"
},
{
"code": "8D88.2",
"title": "Immune mediated autonomic neuropathy"
},
{
"code": "EB02.Y",
"title": "Other conditions mediated by cholinergic activation"
},
{
"code": "NE60",
"title": "Harmful effects of drugs, medicaments or biological substances, not elsewhere classified"
},
{
"code": "6B43",
"title": "Adjustment disorder"
},
{
"code": "NE80.Z",
"title": "Injury or harm arising following infusion, transfusion or therapeutic injection, not elsewhere classified, unspecified"
},
{
"code": "4B24.Z",
"title": "Graft-versus-host disease, unspecified"
},
{
"code": "NE80.3",
"title": "Other serum reactions"
}
] |
=== ICD-11 CODES FOUND ===
|
[] |
4A85.00
|
Drug-induced liver hypersensitivity disease
|
A 36-year-old woman initially went to a neurologist with complaints of frequent soreness in her right leg, and pain in the right instep, suspected to be a pinched nerve from the spine. Then, the longer the feeling of a lump in the right popliteal fossa, the diagnosis was “Baker's cyst.” Since 5 years ago, the right knee has felt sore, and since 4 years ago, it felt like a lump was getting bigger. The right instep to the lateral side of the ankle and the 1/3 distal of the lower leg sometimes felt hot, like burning, or painful like pulling. Since 2 years ago, it sometimes feels like an electric shock from the fold of the right knee to the right instep along the lateral side of the lower leg if the lump in the right popliteal fossa is hit (Tinel's sign is positive). The lump gets bigger, hurts more often, and interferes with rest or sleep at night. A physical examination showed that the patient was in pain, and no abnormalities were found in the vertebrae or elsewhere. In the right popliteal fossa, a mass was palpable, deep, more lateral, spongy, flat, mobile, single, and tender, and Tinel's sign was positive. Hypoesthesia sensibility in the right instep to the lateral ankle and distal to the right lower leg, and a slight drop in the foot . On radiographic examination of the right knee, a circumscript, single, more lateral mass was seen, measuring approximately 4 cm × 5 cm in the popliteal fossa .
| 3.744141
| 0.986328
|
sec[1]/p[0]
|
en
| 0.999999
|
39262572
|
https://doi.org/10.1155/2024/9397436
|
[
"instep",
"lump",
"popliteal",
"fossa",
"like",
"since",
"knee",
"felt",
"pain",
"bigger"
] |
[
{
"code": "ME61",
"title": "Subcutaneous swelling, mass or lump of uncertain or unspecified nature"
},
{
"code": "MD82",
"title": "Intra-abdominal or pelvic swelling, mass or lump"
},
{
"code": "LB30.61",
"title": "Fused pelvic kidney"
},
{
"code": "MF30",
"title": "Breast lump or mass female"
},
{
"code": "CA0H.2",
"title": "Nodules of vocal cords"
},
{
"code": "LD26.4Y",
"title": "Other specified arthrogryposis syndromes"
},
{
"code": "NC95.00",
"title": "Laceration of popliteal artery"
},
{
"code": "BD30.20&XA44K1",
"title": "Acute thromboembolic popliteal artery occlusion"
},
{
"code": "BD30.21&XA44K1",
"title": "Acute thrombotic popliteal artery occlusion"
},
{
"code": "BD53.3",
"title": "Popliteal entrapment syndrome"
}
] |
=== ICD-11 CODES FOUND ===
|
[] |
ME61
|
Subcutaneous swelling, mass or lump of uncertain or unspecified nature
|
The 63-year-old female reported herein developed a very rare postoperative complication of chronic pancreatitis after a commonly performed elective lumbar spine surgery for non-scoliosis arthritis. Her chronic pancreatitis thereafter became associated with a large pancreatic pseudocyst, which was ultimately treated successfully with an open Roux-en-Y cystojejunostomy. Although there are several possible etiologies of pancreatitis occurring soon after spinal surgery in adults, we could not identify, with high certainty, the cause of our patient’s postoperative complication. Leukocytosis was detected within a few days of symptom onset; however, this was not considered as being possibly associated with pancreatitis. This was despite her chief complaints of abdominal pain, nausea, vomiting, and general discomfort, as well as high amylase levels found in her retroperitoneal fluid. Because pancreatitis can be an elusive complication of spinal surgery in an adult, healthcare providers should consider this diagnosis prominently in the differential diagnosis for the underlying cause of these early postoperative symptoms and leukocytosis. We report this case not only because of its rarity but also to alert surgeons and other healthcare providers that pancreatitis can occur acutely in the postoperative period in adults and evolve into a chronic problem (pseudocyst formation) requiring surgical correction.
| 4.074219
| 0.964844
|
sec[3]/p[0]
|
en
| 0.999996
|
39463520
|
https://doi.org/10.7759/cureus.70272
|
[
"pancreatitis",
"postoperative",
"this",
"complication",
"associated",
"pseudocyst",
"spinal",
"adults",
"cause",
"leukocytosis"
] |
[
{
"code": "DC31.Z",
"title": "Acute pancreatitis, unspecified"
},
{
"code": "DC32.Z",
"title": "Chronic pancreatitis, unspecified"
},
{
"code": "DC34",
"title": "Obstructive pancreatitis"
},
{
"code": "NB91.4Z",
"title": "Injury of pancreas, unspecified"
},
{
"code": "DC32.0",
"title": "Calcific pancreatitis"
},
{
"code": "NE81.0Z",
"title": "Haemorrhage or haematoma of other or unspecified site complicating a procedure, not elsewhere classified"
},
{
"code": "NE81.3",
"title": "Postsurgical leak"
},
{
"code": "QF00",
"title": "Acquired absence of limb"
},
{
"code": "DB30.2",
"title": "Adhesions of large intestine with obstruction"
},
{
"code": "MG30.21",
"title": "Chronic postsurgical pain"
}
] |
=== ICD-11 CODES FOUND ===
|
[] |
DC31.Z
|
Acute pancreatitis, unspecified
|
Dense deposit disease (DDD) is an uncommon renal disorder linked to anomalies in the complement bypass pathway, exhibiting an annual incidence of 1–2 cases per million individuals. It predominantly affects children and adolescents and has a dismal prognosis, with approximately 50% of patients advancing to end-stage renal disease (ESRD) within a decade . Clinical manifestations encompass different degrees of hematuria, proteinuria, hypertension, and renal failure. Laboratory tests indicate persistent or recurrent low-complement C3 in the blood , with immunofluorescence revealing C3 deposition in the glomerular basement membrane and mesangial zone, accompanied by minimal or absent immunoglobulin deposition . IgA nephropathy is a glomerulonephritis distinguished by the deposition of IgA immunoglobulins in the glomerular mesangial region. This illness may also present with C3 deposition in the same area and exhibit clinical manifestations akin to DDD, including hematuria, hypertension, or renal failure . The pathological complexity and the challenges in diagnosis and treatment are markedly heightened when both diseases coexist in an individual. We present a rare renal pathological comorbidity: DDD in conjunction with IgA nephropathy, a case that illustrates the characteristic pathological symptoms of both conditions while also elucidating the intricate pathological link that may exist between them.
| 4.347656
| 0.833008
|
sec[0]/p[0]
|
en
| 0.999998
|
39833736
|
https://doi.org/10.1186/s12887-025-05415-z
|
[
"renal",
"deposition",
"pathological",
"complement",
"manifestations",
"hematuria",
"hypertension",
"failure",
"glomerular",
"mesangial"
] |
[
{
"code": "GC2Z&XA6KU8",
"title": "Disease of kidney, not elsewhere classified"
},
{
"code": "GB6Z",
"title": "Kidney failure, unspecified"
},
{
"code": "LB30.1",
"title": "Renal dysplasia"
},
{
"code": "NB92.0Y",
"title": "Other specified injury of kidney"
},
{
"code": "LB30.7",
"title": "Ectopic or pelvic kidney"
},
{
"code": "ME67",
"title": "Skin disorder of uncertain or unspecified nature"
},
{
"code": "9A78.1",
"title": "Corneal pigmentations or deposits"
},
{
"code": "9A61.6",
"title": "Conjunctival or subconjunctival degenerations or deposits"
},
{
"code": "FB40.Y",
"title": "Other specified tenosynovitis"
},
{
"code": "9A77.Y",
"title": "Other specified corneal scars or opacities"
}
] |
=== ICD-11 CODES FOUND ===
|
[] |
GC2Z&XA6KU8
|
Disease of kidney, not elsewhere classified
|
Overall, 79% of adult patients with IAE survived with established, evidence-based care . Patients suffered residual neurological defects however which include primarily cognitive deficits and motor weakness in approximately 25% . Those specific symptoms were evident in the immediate three-month period postinsult. Long-term symptoms and sequelae beyond three months are unknown due to under reporting . This patient was transferred to a LTACH facility for a higher level of specialized care. The three-month follow-up shows that this particular patient was nonresponsive. He has occasional episodes of spontaneous eye opening. He was not following verbal command or responding to external stimuli which was also seen in the acute phase of illness. Hypertonicity in all four extremities with significant contractures was noted in the upper extremities and lower extremities. Hypoactive reflexes were also noted on physical examination. CT head in the three-month period after initial insult showed a minimal degree of cerebral and cerebellar cortical atrophy and a minimal degree of central atrophy of the cerebral hemispheres. Decreased attenuation in white matter of both cerebral hemispheres was noted, consistent with demyelination. At that time, no acute findings of hemorrhage or mass effect were seen. At five-month postinsult, the patient did not have any semblance of partial or complete neurologic recovery.
| 4.054688
| 0.931152
|
sec[2]/p[11]
|
en
| 0.999997
|
33425396
|
https://doi.org/10.1155/2020/6616805
|
[
"three",
"extremities",
"cerebral",
"patients",
"which",
"period",
"postinsult",
"this",
"that",
"degree"
] |
[
{
"code": "1D45",
"title": "Sandfly fever"
},
{
"code": "PA09",
"title": "Unintentional land transport traffic event injuring an occupant of a low powered passenger vehicle"
},
{
"code": "LD50.Y",
"title": "Other specified number anomalies of chromosome X"
},
{
"code": "PA19",
"title": "Unintentional land transport nontraffic event injuring an occupant of a low powered passenger vehicle"
},
{
"code": "NA82.4",
"title": "Multiple fractures of ribs"
},
{
"code": "ND56.1",
"title": "Open wound of unspecified body region"
},
{
"code": "LB9Z",
"title": "Structural developmental anomalies of the skeleton, unspecified"
},
{
"code": "FB56.6",
"title": "Other specified soft tissue disorders"
},
{
"code": "5B51&XS25",
"title": "Severe wasting in infants, children or adolescents"
},
{
"code": "ND55",
"title": "Other injuries of leg, level unspecified"
}
] |
=== ICD-11 CODES FOUND ===
|
[] |
1D45
|
Sandfly fever
|
On examination, she looked skinny and body mass index was 18.49 (156 cm in height and 45 kg in weight). The Mini‐Mental State Examination (MMSE) and Montreal Cognitive Assessment (MoCA) scores were 10 and 6, respectively, suggesting that she had dementia. Facial and appendicular choreatic movements were present. Saccadic eye movements were characterized by variable onset latency and speed. Dysarthria was marked. The basal hormonal evaluation revealed inappropriately low luteinizing hormone (<0.1 mUI/mL), as well as low follicular‐stimulating hormone (1.19 mUI/mL) and estradiol (<18.4 pg/mL). Prolactin and cortisol levels were within normal range. Pelvic echography revealed a postmenopausal uterus (33 × 21 × 35 mm) and the ovaries were not seen. Brain MRI was also performed. The results showed moderate cerebellar atrophy on T1‐weighted image, and extensive bilateral white matter lesions in both cerebral hemispheres, as well as in the brainstem on T2‐weighted fluid‐attenuated inversion recovery image. Susceptibility‐weighted imaging showed no cerebral microbleeds in cortical and subcortical areas . DTI images displayed widespread white matter deterioration as reflected by the decreased fractional anisotropy values and increased radial diffusivity and axial diffusivity values in the voxels of both hemispheres compared with those of controls, while cerebellar white matter fibers relatively spared .
| 4.105469
| 0.925781
|
sec[2]/p[1]
|
en
| 0.999998
|
32358900
|
https://doi.org/10.1002/acn3.51047
|
[
"weighted",
"white",
"matter",
"movements",
"hormone",
"well",
"cerebellar",
"both",
"cerebral",
"hemispheres"
] |
[
{
"code": "MG43.5",
"title": "Excessive weight loss"
},
{
"code": "MG43.6",
"title": "Excessive weight gain"
},
{
"code": "MG44.11",
"title": "Failure to thrive in infant or child"
},
{
"code": "5B80.0Z",
"title": "Overweight, unspecified"
},
{
"code": "JA65.2",
"title": "Excessive weight gain in pregnancy"
},
{
"code": "EF5Y",
"title": "Other specified dermatoses attributable to hyperviscosity or microvascular occlusion"
},
{
"code": "JB41.1",
"title": "Deep phlebothrombosis in the puerperium"
},
{
"code": "EB60.Y",
"title": "Lichen sclerosus of other specified sites"
},
{
"code": "MC80.00",
"title": "White coat hypertension"
},
{
"code": "1F2D.2",
"title": "White piedra"
}
] |
=== ICD-11 CODES FOUND ===
|
[] |
MG43.5
|
Excessive weight loss
|
Most reported cases of orbital emphysema follow a history of trauma, though not necessarily immediately . Patients with a preceding episode of trauma days or weeks before may present acutely with orbital emphysema after an episode of increased intranasal pressure provoked by sneezing or nose-blowing. However, in this case, there was no preceding injury noted. From a review of the literature, we have identified only 21 cases of orbital emphysema where no history of trauma or periocular surgery was evident [ 8 – 28 ]. Cases of orbital emphysema are usually mild and do not affect visual function, with approximately 12% of patients requiring decompression . Of the 21 cases found with no history of trauma [ 8 – 28 ], one had nonurgent surgery due to persistent diplopia lasting more than a week . In this case, fractures of the orbital floor were apparent, with fat entrapment. Two of the 21 cases reported emergency needle decompression . In the first, there was proptosis, restriction of eye movements, raised intraocular pressure (IOP), and a hazy cornea. However, it was noted that there was no RAPD or dyschromatopsia and therefore no evidence of optic nerve compromise. In the second, decompression was performed due to the extent of eyelid swelling precluding assessment of the eye. As such, our case is the first to document clear evidence of optic nerve dysfunction after nontraumatic orbital emphysema.
| 4.191406
| 0.788574
|
sec[2]/p[0]
|
en
| 0.999997
|
33777467
|
https://doi.org/10.1155/2021/8884009
|
[
"orbital",
"cases",
"emphysema",
"trauma",
"decompression",
"patients",
"preceding",
"episode",
"pressure",
"however"
] |
[
{
"code": "9A2Z",
"title": "Disorders of orbit, unspecified"
},
{
"code": "9A22.Z",
"title": "Orbital inflammation, unspecified"
},
{
"code": "9A2Y",
"title": "Other specified disorders of orbit"
},
{
"code": "LA14.2",
"title": "Structural developmental anomalies of orbit"
},
{
"code": "9A24.0",
"title": "Contraction of orbit"
},
{
"code": "JB20.Z",
"title": "Single spontaneous delivery, unspecified"
},
{
"code": "QA48.0",
"title": "Care or examination immediately after delivery"
},
{
"code": "CA21.Z",
"title": "Emphysema, unspecified"
},
{
"code": "CA21.Y",
"title": "Other specified emphysema"
},
{
"code": "KB27.0",
"title": "Interstitial emphysema originating in the perinatal period"
}
] |
=== ICD-11 CODES FOUND ===
|
[] |
9A2Z
|
Disorders of orbit, unspecified
|
In case 1, a 3-year-old ate mozzarella cheese in her kindergarten dining room, sitting at a table. The child developed signs of airway obstruction and the teachers tried to give her aid through several back blows with no success. Unfortunately, the Italian school system does not make it mandatory for teachers to attend first aid classes . These guidelines report that in the absence of legislation in this regard, it is strongly recommended to provide for the constant presence of personnel who have followed a first aid course in the school canteens frequented mainly by children. Furthermore, the same guidelines recommend that families and adults responsible for supervising the child (e.g., educators in kindergartens, summer centers, after-school, teachers, babysitters) know the rules regarding food preparation and behavior at the table for the prevention of suffocation from food (for example, in the case of the use of plastic cutlery, ensure that these are hard and resistant); furthermore, families and adults responsible for the child’s supervision should acquire knowledge and skills regarding maneuvers to unblock airways and cardiopulmonary resuscitation. In this case, the Heimlich maneuver and adequate supervision of the child were needed. The child arrived at the hospital emergency department where the physicians declared death from suspected asphyxiation, which was later confirmed by autopsy.
| 3.326172
| 0.961426
|
sec[2]/p[1]
|
en
| 0.999997
|
32629891
|
https://doi.org/10.3390/ijerph17134700
|
[
"teachers",
"school",
"that",
"these",
"guidelines",
"this",
"furthermore",
"families",
"adults",
"responsible"
] |
[
{
"code": "QE50.1Z",
"title": "Relationships with teachers or classmates, unspecified"
},
{
"code": "QE50.1Y",
"title": "Other specified relationships with teachers or classmates"
},
{
"code": "CA0H.2",
"title": "Nodules of vocal cords"
},
{
"code": "QD91",
"title": "Problem associated with education unavailable or unattainable"
},
{
"code": "QD92",
"title": "Problem with educational progress"
},
{
"code": "QE50.10",
"title": "Dissatisfaction with school environment"
},
{
"code": "QA01.0",
"title": "Examination for admission to educational institution"
},
{
"code": "QA03.Y",
"title": "Other specified routine general health check-up of defined subpopulation"
},
{
"code": "8A80.Z",
"title": "Migraine, unspecified"
},
{
"code": "QA76",
"title": "Medication or substance that is known to be an allergen without injury or harm"
}
] |
=== ICD-11 CODES FOUND ===
|
[] |
QE50.1Z
|
Relationships with teachers or classmates, unspecified
|
Case 3 . A White man aged 50 years traveled to Burkina Faso for 10 days in November 2017 on a mission trip and did not take malaria prophylaxis. A few weeks after returning, he experienced fever and fatigue but did not seek medical attention. A week after the start of his symptoms, his daughter found him at home with altered mental status and a bruise on his forehead. She took him to an emergency department, where the workup revealed acute kidney injury, jaundice, and a positive malaria smear, with no species or parasitemia reported. A CT scan of the head showed no abnormalities. The patient was administered intravenous clindamycin and early the following day was transferred to another hospital, where he was admitted to the ICU and was given IV quinidine gluconate and IV doxycycline for treatment of severe malaria. Shortly after starting quinidine, a widened QRS complex was observed on an electrocardiogram, and quinidine toxicity was suspected. IV artesunate was requested from CDC and administered. However, the patient’s condition deteriorated, requiring intubation and mechanical ventilation, vasopressor support, and continuous renal replacement therapy for acute kidney injury. The patient died on the eighth day after being hospitalized. The malaria smear at the initial hospital was sent to an outside laboratory, and results available later revealed P. falciparum infection with 16% parasitemia.
| 3.8125
| 0.984863
|
sec[2]/sec[16]/sec[3]/p[3]
|
en
| 0.999997
|
33735166
|
https://doi.org/10.15585/mmwr.ss7002a1
|
[
"malaria",
"quinidine",
"kidney",
"injury",
"smear",
"parasitemia",
"white",
"traveled",
"burkina",
"faso"
] |
[
{
"code": "1F42.Z",
"title": "Plasmodium malariae malaria without complication"
},
{
"code": "1F45",
"title": "Malaria without parasitological confirmation"
},
{
"code": "1F4Z",
"title": "Malaria, unspecified"
},
{
"code": "1F44",
"title": "Other parasitologically confirmed malaria"
},
{
"code": "KA64.Y",
"title": "Other specified parasitic diseases in the fetus or newborn"
},
{
"code": "GC2Z&XA6KU8",
"title": "Disease of kidney, not elsewhere classified"
},
{
"code": "GB6Z",
"title": "Kidney failure, unspecified"
},
{
"code": "LB30.1",
"title": "Renal dysplasia"
},
{
"code": "NB92.0Y",
"title": "Other specified injury of kidney"
},
{
"code": "LB30.7",
"title": "Ectopic or pelvic kidney"
}
] |
=== ICD-11 CODES FOUND ===
|
[] |
1F42.Z
|
Plasmodium malariae malaria without complication
|
We present a case of characteristic ST-segment elevation suggestive of anteroseptal STEMI in a patient with no risk factors for coronary artery disease, a recently identified cardiac mass, and without any correlating symptoms. Our patient’s troponin T assay was found to be elevated above the 99th percentile of the upper reference limit on initial assessment without a subsequent rise and/or fall, and thus did not meet the criteria for myocardial infarction according to the Fourth Universal Definition of Myocardial Infarction . Given that the patient was a young, active-duty male (a patient population with a low risk for atherosclerotic coronary disease making STEMI highly unlikely), and that the clinical picture was not suggestive of an acute coronary syndrome (no symptoms of myocardial ischemia), the decision was made to forgo cardiac catheterization. With the time-sensitive and life-threatening nature of STEMIs, we believe this case illustrates the importance of maintaining a suspicion for alternative diagnoses when STEMI does not fit the clinical picture as the decision to begin anticoagulation and perform a left heart catheterization can place the patient at higher risk with less potential benefit in these scenarios. After much thought and discussion, we surmised that the ECG findings were most likely attributable to a persistent current of injury related to the interventricular septal mass.
| 4.082031
| 0.967773
|
sec[2]/p[0]
|
en
| 0.999997
|
33884235
|
https://doi.org/10.7759/cureus.13981
|
[
"stemi",
"risk",
"coronary",
"myocardial",
"that",
"suggestive",
"cardiac",
"without",
"infarction",
"picture"
] |
[
{
"code": "BA41.0",
"title": "Acute ST elevation myocardial infarction"
},
{
"code": "BA41.1",
"title": "Acute non-ST elevation myocardial infarction"
},
{
"code": "BA42.0",
"title": "Subsequent myocardial infarction, ST elevation myocardial infarction"
},
{
"code": "BA42.0&XA7RE3",
"title": "Subsequent myocardial infarction, ST elevation myocardial infarction, anterior wall"
},
{
"code": "BA42.0&XA3RM8",
"title": "Subsequent myocardial infarction, ST elevation myocardial infarction, inferior wall"
},
{
"code": "QC4Y",
"title": "Personal history of other specified health problems"
},
{
"code": "QA43.Z",
"title": "Supervision of high-risk pregnancy, unspecified"
},
{
"code": "QA43.Y",
"title": "Other specified supervision of high-risk pregnancy"
},
{
"code": "QD84.Z",
"title": "Occupational exposure to risk-factors, unspecified"
},
{
"code": "MB26.A",
"title": "Suicidal ideation"
}
] |
=== ICD-11 CODES FOUND ===
|
[] |
BA41.0
|
Acute ST elevation myocardial infarction
|
In May 2019, a patient with a clinical history of atrial fibrillation and hypertensive heart disease arrived at a local hospital in Sichuan province. No local or international travel or contact with wildlife or household animals was reported. A meal with rice noodles and raw vegetables outside the hospital was the only significant record for the initial medical history. Acute syndromes of high fever, expiratory dyspnoea, chills and diarrhoea were observed on the next day (Day 2 at 2AM). The patient also complained of headache and abdominal pain. Laboratory investigation indicated an elevated white-blood-cell count (12.97×10 9 /l). Oral berberine and intramuscular injection of gentamicin were given as primary treatment. Two hours later, body temperature had increased to 40 °C, accompanied by watery diarrhoea with tenesmus, nausea and vomiting. Cefoperazone/sulbactam and pantoprazole were administered as new treatment. On Day 3, most syndromes returned to normal, except for nausea, vomiting and watery diarrhoea. On Days 5 and 9, blood and stool samples were collected, respectively. The blood specimen confirmed positive culture for Salmonella , but no Shigella was detected or isolated from either sample. Sustained treatment with cefotaxime and pazufloxacin was given in the next 3 days and the patient recovered. The patient was diagnosed with infectious diarrhoea, complicated by invasive syndromes.
| 3.771484
| 0.984863
|
sec[1]/sec[0]/p[0]
|
en
| 0.999996
|
32589568
|
https://doi.org/10.1099/mgen.0.000401
|
[
"diarrhoea",
"syndromes",
"blood",
"local",
"next",
"watery",
"nausea",
"vomiting",
"atrial",
"fibrillation"
] |
[
{
"code": "ME05.1",
"title": "Diarrhoea"
},
{
"code": "1A40.Z",
"title": "Infectious gastroenteritis or colitis without specification of infectious agent"
},
{
"code": "DA90.0",
"title": "Syndromic diarrhoea"
},
{
"code": "DD91.2",
"title": "Functional diarrhoea"
},
{
"code": "1A2Z",
"title": "Viral intestinal infections, unspecified"
},
{
"code": "1D9Z",
"title": "Unspecified viral infection of unspecified site"
},
{
"code": "LD27.0Y",
"title": "Other specified ectodermal dysplasia syndromes"
},
{
"code": "5A44",
"title": "Insulin-resistance syndromes"
},
{
"code": "LD24.04",
"title": "Chondrodysplasia punctata"
},
{
"code": "3C0Z",
"title": "Diseases of the blood or blood-forming organs, unspecified"
}
] |
=== ICD-11 CODES FOUND ===
|
[] |
ME05.1
|
Diarrhoea
|
A 76‐year‐old man visited a private hospital with a chief complaint of left lower limb pain. Computed tomography and MRI revealed an irregular enlarged prostate in the left lobe, left obturator lymph node involvement, and multiple bone metastases . Bone scintigraphy confirmed the multiple bone metastases . A transrectal prostate biopsy was performed, as his serum PSA level was 365.156 ng/mL (normal range: <4.0 ng/mL). Histological evaluation led to the initial diagnosis of adenocarcinoma of Gleason score 4 + 4 and clinical stage T3a N1 M1b. ADT was performed immediately. The nadir PSA level was 0.213 ng/mL 13 months after ADT, the obturator lymph node was unclear but gradually increased. He developed CRPC 6 months after the initial treatment. Therefore, he received enzalutamide 160 mg/day and attained a serum PSA level of 0.002 ng/mL 7 months after the second treatment. The prostate volume was clearly decreased on MRI . The multiple bone metastases had disappeared on bone scintigraphy . As we deemed the patients’ mCRPC to be curable with immediate surgery, we performed RARP without PLND 1 year after diagnosis. The console time was 54 min, and the estimated blood loss was 10 mL. No surgery‐related adverse events occurred. Histopathological examination revealed that PCa cells disappeared into the prostate, and he attained pCR . He maintained an undetectable serum PSA level 6 months after surgery.
| 3.925781
| 0.978027
|
sec[1]/p[0]
|
en
| 0.999996
|
33426491
|
https://doi.org/10.1002/iju5.12229
|
[
"bone",
"prostate",
"multiple",
"metastases",
"serum",
"obturator",
"lymph",
"node",
"scintigraphy",
"attained"
] |
[
{
"code": "FC0Z",
"title": "Diseases of the musculoskeletal system or connective tissue, unspecified"
},
{
"code": "FB84.Z",
"title": "Osteomyelitis or osteitis, unspecified"
},
{
"code": "FB80.Z",
"title": "Disorder of bone density or structure, unspecified"
},
{
"code": "FB86.11",
"title": "Hypertrophy of bone"
},
{
"code": "FB86.1Z",
"title": "Bone hyperplasias, unspecified"
},
{
"code": "GA90",
"title": "Hyperplasia of prostate"
},
{
"code": "GA91.Z",
"title": "Inflammatory or other diseases of prostate, unspecified"
},
{
"code": "GA91.Y",
"title": "Other specified inflammatory or other diseases of prostate"
},
{
"code": "GA91.0",
"title": "Chronic prostatitis"
},
{
"code": "MF40.1",
"title": "Problems of the prostate"
}
] |
=== ICD-11 CODES FOUND ===
|
[] |
FC0Z
|
Diseases of the musculoskeletal system or connective tissue, unspecified
|
Patient 2 was a 73-year-old right-handed woman with a 5-year history of progressive decline in verbal communication, characterized by sparse and slow speech production. She did not speak unless addressed but produced stereotyped phrases, generated lengthy monologs, while echoing most of what she heard and completing simple open-ended sentences. It was also noticed that her regional accent was reverted to a previous variant learned during childhood . Her auditory comprehension and naming abilities were mildly impaired, but repetition was virtually intact. Two years after showing first deficits in spoken speech production, she suffered four falls in 1 year and developed apathy, bradyphrenia and emotional incontinence displaying uncontrollable episodes of crying and laughing. Neurological examination disclosed slow, hypometric saccades, decreased rate blink, bilateral limb rigidity with impaired finger and toe tapping, as well as tremor and bilateral hand grasping. She also showed neck rigidity, wide-based gait, postural instability, and problems arising from a chair and shortcomings on sitting down. Features consistent with PSP were revealed with the PSPRS . Family history was negative for movement disorders or dementia. A brain MRI showed moderate cortical, subcortical, and midbrain tegmentum atrophy . Treatments with carbidopa/levodopa (25/250 mg/tid) and amantadine (300 mg/bid) were unhelpful.
| 4.042969
| 0.980469
|
sec[1]/p[2]
|
en
| 0.999998
|
34017242
|
https://doi.org/10.3389/fnagi.2021.635896
|
[
"slow",
"speech",
"production",
"impaired",
"rigidity",
"handed",
"decline",
"verbal",
"communication",
"characterized"
] |
[
{
"code": "MB23.N",
"title": "Psychomotor retardation"
},
{
"code": "9C85.00",
"title": "Disorders of the saccadic pulse"
},
{
"code": "KA20.1Z",
"title": "Intrauterine growth restriction, unspecified"
},
{
"code": "8E0Y",
"title": "Other specified human prion diseases"
},
{
"code": "8C61",
"title": "Congenital myasthenic syndromes"
},
{
"code": "MA80.Z",
"title": "Speech or language disturbances, unspecified"
},
{
"code": "MB23.L",
"title": "Pressured speech"
},
{
"code": "6B60.5",
"title": "Dissociative neurological symptom disorder, with speech disturbance"
},
{
"code": "MB25.02",
"title": "Disorganised thinking"
},
{
"code": "MA81",
"title": "Speech dysfluency"
}
] |
=== ICD-11 CODES FOUND ===
|
[] |
MB23.N
|
Psychomotor retardation
|
Histologically, the tumor was an invasive ductal carcinoma with 4.8 × 2.0 cm in size. Several intraductal components and lymphatic invasion were observed. The stage was determined to be pT2N1M0 (stage IIB). Immunohistochemical examination revealed that the tumor was ER-positive, PgR-negative, and HER2-positive, with a Ki-67 index of 20% . Although bilateral breast cancer subtypes were similar, eventually, we judged the left breast cancer to be a second primary lesion as the reason for existence because of the intraductal components . Postoperatively, chemotherapy and targeted therapy (docetaxel, trastuzumab, and pertuzumab) and endocrine therapy (toremifene and leuprorelin) were administered. Fig. 2 Histopathological findings of bilateral breast cancers. The right breast cancer is shown in a – e , and the left breast cancer is shown in f – j : hematoxylin-eosin staining ( a , f ), ER ( b , g ), PgR ( c , h ), HER2 ( d , i ), and Ki-67 ( e , j ). Immunohistochemical staining results were ER, 5; PgR, 4; HER2, 2+; and Ki-67, 35.6% for the right lesion, and ER, 5; PgR, 1; HER2, 2+; and Ki-67, 20% for the left lesion Fig. 3 Macroscopic distribution of cancer in the left breast. A few intraductal components were present (dotted red line) in part of the invasive area (red line) Fig. 4 Intraductal metastatic carcinoma lesion in the left breast. Hematoxylin-eosin staining ( a ), ER ( b ), and CD10 ( c )
| 4.003906
| 0.886719
|
sec[1]/p[1]
|
en
| 0.999998
|
32833091
|
https://doi.org/10.1186/s40792-020-00966-y
|
[
"breast",
"cancer",
"intraductal",
"lesion",
"components",
"staining",
"tumor",
"invasive",
"carcinoma",
"stage"
] |
[
{
"code": "GB23",
"title": "Certain specified disorders of breast"
},
{
"code": "GB21.Z",
"title": "Inflammatory disorders of breast, unspecified"
},
{
"code": "GB21.Y",
"title": "Other specified inflammatory disorders of breast"
},
{
"code": "QF01.0",
"title": "Acquired absence of breast"
},
{
"code": "GB23.3",
"title": "Atrophy of breast"
},
{
"code": "2D4Z",
"title": "Unspecified malignant neoplasms of unspecified sites"
},
{
"code": "2C0Z",
"title": "Malignant neoplasms of intestine, unspecified"
},
{
"code": "2B5Z",
"title": "Malignant mesenchymal neoplasm of unspecified type"
},
{
"code": "2E2Z",
"title": "Malignant neoplasm metastasis, unspecified"
},
{
"code": "2D42",
"title": "Malignant neoplasms of ill-defined sites"
}
] |
=== ICD-11 CODES FOUND ===
|
[] |
GB23
|
Certain specified disorders of breast
|
A 71-year-old healthy female visited to our emergency department because of upper back pain for 2 days, and she presented with fever. She was admitted for further evaluation and management. She denied any traumatic injury and acupuncture and invasive therapeutics. At admission Day, her vital sign showed a blood pressure of 140/90 mm Hg, body temperature 38.2°C, respiratory rate 26/min and heart beat 110/min. Physical examination and important clinical findings showed the painful sensation over middle part of T spine area and there were no significant neurological deficit. The initial laboratory data revealed white cell count, 9300/mm 3 ; ESR, 86 mm/h; CRP, 0.28 mg/L and PCT, 0.03 ng/mL. The MRI of the spine showed that abnormal signal intensity in the T8–9 vertebrae. The T8–9 VO was impressed. S aureus (2/2 sets) was identified by using matrix-assisted laser desorption ionization-time of flight mass spectrometry (bioMerieux, Hazlewood, Mo.) on 3 th admission day, and it was oxacillin susceptible strain according to the susceptibility test. Oxacillin susceptible S. aureus VO was suspected, and parenteral oxacillin 2000 mg every 4 hours was prescribed. She received the 42-days combination regimens without adverse and unanticipated events. The serial follow-up laboratory data normalized. She was discharged on 44 th admission days and was follow-up at outpatient department where she recovered well.
| 3.820313
| 0.98291
|
sec[1]/sec[1]/p[0]
|
en
| 0.999996
|
33120821
|
https://doi.org/10.1097/MD.0000000000022852
|
[
"oxacillin",
"department",
"spine",
"laboratory",
"data",
"aureus",
"susceptible",
"healthy",
"visited",
"emergency"
] |
[
{
"code": "MG51.10",
"title": "Penicillin resistant Streptococcus pneumoniae"
},
{
"code": "FB1Z",
"title": "Conditions associated with the spine, unspecified"
},
{
"code": "FA7Z",
"title": "Structural disorders of spine, unspecified"
},
{
"code": "FA9Z",
"title": "Inflammation of spine, unspecified"
},
{
"code": "LB73.2Z",
"title": "Structural developmental anomalies of spine, unspecified"
},
{
"code": "FA82",
"title": "Spinal stenosis"
},
{
"code": "QA00.C",
"title": "Laboratory examination"
},
{
"code": "MG71.Z",
"title": "Abnormal laboratory results, not elsewhere classified, unspecified"
},
{
"code": "MG71.Y",
"title": "Other specified abnormal laboratory results, not elsewhere classified"
},
{
"code": "QA0B",
"title": "Preprocedural examination"
}
] |
=== ICD-11 CODES FOUND ===
|
[] |
MG51.10
|
Penicillin resistant Streptococcus pneumoniae
|
Here, we present a child with an epileptic encephalopathy resulting from a homozygous variant in the CAD gene (c.98T > G.p.Met33Arg) whose seizures resolved and encephalopathy improved with TAU treatment. There is a growing body of work demonstrating that refractory epilepsy and encephalopathy are frequently encountered in patients with inborn errors of metabolism. 9 , 10 Furthermore, the effectiveness of therapies that are targeted to specific metabolic pathways has been well‐documented. 11 , 12 , 13 For example, Hunt et al. documented a case of neonatal epilepsy resolved by daily pyridoxine treatment, with genetic studies in the past 2 decades highlighting the involvement of ALDH7A1 and PROSC in pyridoxine‐dependent epilepsy. 14 , 15 Since then, various metabolic pathways involving biotin recycling, glucose transport, creatine synthesis, purine metabolism, and Coenzyme Q10 deficiency have been implicated in pediatric epilepsy syndromes, with substantial response to replacement therapies. 9 The success of uridine supplementation in the treatment of several cases of epileptic encephalopathy resulting from CAD mutations further demonstrates the impact of genetic‐based epilepsy therapies. Exome sequencing as well as targeted gene panels have been shown to play an increasingly important role in elucidating these rare and often de novo genetic variants underlying epileptic encephalopathies. 16 , 17
| 4.367188
| 0.689941
|
sec[3]/p[0]
|
en
| 0.999998
|
33249780
|
https://doi.org/10.1002/acn3.51257
|
[
"epilepsy",
"encephalopathy",
"epileptic",
"therapies",
"genetic",
"resulting",
"gene",
"resolved",
"that",
"metabolism"
] |
[
{
"code": "8A6Z",
"title": "Epilepsy or seizures, unspecified"
},
{
"code": "8A60.5",
"title": "Epilepsy due to injuries to the head"
},
{
"code": "8A61.1Z",
"title": "Genetic epileptic syndromes with onset in infancy, unspecified"
},
{
"code": "8A06.Y/8A6Z",
"title": "Myoclonus due to epilepsy"
},
{
"code": "8A61.40",
"title": "Reflex epilepsies"
},
{
"code": "8E47",
"title": "Encephalopathy, not elsewhere classified"
},
{
"code": "8D43.0Y&XM0ZH6",
"title": "Encephalopathy due to lead"
},
{
"code": "NA07.0Y",
"title": "Other specified concussion"
},
{
"code": "8D43.0Z",
"title": "Encephalopathy due to toxicity, unspecified"
},
{
"code": "NE80.3",
"title": "Other serum reactions"
}
] |
=== ICD-11 CODES FOUND ===
|
[] |
8A6Z
|
Epilepsy or seizures, unspecified
|
Blood cultures obtained at the time of arrival to the ED returned positive for S. intermedius growth. All intraoperative samples also grew S. intermedius. One intraoperative sample revealed polymicrobial growth that included S. intermedius, Streptococcus salivarius, Rothia mucilaginosa, and Rothia dentocariosa. Surgical pathology of the C4 and C5 vertebral tissue culture grew S. intermedius. The cerebrospinal fluid analysis showed pleocytosis, elevated protein, and decreased glucose but no organism growth. The infectious disease department was consulted at this time for antibiotic recommendations. Due to the growth of the same organism from blood and operative cultures originating from a deep neck space infection complicated by cervical epidural abscess and osteomyelitis in the setting of an IJV thrombosis, a diagnosis of LS secondary to S. intermedius was made. Culture sensitivity results eventually showed sensitivity to ceftriaxone and penicillin after which cefepime was converted to high dose intravenous ceftriaxone two grams every 12 hours with metronidazole 500 milligrams three times daily continued for anaerobic coverage. The decision to administer high dose ceftriaxone was made considering the possibility of meningeal infection. Repeat blood cultures drawn 48 hours later were negative. Of note, two-dimensional transthoracic echocardiography showed no evidence of cardiac valve vegetation.
| 3.978516
| 0.967285
|
sec[1]/p[8]
|
en
| 0.999994
|
32455086
|
https://doi.org/10.7759/cureus.7787
|
[
"intermedius",
"growth",
"blood",
"cultures",
"ceftriaxone",
"time",
"intraoperative",
"grew",
"rothia",
"culture"
] |
[
{
"code": "2F9Z",
"title": "Neoplasms of unknown behaviour of unspecified site"
},
{
"code": "MG44.1Z",
"title": "Lack of expected normal physiological development, unspecified"
},
{
"code": "5A61.3",
"title": "Growth hormone deficiency"
},
{
"code": "8C7Y",
"title": "Other specified primary disorders of muscles"
},
{
"code": "FB86.Z",
"title": "Disorders associated with bone growth, unspecified"
},
{
"code": "3C0Z",
"title": "Diseases of the blood or blood-forming organs, unspecified"
},
{
"code": "MF50.4Z",
"title": "Haematuria, unspecified"
},
{
"code": "MA12.1",
"title": "Finding of cocaine in blood"
},
{
"code": "MA12.4",
"title": "Finding of steroid agent in blood"
},
{
"code": "MA12.2",
"title": "Finding of hallucinogen in blood"
}
] |
=== ICD-11 CODES FOUND ===
|
[] |
2F9Z
|
Neoplasms of unknown behaviour of unspecified site
|
Case 4 (a bortezomib-based regimen intolerant case).The patient had a history of severe malabsorption syndrome, for which parenteral nutrition was started two years before. The patient presented with cachexia and depressive syndrome. Laboratory tests revealed nephrotic syndrome (proteinuria 8.2 g/24 h), Bence–Jones proteinuria associated with marked hypoprotidemia (serum albumin 1.2 g/dL and total serum proteins of 3.5 g/dL). She had an IgDλ M component with FLC λ 134 mg/L and FLC ratio 0.12. Tissue histological examination confirmed the clinical suspicion of amyloid deposits in the kidney, intestinal mucosa, and periombelical fat. Bone marrow showed a λ-restricted plasma cell clone (13%), and testing with Congo red staining was positive. No signs of cardiac or peripheral nervous system involvement were found. The patient was initially treated with bortezomib (1.3 mg/m 2 ), cyclophosphamide , and dexamethasone (160 mg/week), but the protocol was discontinued after the first cycle because of intolerance and the appearance of leukocytopenia. Treatment with DARA resulted in a rapid decrease in proteinuria (from 8.2 to 1.9 g/24 h at the 13th dose) with disappearance of Bence–Jones proteinuria and serum M component and normalization of FLC ratio. No infusion related reactions were observed. After the 13thinfusion, the patient was hospitalized for pneumonia and treatment was temporarily discontinued.
| 4.039063
| 0.976074
|
sec[1]/p[6]
|
en
| 0.999998
|
33050272
|
https://doi.org/10.3390/jcm9103232
|
[
"proteinuria",
"serum",
"bortezomib",
"bence",
"jones",
"component",
"discontinued",
"based",
"regimen",
"intolerant"
] |
[
{
"code": "MF96.Z",
"title": "Proteinuria, unspecified"
},
{
"code": "MF96.Y",
"title": "Other specified proteinuria"
},
{
"code": "GB4Y",
"title": "Other specified glomerular diseases"
},
{
"code": "MF96.0",
"title": "Orthostatic proteinuria"
},
{
"code": "JA22.0",
"title": "Gestational proteinuria without hypertension"
},
{
"code": "NE80.3",
"title": "Other serum reactions"
},
{
"code": "5D0Y",
"title": "Other specified metabolic disorders"
},
{
"code": "5B91.0",
"title": "Hypercalcaemia"
},
{
"code": "4A84.Y",
"title": "Other specified anaphylaxis"
},
{
"code": "5C50.F2",
"title": "Homocarnosinosis"
}
] |
=== ICD-11 CODES FOUND ===
|
[] |
MF96.Z
|
Proteinuria, unspecified
|
All the operations were performed with a fixed plant posterior-stabilized total knee prosthesis in a medial parapatellar approach. A general anesthetic with an adductor canal block was used for all the patients . And appropriate perioperative parenteral antibiotics were administered for infection prophylaxis. High-pressure pulsatile lavage was used to clean the bone surfaces and soft tissues. Before wound closure, all knees received an intraarticular injection of a cocktail containing ropivacaine, morphine, and ketorolac to enhance postoperative analgesia without drain insertion. Elastic compressive dressing was also used. Each patient received the same perioperative regimen: tranexamic acid (TXA), pain control and rehabilitation. All patients received 1-g intravenous tranexamic acid 30 min before skin incision, and a solution of 1-g tranexamic acid in 50-mL normal saline solution was injected into knee articular cavity after closure of the capsule . Multimodal postoperative pain management and accelerated physical therapy were performed as previously described. Low molecular weight heparin was administered subcutaneously 12 h after the operation and was continued for 2 weeks for thrombosis prophylaxis. After recovery from anesthesia, quadriceps femoris muscle isometric contraction was immediately started, and rehabilitation began on the first postoperative day, including muscle power training.
| 3.994141
| 0.372559
|
sec[1]/sec[1]/p[0]
|
en
| 0.999998
|
32843003
|
https://doi.org/10.1186/s12891-020-03595-6
|
[
"used",
"postoperative",
"tranexamic",
"acid",
"knee",
"patients",
"perioperative",
"prophylaxis",
"closure",
"pain"
] |
[
{
"code": "QE11.Z",
"title": "Hazardous drug use, unspecified"
},
{
"code": "6C4Z",
"title": "Disorders due to substance use, unspecified"
},
{
"code": "QE11.3",
"title": "Hazardous use of cocaine"
},
{
"code": "QE11.2",
"title": "Hazardous use of sedatives, hypnotics or anxiolytics"
},
{
"code": "QE11.1",
"title": "Hazardous use of cannabis"
},
{
"code": "NE81.0Z",
"title": "Haemorrhage or haematoma of other or unspecified site complicating a procedure, not elsewhere classified"
},
{
"code": "NE81.3",
"title": "Postsurgical leak"
},
{
"code": "QF00",
"title": "Acquired absence of limb"
},
{
"code": "DB30.2",
"title": "Adhesions of large intestine with obstruction"
},
{
"code": "MG30.21",
"title": "Chronic postsurgical pain"
}
] |
=== ICD-11 CODES FOUND ===
|
[] |
QE11.Z
|
Hazardous drug use, unspecified
|
Idiopathic dilatation of the pulmonary artery (IDPA) is a rare condition characterized by enlargement of the pulmonary artery in the absence of an obvious trigger with an incidence of 1 per 14,000 autopsies . The diagnostic criteria for IDPA are (1) diastolic change of the pulmonary artery mainstem; (2) no abnormal shunt inside or outside the heart; (3) no lesions of the arteries that could cause histological changes, such as syphilis and arteriosclerosis; (4) no chronic heart or lung disease; and (5) normal right ventricular pressure. Generally, a patient who meets all the above-mentioned criteria is diagnosed with IDPA. Dilatation of the pulmonary artery may be secondary to an underlying disorder, such as congenital heart disease (including patent ductus arteriosus), chronic heart failure, chronic respiratory disease, vasculitis, Marfan syndrome, and trauma. Given the potential complications associated with IDPA, such as rupture of the pulmonary artery , pulmonary valve insufficiency , and myocardial ischemia due to displacement of the coronary arteries , surgery is a valid option for the treatment of this condition. Although there are several reports on surgery for dilated pulmonary arteries, few have explored the feasibility of anesthetic management for IDPA . We present a case of IDPA complicated by an extracardiac abnormality (Kommerell’s diverticulum), and discuss the management of IDPA.
| 4.230469
| 0.891113
|
sec[0]/p[0]
|
en
| 0.999996
|
33398467
|
https://doi.org/10.1186/s40981-020-00404-w
|
[
"pulmonary",
"idpa",
"artery",
"heart",
"arteries",
"dilatation",
"criteria",
"idiopathic",
"rare",
"characterized"
] |
[
{
"code": "CB40.Y",
"title": "Other specified diseases of the respiratory system"
},
{
"code": "LA75.1",
"title": "Agenesis of lung"
},
{
"code": "CA40.Z",
"title": "Pneumonia, organism unspecified"
},
{
"code": "CB41",
"title": "Respiratory failure"
},
{
"code": "NB32.3Y",
"title": "Other injury of lung"
},
{
"code": "BD5Z",
"title": "Diseases of arteries or arterioles, unspecified"
},
{
"code": "BD52",
"title": "Certain specified disorders of arteries or arterioles"
},
{
"code": "BD52.3",
"title": "Rupture of artery"
},
{
"code": "BD52.2",
"title": "Stricture of artery"
},
{
"code": "BD40.Z",
"title": "Atherosclerotic chronic arterial occlusive disease, unspecified"
}
] |
=== ICD-11 CODES FOUND ===
|
[] |
CB40.Y
|
Other specified diseases of the respiratory system
|
He was transplanted after seven months of waitlist time. Other than the requirement of multiple magnesium and potassium replacements to maintain serum levels, there were no additional anesthetic or operative management issues related to his GS. Subsequent echocardiograms revealed normal graft function. Posttransplant, the use of tacrolimus in the setting of his underlying GS elicited recalcitrant hypomagnesemia. Postoperative course was complicated by ventricular tachycardia without evidence of rejection, reperfusion injury, or coronary artery abnormality. A lidocaine infusion was initiated and subsequently transitioned to enteral amiodarone. Additionally, he developed significant cramping and tetany in his extremities limiting mobility, which improved with higher serum magnesium levels. A magnesium infusion treated both unique postoperative symptoms. Transition to enteral magnesium was challenging with high stool output and resultant metabolic acidosis. Ultimately, he was discharged home 79 days posttransplant (289 hospitalization days including waitlist time) on a regimen of magnesium oxide, magnesium chloride, sodium bicarbonate, and spironolactone, in addition to amiodarone, enalapril, and immunosuppression with steroids, tacrolimus, and mycophenolate mofetil. His neurologic examination continued to improve throughout hospitalization, and at discharge, he had only mild right-sided weakness.
| 3.851563
| 0.97998
|
sec[0]/p[2]
|
en
| 0.999997
|
32645769
|
https://doi.org/10.1177/2150135120912227
|
[
"magnesium",
"waitlist",
"time",
"serum",
"posttransplant",
"tacrolimus",
"postoperative",
"infusion",
"enteral",
"amiodarone"
] |
[
{
"code": "5C64.41",
"title": "Hypomagnesaemia"
},
{
"code": "5C64.4Z",
"title": "Disorders of magnesium metabolism, unspecified"
},
{
"code": "GB90.4Y",
"title": "Other specified renal tubular function disorders"
},
{
"code": "MA18.1",
"title": "Abnormal level of blood mineral"
},
{
"code": "8D40.2",
"title": "Myopathy due to nutritional deficiency"
},
{
"code": "PL13.52",
"title": "Incorrect timing of drug or medicament, as mode of injury"
},
{
"code": "QF2A",
"title": "Difficulty or need for assistance with community participation"
},
{
"code": "MF50.1",
"title": "Pollakiuria"
},
{
"code": "JA25.3",
"title": "Eclampsia, time period unspecified"
},
{
"code": "KD3B.Z",
"title": "Unspecified time of fetal death, cause not specified"
}
] |
=== ICD-11 CODES FOUND ===
|
[] |
5C64.41
|
Hypomagnesaemia
|
In January 2015, she presented with a 3-month history of worsening chest pain and rapid bilateral axillary lymph node enlargement. On examination, there was a lymph node of 2 cm diameter on each side of the armpit. On the basis of biopsy result, left axillary lymph node was GS. The immunohistochemistry examination showed bcl-2(+), CD34(+), CD117(+), MPO(+), Lysonzyme(+), TDT(+), Ki67(+5∼10%), CD3(−), CD5(−), CD20(−), P53(−), CD10(−), CD68(−), CD30(−), CD(79), Bcl-6(−), CD21(−), CD56 (−), CD15 (−), ALK (−), CyclinD1 (−), Mum-1 (−), CD23 (−), CD38(−). Peripheral blood smear, bone marrow examinations, and flow cytometry of bone marrow were normal. Conventional cytogenetics of bone marrow revealed a normal karyotype of 46, XX , but PML/RARA fusion gene by reverse transcription-polymerase chain reaction (RT-PCR) was 0.019% . In addition, PML/RARA fusion signal by FISH was positive for lymph node . Computed tomography (CT) scan of chest revealed the increase of bone density in the body of sternum . She received DA regimen (2 cycles) after which no enlarged lymph nodes were found by ultrasonography, and then received another DA, EA (etoposide70 mg/m 2 /day, d1∼4; cytarabine 150 mg/m 2 /day, d1∼7) regimens, and ATRA (25 mg/m 2 /day, d1∼28, 2 cycles). She received prophylactic intrathecal chemotherapy (dexamethasone 10 mg, cytarabine 30 mg, methotrexate 15 mg) during induction and consolidation therapy.
| 4.03125
| 0.973145
|
sec[1]/p[2]
|
en
| 0.999996
|
33832087
|
https://doi.org/10.1097/MD.0000000000025257
|
[
"lymph",
"node",
"bone",
"marrow",
"chest",
"axillary",
"rara",
"fusion",
"cycles",
"cytarabine"
] |
[
{
"code": "BD9Z",
"title": "Disorders of lymphatic vessels or lymph nodes, unspecified"
},
{
"code": "BD90.Z",
"title": "Lymphadenitis, unspecified"
},
{
"code": "BD90.Y",
"title": "Other specified lymphadenitis"
},
{
"code": "BD9Y",
"title": "Other specified disorders of lymphatic vessels or lymph nodes"
},
{
"code": "MA01.Z",
"title": "Enlarged lymph nodes, unspecified"
},
{
"code": "FA20.0",
"title": "Seropositive rheumatoid arthritis"
},
{
"code": "MF30",
"title": "Breast lump or mass female"
},
{
"code": "BB40",
"title": "Acute or subacute infectious endocarditis"
},
{
"code": "FA0Z",
"title": "Osteoarthritis, unspecified"
},
{
"code": "FA85.10",
"title": "Localised central endplate defect"
}
] |
=== ICD-11 CODES FOUND ===
|
[] |
BD9Z
|
Disorders of lymphatic vessels or lymph nodes, unspecified
|
Carotid cavernous fistula (CCF) is an abnormal arteriovenous communication formed between carotid artery and its branches due to the influence of external factors such as trauma, or spontaneous and cavernous sinus, resulting in a series of clinical symptoms and signs associated with the blood stealing and increased venous pressure. The disease is typically caused by head trauma and skull base fracture and has an incidence rate of 1-2.5% in cases of head trauma . Traumatic CCF cases are characteristically unilateral, and those of bilateral traumatic carotid cavernous fistula (TCCF) are extremely rare. The clinical symptoms of CCF are associated with the direction of venous drainage. The most common etiology of ocular venous drainage is characterized by the presence of the “triple syndromes” typically associated with carotid cavernous fistula in a majority of the patients with intracranial vascular murmur, pulsatile exophthalmos, bulbar conjunctival congestion, or edema. Ocular venous drainage will have “triple syndrome” manifested by intracranial vascular murmur, pulsating eyeball, conjunctival hyperemia, or edema, usually associated with carotid cavernous fistula . We believe that this case of bilateral CCF with pertinent symptoms in only one eye is very rare and combined with contralateral oculomotor nerve injury, it may be easily misdiagnosed and missed and, therefore, deserves our attention.
| 4.269531
| 0.774902
|
sec[0]/p[0]
|
en
| 0.999995
|
34736536
|
https://doi.org/10.1186/s41016-021-00265-x
|
[
"carotid",
"cavernous",
"fistula",
"associated",
"venous",
"trauma",
"drainage",
"typically",
"head",
"cases"
] |
[
{
"code": "8D88.Y",
"title": "Other specified autonomic neuropathies"
},
{
"code": "8B10.Y",
"title": "Other specified transient ischaemic attack"
},
{
"code": "2F9A",
"title": "Neoplasms of unknown behaviour of endocrine glands"
},
{
"code": "BD55",
"title": "Asymptomatic stenosis of intracranial or extracranial artery"
},
{
"code": "NA60.00",
"title": "Laceration of carotid artery, minor"
},
{
"code": "9C81.4",
"title": "Cavernous sinus syndromes"
},
{
"code": "GB06.Y",
"title": "Other specified disorders of penis"
},
{
"code": "LA90.10",
"title": "Macrocystic lymphatic malformation"
},
{
"code": "8B22.43",
"title": "Carotid cavernous fistula"
},
{
"code": "NA07.Y",
"title": "Other specified intracranial injury"
}
] |
=== ICD-11 CODES FOUND ===
|
[] |
8D88.Y
|
Other specified autonomic neuropathies
|
Systemic corticosteroids have been accepted as the gold standard treatment for ameliorating the clinical symptoms of DRESS. However, they need to be tapered over 6–8 weeks to prevent the relapse of various symptoms . The usage of intravenous immunoglobulin (IVIG) for patients with life-threatening signs such as renal failure or respiratory failure has also been recommended . Meanwhile, some authors have reported the beneficial effects of the concomitant use of N-acetyl cysteine because of its detoxifying capabilities . Alexander et al. reported a dramatic, sustained clinical response to therapeutic plasma exchange after a steroid treatment failed in a pediatric case of DRESS associated with either lamotrigine or bupropion, leading to multiorgan involvement and life-threatening complications of respiratory failure and cardiac arrest . Our patient presented fever and dyspnea initially, and her symptoms progressed to pulmonary insufficiency requiring non-invasive positive pressure ventilator support. Furthermore, our patient developed jaundice with severe liver dysfunction, and the immunosuppressant drugs of mycophenolate and tacolimus were even prescribed after steroid therapy due to hepatic failure. The use of immunosuppressant drugs for DRESS has never previously been reported in the literature. Our patient may thus be the first patient with a case of DRESS treated with immunosuppressant drugs.
| 4.164063
| 0.89502
|
sec[2]/p[5]
|
en
| 0.999998
|
34828776
|
https://doi.org/10.3390/children8111063
|
[
"dress",
"failure",
"immunosuppressant",
"drugs",
"life",
"threatening",
"respiratory",
"steroid",
"systemic",
"corticosteroids"
] |
[
{
"code": "EH65",
"title": "DRESS syndrome"
},
{
"code": "QB85",
"title": "Attention to surgical dressings, drains or sutures"
},
{
"code": "QF24",
"title": "Difficulty or need for assistance with self-care"
},
{
"code": "DA96.05",
"title": "Intestinal failure"
},
{
"code": "MG4A",
"title": "Multi organ failure"
},
{
"code": "CB41",
"title": "Respiratory failure"
},
{
"code": "CB41.2Z",
"title": "Respiratory failure, unspecified"
},
{
"code": "GB6Z",
"title": "Kidney failure, unspecified"
},
{
"code": "NE60",
"title": "Harmful effects of drugs, medicaments or biological substances, not elsewhere classified"
},
{
"code": "1E83",
"title": "Wart virus proliferation in immune-deficient states"
}
] |
=== ICD-11 CODES FOUND ===
|
[] |
EH65
|
DRESS syndrome
|
A 49-year-old male patient presented with a venous leg ulcer at his right lower leg . Relevant comorbidities included DM, HT, hyperlipidaemia, and a medical history of thrombosis of the right lower leg without acute signs of ischemia. Previous treatments with antiseptic dressing with silver nanoparticles for seven weeks were ineffective. Upon presentation, the wound dimensions were 5 cm in length, 3 cm in width, and 1.5 cm in depth. The wound consisted of 90% of granulation tissue, 5% slough, and 5% epithelialization tissue. Low levels of exudate (thin, water-like) were produced. Local signs of infection included pain, malodour, and delayed healing. A wound swab confirmed the presence of Enterococcus faecalis (resistant to trimethoprim + sulphonamide, neomycin, clindamycin, gentamicin; and sensitive to ampicillin, nitrofurantoin, bacitracin, ciprofloxacin, chloramphenicol). Pain level was 5 during the daytime and 8 during treatment. L-Mesitran ® Soft (MGH) was applied to the wound and L-Mesitran ® Foam was applied as a secondary dressing. Dressing changes were performed by the patient at home at 48 h intervals. Pain and malodour levels gradually decreased and after 14 days of treatment, the pain was absent during the daytime (VAS score of 0) and at level 1 during treatment. Only L-Mesitran ® Foam was applied. The wound was completely healed after 17 days of MGH treatment without complications .
| 3.927734
| 0.981445
|
sec[1]/sec[8]/p[0]
|
en
| 0.999997
|
34438968
|
https://doi.org/10.3390/antibiotics10080918
|
[
"wound",
"pain",
"dressing",
"mesitran",
"included",
"without",
"tissue",
"malodour",
"daytime",
"foam"
] |
[
{
"code": "MD11.5",
"title": "Dyspnoea"
},
{
"code": "NB32.3Z",
"title": "Injury of lung, unspecified"
},
{
"code": "NB91.1Z",
"title": "Injury of liver, unspecified"
},
{
"code": "NA01.Z&XA9T94",
"title": "Temporal wound"
},
{
"code": "ND56.1",
"title": "Open wound of unspecified body region"
},
{
"code": "MG3Z",
"title": "Pain, unspecified"
},
{
"code": "8E43.Z",
"title": "Pain disorders, unspecified"
},
{
"code": "MG31.Z",
"title": "Acute pain, unspecified"
},
{
"code": "MG30.Z",
"title": "Chronic pain, unspecified"
},
{
"code": "FB56.2",
"title": "Myalgia"
}
] |
=== ICD-11 CODES FOUND ===
|
[] |
MD11.5
|
Dyspnoea
|
The proportion of bacterial infections in adult HLH is very small, and most of them are related to tuberculosis ( 4 ). In this case report, Klebsiella pneumoniae is a rare factor for secondary HLH, which has been reported only a few times ( 5 ). Klebsiella pneumoniae is a common opportunistic pathogen, when the host’s immune capacity decreases it can induce infection and abscesses in many locations. We know from some case reports that Klebsiella pneumoniae can cause a psoas abscess and spinal infection, and, in addition, patients with psoas abscesses usually have fever and back or hip pain, but both are very rare ( 6 , 7 ). Because of the following reasons, the possibility of abscesses caused by other reasons (such as autoantibodies against IL-12 or insufficient haploid function of GATA2) was minimal. First, the patient had been treated with meropenem before the second admission, all indicators improved, and the blood culture was negative when he was re-admitted. Second, it is almost impossible for patients to infect other bacteria outside the hospital. Third, the patient had no discomfort after abscess drainage. Thus, we concluded that it had been caused by the initial spread of Klebsiella pneumoniae , resulting in bilateral psoas abscesses and intraspinal abscess, but not relapse or aggravation of HLH. This suggested that, initially, the patient had HLH associated with Klebsiella pneumoniae .
| 4.007813
| 0.84375
|
sec[2]/p[0]
|
en
| 0.999998
|
34335586
|
https://doi.org/10.3389/fimmu.2021.684805
|
[
"klebsiella",
"pneumoniae",
"abscesses",
"that",
"psoas",
"abscess",
"this",
"rare",
"infection",
"patients"
] |
[
{
"code": "CA40.03",
"title": "Pneumonia due to Klebsiella pneumoniae"
},
{
"code": "1C4Y",
"title": "Other specified bacterial diseases"
},
{
"code": "1A0Y&XN7WL",
"title": "Colitis due to Klebsiella oxytoca"
},
{
"code": "MG50.55",
"title": "Polymyxin resistant Klebsiella pneumoniae"
},
{
"code": "MG50.51",
"title": "Fluoroquinolone resistant Klebsiella pneumoniae"
},
{
"code": "CA40.Z",
"title": "Pneumonia, organism unspecified"
},
{
"code": "CA40.Y",
"title": "Other specified pneumonia"
},
{
"code": "KB24",
"title": "Congenital pneumonia"
},
{
"code": "CA40.1Z",
"title": "Viral pneumonia, unspecified"
},
{
"code": "CA40.0Z",
"title": "Bacterial pneumonia, unspecified"
}
] |
=== ICD-11 CODES FOUND ===
|
[] |
CA40.03
|
Pneumonia due to Klebsiella pneumoniae
|
A 76-year-old male with a history of BCC and squamous cell carcinoma presented to clinic for Mohs micrographic surgery to extirpate a biopsy-proven BCC, two weeks after initial biopsy . Mohs surgery was performed with frozen sections, and the first stage revealed atypical basaloid cells surrounding atypical foamy cells with scalloped nuclei at the deep margin, concerning for SC . The atypical cells were cleared on the second stage, leaving a 2.2 x 1.4 cm surgical defect size. Though SC was favored, given the equivocal pathology on frozen section (BCC with adnexal differentiation vs SC), these slides were sent to dermatopathology for tumor confirmation. A dermatopathologist re-examined the initial biopsy slide, and confirmed nodular BCC alone to be present, as seen in Figure 1A , with morphologic features being distinct and different from the sebaceous carcinoma . The first Mohs stage was re-embedded for permanent sectioning and immunohistochemical staining. SC at the deep margin was confirmed as seen in Figures 1B - 1D , as well the presence of a separate focus of BCC extending into the superficial dermis, as seen in Figure 1B and Figure 1E . The dermatopathologist agreed with the diagnosis of sebaceous carcinoma and favored this to be a collision lesion rather than a contiguous BCC with sebaceous differentiation. The patient continues to follow up at six-month intervals for tumor surveillance.
| 4.007813
| 0.977539
|
sec[1]/p[0]
|
en
| 0.999996
|
34667690
|
https://doi.org/10.7759/cureus.18015
|
[
"carcinoma",
"mohs",
"biopsy",
"stage",
"atypical",
"cells",
"sebaceous",
"frozen",
"margin",
"favored"
] |
[
{
"code": "2D41",
"title": "Unspecified carcinoma of unspecified site"
},
{
"code": "2C3Y",
"title": "Other specified malignant neoplasms of skin"
},
{
"code": "2E6Z",
"title": "Carcinoma in situ of unspecified site"
},
{
"code": "2C25.5",
"title": "Unspecified malignant epithelial neoplasm of bronchus or lung"
},
{
"code": "2C90.Y",
"title": "Other specified malignant neoplasms of kidney, except renal pelvis"
},
{
"code": "8A84.Y",
"title": "Other specified secondary headache"
},
{
"code": "JA85.Y",
"title": "Maternal care for other specified fetal abnormality or damage"
},
{
"code": "KD39.3",
"title": "Fetus or newborn affected by complications of fetal surgery"
},
{
"code": "PK81.5",
"title": "Biopsy procedure, not elsewhere classified, associated with injury or harm in therapeutic use"
},
{
"code": "PK81.4",
"title": "Bone marrow aspiration or biopsy associated with injury or harm in therapeutic use"
}
] |
=== ICD-11 CODES FOUND ===
|
[] |
2D41
|
Unspecified carcinoma of unspecified site
|
At that point, he was wheelchair dependent, having spastic quadriparesis with continued spells of seizures despite the therapy with high doses of levetiracetam and clobazam. The patient was continued on clonidine, baclofen, and gabapentin. We started him on a trial of high dose biotin (5 mg/kg/day divided three times a day) and thiamine (100 mg three times a day) for suspected biotin-thiamine-responsive basal ganglia disease (BTBGD) despite the absence of pathogenic variants in the whole-exome sequence (WES) result given the devastating neurologic impairment and low side effect profile of biotin and thiamine. The caregiver reported a significant improvement in awareness, non-verbal communication, spontaneous movement, and spasticity. A mitochondrial genome panel tested was unremarkable. A follow-up brain MRI, almost a year after, did not show new brain lesions; however, interval development of cystic encephalomalacia was noted in the bilateral posterior frontal lobe, insular cortex, and bilateral thalami, and ventricles were noted to be more enlarged in size . Moreover, volume loss was noted in the left hippocampus and body the of corpus callosum . A repeated anti-NMDA IgG antibodies test in serum was positive a year after the initial presentation. Currently, the patient is three years old with stable neurologic impairment, seizure control, and unreported relapse since the initial presentation.
| 3.955078
| 0.981934
|
sec[1]/p[2]
|
en
| 0.999996
|
34692314
|
https://doi.org/10.7759/cureus.18103
|
[
"biotin",
"three",
"thiamine",
"despite",
"times",
"neurologic",
"impairment",
"brain",
"that",
"point"
] |
[
{
"code": "5B5G",
"title": "Biotin deficiency"
},
{
"code": "4B4Y",
"title": "Other specified diseases of the immune system"
},
{
"code": "5C63.Y",
"title": "Other specified disorders of vitamin or non-protein cofactor absorption or transport"
},
{
"code": "1D45",
"title": "Sandfly fever"
},
{
"code": "PA09",
"title": "Unintentional land transport traffic event injuring an occupant of a low powered passenger vehicle"
},
{
"code": "LD50.Y",
"title": "Other specified number anomalies of chromosome X"
},
{
"code": "PA19",
"title": "Unintentional land transport nontraffic event injuring an occupant of a low powered passenger vehicle"
},
{
"code": "NA82.4",
"title": "Multiple fractures of ribs"
},
{
"code": "5B5A.Z",
"title": "Vitamin B1 deficiency, unspecified"
},
{
"code": "5B5A.00",
"title": "Dry beriberi"
}
] |
=== ICD-11 CODES FOUND ===
|
[] |
5B5G
|
Biotin deficiency
|
The mechanism of this patient’s electrolyte abnormalities is unproven, however, it most likely involves the calcium-parathyroid-vitamin D axis. Denosumab acts as a RANK-L ligand antibody, preventing osteoclast activation and the resulting bone resorption . As a result, calcium remained bound in the patient’s bones rather than in the serum, causing the parathyroid gland to respond to this perceived hypocalcemia by secreting abnormally high levels of PTH . PTH normally results in increased calcium reabsorption and phosphorus excretion by the kidneys; however, this patient also had a vitamin D deficiency, potentially secondary to elevated FGF23 and resulting inhibition of 1-alpha hydroxylase which typically activates inactive 25-OH vitamin D into active 1,25-OH vitamin D. As a result of this diminished vitamin D and decreased bone resorption, the patient was excreting phosphorus more than usual and was unable to maintain normal levels of calcium or phosphorus . This mechanism explains why repletion was initially unsuccessful as the denosumab continued to exert its effect on bone. However, as the denosumab was slowly eliminated, calcium was able to leave the bone, resulting in decreased PTH. Moreover, vitamin D repletion assisted in the reabsorption of both calcium and phosphorus by the kidneys. Therefore, the patient was successfully able to maintain the electrolyte levels with adequate repletion.
| 4.3125
| 0.752441
|
sec[2]/p[1]
|
en
| 0.999995
|
PMC8812272
|
https://doi.org/10.7759/cureus.20928
|
[
"calcium",
"vitamin",
"this",
"bone",
"phosphorus",
"however",
"denosumab",
"resulting",
"repletion",
"mechanism"
] |
[
{
"code": "5B5K.1Z",
"title": "Calcium deficiency, unspecified"
},
{
"code": "5B91.0",
"title": "Hypercalcaemia"
},
{
"code": "5B5K.1Y",
"title": "Other specified calcium deficiency"
},
{
"code": "5C64.5",
"title": "Disorders of calcium metabolism"
},
{
"code": "FB40.Y",
"title": "Other specified tenosynovitis"
},
{
"code": "5B7Z",
"title": "Unspecified undernutrition"
},
{
"code": "5B90.Z",
"title": "Unspecified vitamin excesses"
},
{
"code": "5B55.Z",
"title": "Vitamin A deficiency, unspecified"
},
{
"code": "6C4H.1Z",
"title": "Harmful pattern of use of non-psychoactive substances, unspecified"
},
{
"code": "5B90.Y",
"title": "Other specified vitamin excess"
}
] |
=== ICD-11 CODES FOUND ===
|
[] |
5B5K.1Z
|
Calcium deficiency, unspecified
|
An 81-year-old woman with a known history of hypertension and chronic obstructive pulmonary disease presented to the emergency department with unrelenting left-sided facial pain and was ultimately admitted for severe TN. Her pain had started four months ago. She described it as a stabbing sensation that started from her left ear and radiated down her left jaw and tongue. She experienced around 30 painful episodes throughout each day, with each episode lasting a few minutes. Her pain mostly occurred sporadically, but she did notice that chewing triggered the onset of her pain. She rated her pain a 10/10 during her episodes with no relieving factors. She also had associated dysphagia, generalized weakness, and a 25-pound weight loss over the past four months. She had undergone a prior MRI head with pre- and post-gadolinium-based contrast agents (GBCA) two months ago, which had not shown any significant findings. Her initial medication regimen had consisted of carbamazepine, pregabalin, and acetaminophen/codeine, which had been later switched to trials of lamotrigine, pregabalin, bupropion, and fentanyl patches. Pharmacologic therapy failed to provide any relief. Initial physical exam was significant for hypotension with systolic blood pressures in the 90s. Her labs were significant for hypokalemia (2.3 mmol/L), hypomagnesemia (1.5 mg/dL), hypoalbuminemia (2.0 g/dL), and leukocytosis (16,000/mm³).
| 3.884766
| 0.983398
|
sec[1]/p[0]
|
en
| 0.999998
|
PMC8765329
|
https://doi.org/10.7759/cureus.20514
|
[
"pain",
"four",
"that",
"episodes",
"which",
"pregabalin",
"known",
"hypertension",
"obstructive",
"pulmonary"
] |
[
{
"code": "MG3Z",
"title": "Pain, unspecified"
},
{
"code": "8E43.Z",
"title": "Pain disorders, unspecified"
},
{
"code": "MG31.Z",
"title": "Acute pain, unspecified"
},
{
"code": "MG30.Z",
"title": "Chronic pain, unspecified"
},
{
"code": "FB56.2",
"title": "Myalgia"
},
{
"code": "LD50.Y",
"title": "Other specified number anomalies of chromosome X"
},
{
"code": "NA82.4",
"title": "Multiple fractures of ribs"
},
{
"code": "BA4Z",
"title": "Acute ischaemic heart disease, unspecified"
},
{
"code": "8A80.Z",
"title": "Migraine, unspecified"
},
{
"code": "QA76",
"title": "Medication or substance that is known to be an allergen without injury or harm"
}
] |
=== ICD-11 CODES FOUND ===
|
[] |
MG3Z
|
Pain, unspecified
|
After treatment, the patient's oxygenation improved, the peak airway pressure decreased, and the pulmonary static compliance increased (Table 1 ). Leukocytes, high procalcitonin, C-reactive protein, and interleukin-6decreased to normal. On day 10, the patient stopped prone ventilation, adjusted the depth of sedation to shallow sedation (CPOT 0, RASS –2 to 0), and adjusted the ventilator mode to synchronized intermittent mandatory ventilation (SIMV PC 10cmH 2 0, PS 10cmH 2 0, PEEP 8cmH 2 0, F12 breaths min –1 , FiO 2 40%). The changes of ECMO and ventilator parameters are shown in Table 1 . ECMO support was stopped on day 12, tracheal intubation was removed after 2 days, and sequential transnasal high flow oxygen inhalation was applied. The patient was transferred to the respiratory department on day 15 and discharged after 12 days. During the treatment, we intermittently used bedside chest X-ray and CT scans of chest to understand the pulmonary lesions. Bedside chest X-ray showed that the right lung lesions are less severe than before on day 5 and 8 . After the patient's condition was slightly stable, we began to perform CT scans of chest intermittently, and the results showed that the lung lesions gradually improved . During the treatment, the patient's tolerance and compliance were good, and no adverse events occurred. Two months later, follow-up showed that the patient was in good condition.
| 3.810547
| 0.970215
|
sec[1]/p[4]
|
en
| 0.999997
|
PMC8735805
|
https://doi.org/10.1097/MD.0000000000028482
|
[
"chest",
"lesions",
"that",
"improved",
"pulmonary",
"compliance",
"ventilation",
"adjusted",
"sedation",
"ventilator"
] |
[
{
"code": "CB7Z",
"title": "Diseases of the respiratory system, unspecified"
},
{
"code": "CB27",
"title": "Pleural effusion"
},
{
"code": "CA44",
"title": "Pyothorax"
},
{
"code": "MD30.Z",
"title": "Chest pain, unspecified"
},
{
"code": "NA80.Y&XJ1C6",
"title": "Thoracic haematoma"
},
{
"code": "FA5Z",
"title": "Arthropathies, unspecified"
},
{
"code": "FC0Z",
"title": "Diseases of the musculoskeletal system or connective tissue, unspecified"
},
{
"code": "ME60.Z",
"title": "Skin lesion of unspecified nature"
},
{
"code": "MD41",
"title": "Clinical findings on diagnostic imaging of lung"
},
{
"code": "GC2Z&XA6KU8",
"title": "Disease of kidney, not elsewhere classified"
}
] |
=== ICD-11 CODES FOUND ===
|
[] |
CB7Z
|
Diseases of the respiratory system, unspecified
|
ECG revealed normal sinus rhythm with Q waves in leads II, III, and aVF, and ST segment depression in V5-V6 . Chest radiography and CT revealed a CTR of 72% and severe calcified coronary arteries . UCG revealed poor left ventricular function (LVDd/Ds, 56/43 mm; LVEF, 44%; inferoposterior wall hypokinesis; and mild MR with RVP of 29 mmHg). Cineangiography 4 years ago had shown only 25% stenosis in the proximal LAD; at admission, it revealed 90% stenosis in the same proximal LAD, 99% stenosis in the proximal LCX, and 95% stenosis in the posterolateral branch (PL) of LCX . He had low blood pressure during dialysis and underwent emergency surgery (Euro II risk score 40.7%; STS risk score, 33.2%). Fig. 4 Preoperative electrocardiogram in Case 2. Electrocardiogram (ECG) showing normal sinus rhythm with Q waves in leads II, III, and aVF, and ST segment depression in V5-V6 Fig. 5 Preoperative chest radiography and computed tomography in Case 2. Chest radiography and computed tomography (CT) showing a CTR of 72% and severely calcified coronary arteries Fig. 6 Coronary angiogram in Case 2. a Four years previously. A right coronary artery hypoplastic artery with no stenosis in the left anterior descending artery (LAD) and left circumflex artery (LCX). b At admission. The images show 90% stenosis in the proximal LAD, 99% stenosis in the proximal LCX, and 90% stenosis in the posterolateral branch of the LCX
| 3.984375
| 0.944336
|
sec[1]/sec[1]/p[2]
|
en
| 0.999998
|
34082711
|
https://doi.org/10.1186/s12872-021-02076-5
|
[
"stenosis",
"coronary",
"artery",
"chest",
"radiography",
"sinus",
"rhythm",
"waves",
"leads",
"segment"
] |
[
{
"code": "DB30.Y",
"title": "Other specified obstruction of large intestine"
},
{
"code": "BB60.Z",
"title": "Mitral valve stenosis, unspecified"
},
{
"code": "BD52.2",
"title": "Stricture of artery"
},
{
"code": "FA82",
"title": "Spinal stenosis"
},
{
"code": "DA40.0",
"title": "Gastric outlet obstruction"
},
{
"code": "BA8Z",
"title": "Diseases of coronary artery, unspecified"
},
{
"code": "BA4Z",
"title": "Acute ischaemic heart disease, unspecified"
},
{
"code": "BA41.Z",
"title": "Acute myocardial infarction, unspecified"
},
{
"code": "BA5Z",
"title": "Chronic ischaemic heart disease, unspecified"
},
{
"code": "LA8C.2",
"title": "Congenital coronary arterial fistula"
}
] |
=== ICD-11 CODES FOUND ===
|
[] |
DB30.Y
|
Other specified obstruction of large intestine
|
On day 3, yeast-like cells grew in an aerobic blood culture bottle obtained on admission . The serum beta-D glucan measured was 10.8 pg/mL (Wako Pure Chemical Industries, Ltd., Tokyo, Japan; cut-off value, < 11 pg/mL). Retinal examination revealed no evidence of endophthalmitis. Micafungin was added at a dose of 100 mg every 24 h against suspected candidemia after an additional set of blood cultures, and the CVC was removed. The patient became afebrile after initiating systemic antifungal therapy. The colony appeared rough and pale orange in color. The colony’s India ink stain did not show a zone of clearance of ink particles, like Cryptococcus . Colonies were tested on CHROMagar Candida medium (Kanto Chemical Co., Inc., Tokyo, Japan) using matrix-assisted laser desorption/ionization time-of-flight mass spectrometry (MALDI-TOF MS, the MALDI Biotyper Reference Library Ver.4.0.0.1, Filamentous Fungi Library Ver.1.0; Bruker Daltoniks, Germany), identifying them as Aureobasidium pullulans (score 1.919). On day 6, the high fever recurred. Micafungin was stopped and liposomal-amphotericin B (L-AMB) was started (3 mg/kg/day) for Cryptococcus albidus owing to a 90% probability of identification using the VITEK2 COMPACT Microbial Detection System (version 8.01 database: SYSMEX bioMerieux Co., Ltd., Tokyo, Japan). Fig. 1 Microscopy images of Aureobasidium melanigenum Gram stain of positive blood cultures
| 4.070313
| 0.94873
|
sec[1]/p[2]
|
en
| 0.999998
|
PMC8981616
|
https://doi.org/10.1186/s12879-022-07310-9
|
[
"blood",
"tokyo",
"japan",
"like",
"chemical",
"micafungin",
"cultures",
"colony",
"stain",
"cryptococcus"
] |
[
{
"code": "3C0Z",
"title": "Diseases of the blood or blood-forming organs, unspecified"
},
{
"code": "MF50.4Z",
"title": "Haematuria, unspecified"
},
{
"code": "MA12.1",
"title": "Finding of cocaine in blood"
},
{
"code": "MA12.4",
"title": "Finding of steroid agent in blood"
},
{
"code": "MA12.2",
"title": "Finding of hallucinogen in blood"
},
{
"code": "1C62.Z",
"title": "Human immunodeficiency virus disease without mention of tuberculosis or malaria, clinical stage unspecified"
},
{
"code": "1E32",
"title": "Influenza, virus not identified"
},
{
"code": "5C56.0Y",
"title": "Other specified sphingolipidosis"
},
{
"code": "8A44.1",
"title": "Adrenoleukodystrophy"
},
{
"code": "4A01.31",
"title": "DNA repair defects other than combined T-cell or B-cell immunodeficiencies"
}
] |
=== ICD-11 CODES FOUND ===
|
[] |
3C0Z
|
Diseases of the blood or blood-forming organs, unspecified
|
Both leprosy and syphilis were considered in the clinical differential diagnosis of our patient’s infiltrative plaques. However, leprosy and syphilis may present a challenging differential diagnosis . Both diseases have polymorphous presentations and occasionally present unclear laboratory tests which hinder definitive clinical diagnosis of the presenting lesions . Syphilis,” the great imitator” , normally presents as a papulo-squamous rash, but in HIV positive patients it may present atypically as annular, nodular or granulomatous plaques . In fact, nodular and granulomatous syphilis has occasionally been clinically and/or histopathologically misdiagnosed as indeterminate or tuberculoid or borderline lepromatous leprosy especially in HIV infected patients . Given the aforementioned confusion in earlier cases even on histology, the weakly reactive RPR result and the initial improvement on ceftriaxone and doxycycline, syphilitic palmar keratoderma and ocular lesions remained a possible diagnosis in our patient. The initial improvement following the institution of ceftriaxone and doxycycline for the suspected syphilis was probably just due to rifampicin monotherapy for leprosy whilst the patient was receiving PTB treatment from his base hospital. Whilst this regimen is effective for early syphilis , it is not effective for leprosy. Of the tetracyclines, only minocycline is effective for leprosy .
| 4.1875
| 0.902832
|
sec[2]/p[1]
|
en
| 0.999998
|
34193098
|
https://doi.org/10.1186/s12890-021-01572-w
|
[
"leprosy",
"syphilis",
"present",
"effective",
"both",
"differential",
"plaques",
"occasionally",
"lesions",
"patients"
] |
[
{
"code": "1B20.Z",
"title": "Leprosy, unspecified"
},
{
"code": "1B20.3",
"title": "Complications of leprosy"
},
{
"code": "1B20.2",
"title": "Leprosy reactions"
},
{
"code": "8D83",
"title": "Autonomic nervous system disorder due to infection"
},
{
"code": "1B20.0",
"title": "Paucibacillary leprosy"
},
{
"code": "1A6Z",
"title": "Syphilis, unspecified"
},
{
"code": "1A60.Z",
"title": "Congenital syphilis, unspecified"
},
{
"code": "1A61.0",
"title": "Primary genital syphilis"
},
{
"code": "1A61.Z",
"title": "Early syphilis, unspecified"
},
{
"code": "QC90.2",
"title": "Contact with or exposure to infections with a predominantly sexual mode of transmission"
}
] |
=== ICD-11 CODES FOUND ===
|
[] |
1B20.Z
|
Leprosy, unspecified
|
This patient is a 54-year-old homeless man with a history of schizophrenia who presented to the emergency department in November 2020 after being found down with a penetrating injury to the left anterior chest. On arrival, his GCS was 3, and he was being ventilated via bag valve mask by EMS. He was promptly intubated for airway protection. His vital signs were stable. On further exposure, he had an approximately 3 cm stab wound to his left anterior chest, medial to the nipple line, at approximately the level of the 7 th rib. Otherwise, he was noted to have ecchymosis of the occiput and superficial lacerations to his right flank. Also concerning was a fixed and dilated left pupil. A pan CT scan revealed a left subdural hematoma with bilateral frontal hemorrhagic contusions and 5 mm of midline shift. Additionally, he was found to have a moderate left-sided pneumothorax and associated left anterior 7 th rib fracture for which he underwent chest tube placement with resolution of his pneumothorax . A cerebral oxygenation monitor and drainage device was placed by neurosurgery revealing an elevated opening pressure of 31 mmHg. At this time, there was also concern for possible diaphragmatic injury given the location of his stab wound and imaging findings; however, in the setting of his elevated intracranial pressures, he was deemed unsafe for the OR and admitted to the neuro ICU for further monitoring.
| 3.685547
| 0.985352
|
sec[2]/p[0]
|
en
| 0.999997
|
34395014
|
https://doi.org/10.1155/2021/5531557
|
[
"chest",
"this",
"injury",
"approximately",
"stab",
"wound",
"pneumothorax",
"homeless",
"schizophrenia",
"emergency"
] |
[
{
"code": "CB7Z",
"title": "Diseases of the respiratory system, unspecified"
},
{
"code": "CB27",
"title": "Pleural effusion"
},
{
"code": "CA44",
"title": "Pyothorax"
},
{
"code": "MD30.Z",
"title": "Chest pain, unspecified"
},
{
"code": "NA80.Y&XJ1C6",
"title": "Thoracic haematoma"
},
{
"code": "4A01.03",
"title": "Transient hypogammaglobulinaemia of infancy"
},
{
"code": "ND56.Z",
"title": "Unspecified injury to unspecified part of trunk, limb or body region"
},
{
"code": "ND37",
"title": "Unspecified multiple injuries"
},
{
"code": "NB91.Y&XA9607",
"title": "Injury of intestine"
},
{
"code": "ND56.4",
"title": "Injury of nerve of unspecified body region"
}
] |
=== ICD-11 CODES FOUND ===
|
[] |
CB7Z
|
Diseases of the respiratory system, unspecified
|
These nodules often require lung segmentectomy, even combined segmentectomy. We can perform 3D reconstruction of the lobes, bronchial vessels, and other anatomic structures to simulate segmentectomy preoperatively. Localisation by 3D-CTLR allows identification of safe surgical margins, especially for lung nodules located in the plane between segments or subsegments, and then allows the anatomic resection of the targeted segment to be minimised while meeting oncologic requirements . Fig. 6 Illustration of the localisation procedure in patient 5. A A computed tomography image showing a nodule (yellow arrow) in the right upper lobe. B Three-dimensional computed tomography lung reconstruction showing that the nodule is located in the upper lobe of the right lung, far from the pleura, but that the projection position of the nodule on the lung surface is approximately the middle of the upper and lower boundaries. Three-dimensional computed tomography bronchography and angiography suggest that the nodules are located at the apical segment of the right upper lobe but close to the posterior segment. C Localisation is based on the distance between the nodule and the upper and lower boundaries and a burn mark made with an electrotome. D The resected specimen after apical segmentectomy shows adequate surgical margins (arrow). The final histology confirmed this lesion to be minimally invasive adenocarcinoma
| 4.015625
| 0.555176
|
sec[1]/sec[1]/sec[1]/p[0]
|
en
| 0.999999
|
34565436
|
https://doi.org/10.1186/s13019-021-01666-w
|
[
"lung",
"segmentectomy",
"nodule",
"nodules",
"localisation",
"located",
"segment",
"computed",
"tomography",
"lobe"
] |
[
{
"code": "CB40.Y",
"title": "Other specified diseases of the respiratory system"
},
{
"code": "LA75.1",
"title": "Agenesis of lung"
},
{
"code": "CA40.Z",
"title": "Pneumonia, organism unspecified"
},
{
"code": "CB41",
"title": "Respiratory failure"
},
{
"code": "NB32.3Y",
"title": "Other injury of lung"
},
{
"code": "2E88",
"title": "Benign endometrial stromal nodule"
},
{
"code": "FA20.0",
"title": "Seropositive rheumatoid arthritis"
},
{
"code": "1F20.Z",
"title": "Aspergillosis, unspecified"
},
{
"code": "MF30",
"title": "Breast lump or mass female"
},
{
"code": "5A01.1",
"title": "Nontoxic single thyroid nodule"
}
] |
=== ICD-11 CODES FOUND ===
|
[] |
CB40.Y
|
Other specified diseases of the respiratory system
|
Failure to respond to appropriate antibiotics should prompt a broader differential diagnosis beyond bacterial infection in cases of persistent dermatologic lesions. Cutaneous blastomycosis specifically should be suspected in patients with frequent travel in the Midwestern United States, despite the absence of recent respiratory infections. Primary inoculation, although rare, should be considered in patients with significant history of outdoor activities. In our patient, the initial diagnosis was hinged on a bacterial infection with Enterococcus faecalis . The patient’s travel history could have prompted an earlier investigation into fungal etiologies. However, it was not until the patient failed to respond to antibiotics that a biopsy and fungal cultures were sent, resulting in significant delay in treatment despite extensive spread of lesions. In order to prevent misdiagnosis and delays in treatment, fungal etiologies should be high on the differential; histopathologic specimen and fungal cultures should be sent at the outset for accurate diagnosis and treatment. In cases of fungal infection particularly blastomycosis, histology is key as it helps differentiate blastomycosis from various other infections and malignancies that have a similar presentation. Likewise, thorough exposure history and knowledge of other mimickers is just as important in prompt diagnosis and treatment of blastomycosis.
| 4.21875
| 0.644531
|
sec[3]/p[0]
|
en
| 0.999998
|
PMC8881278
|
https://doi.org/10.7759/cureus.21634
|
[
"fungal",
"blastomycosis",
"infection",
"respond",
"antibiotics",
"prompt",
"differential",
"bacterial",
"cases",
"lesions"
] |
[
{
"code": "9A71&XN8AY",
"title": "Fungal keratitis"
},
{
"code": "DA25.11",
"title": "Fungal oesophageal ulcer"
},
{
"code": "DA60.7&XN8AY",
"title": "Fungal gastric ulcer"
},
{
"code": "DA63.62",
"title": "Fungal duodenal ulcer"
},
{
"code": "FA10.2",
"title": "Fungal infection of joint"
},
{
"code": "1F22",
"title": "Blastomycosis"
},
{
"code": "1F27.Z",
"title": "Cryptococcosis, unspecified"
},
{
"code": "1F2E.Z",
"title": "Paracoccidioidomycosis, unspecified"
},
{
"code": "1F2E.0",
"title": "Pulmonary paracoccidioidomycosis"
},
{
"code": "1F2E.Y",
"title": "Other specified paracoccidioidomycosis"
}
] |
=== ICD-11 CODES FOUND ===
|
[] |
9A71&XN8AY
|
Fungal keratitis
|
The infant was transferred to the general paediatric ward on Day 5 and discharged home on Day 26. Initially, he was treated with a regimen of basal and rapid insulin and then transitioned to an insulin pump. L‐Thyroxine treatment was stopped at 1 month with normalization of the thyroid balance (TSH 0.21 μUI/ml; T4 10.6 pg/ml); without any antibodies stating a transient acute central hypothyroidism; and diabetes was well balanced. Chromosomal analysis by CGH‐Array was normal and the search for fragile X syndrome was negative. The metabolic workup was unremarkable (mucopolysaccharides, urinary oligosaccharides, plasma lysosphingolipids and Gaucher disease). In addition, the mitochondrial DNA study was normal. He was treated with anticoagulants (antivitamin K: coumadin 2 mg per day) for 3 months. The brain MRI performed at 3 months showed partial repermeabilization of the superior sagittal sinus with persistence of a heterogeneous aspect of the sinus, seat of some linear images probably corresponding to residual filiform thrombi, a complete repermeabilization of the transverse sinuses and the torcular and cortical veins, and an appearance of FLAIR signal abnormalities of the periventricular white matter, probably sequelae of the abnormalities visualized in diffusion on the first MRI . The neurological examination returned to baseline, and he is followed up in a specialized neurodevelopment centre.
| 3.939453
| 0.979492
|
sec[1]/p[5]
|
en
| 0.999997
|
36722309
|
https://doi.org/10.1002/edm2.389
|
[
"treated",
"insulin",
"repermeabilization",
"sinus",
"probably",
"abnormalities",
"transferred",
"general",
"paediatric",
"ward"
] |
[
{
"code": "5A44",
"title": "Insulin-resistance syndromes"
},
{
"code": "5A4Y",
"title": "Other specified disorders of glucose regulation or pancreatic internal secretion"
},
{
"code": "QB51.5",
"title": "Presence of endocrine implants"
},
{
"code": "EF02.0",
"title": "Fat hypertrophy"
},
{
"code": "PK9C.2",
"title": "Other or unspecified medical devices associated with injury or harm, prosthetic or other implants, materials or accessory devices"
},
{
"code": "CA0A.Z",
"title": "Chronic rhinosinusitis, unspecified"
},
{
"code": "CA0Y&XA3523",
"title": "Nasal sinus obstruction"
},
{
"code": "CA0J.Y",
"title": "Other specified nasal polyp"
},
{
"code": "LB03.Y",
"title": "Other specified structural developmental anomalies of umbilical cord"
},
{
"code": "DA09.61",
"title": "Periapical abscess with sinus"
}
] |
=== ICD-11 CODES FOUND ===
|
[] |
5A44
|
Insulin-resistance syndromes
|
Unfortunately, the horseshoe-shaped object had sharp and jagged edges. Our primary concern was a vocal cord injury during retrieval. As such, we entertained several different extraction modalities, including flexible bronchoscopy, rigid bronchoscopy, and, as a last resort, cardiothoracic surgery. After extensive discussion, an initial attempt with a flexible bronchoscope was agreed upon. After intubation with an 8.0 endotracheal tube, a flexible bronchoscope was introduced into the airway. Initially, under direct visualization, toothed forceps were used to dislodge the dental device from the bronchial wall. The resultant minor bleeding was controlled by instilling chilled saline. After mobilization, the forceps were also used to "aim" the dental device so that the rounded shape would cross the vocal cords first. Unfortunately, the forceps were unable to provide enough grip to ensure safe retrieval. To ensure a tighter grip, a lasso was introduced and threaded over and around the dental device. Using the lasso, the dental device was pulled up to the bronchoscope. Then, simultaneously, the dental device, bronchoscope, and endotracheal tube were all removed. Initially, likely because of the size of the dental device, the patient did experience bronchospasm complicated by hypoxia, requiring reintubation. Fortunately, after steroid treatment, the patient was safely extubated and able to return home.
| 3.794922
| 0.974609
|
sec[1]/p[3]
|
en
| 0.999996
|
37323356
|
https://doi.org/10.7759/cureus.39074
|
[
"dental",
"device",
"bronchoscope",
"flexible",
"forceps",
"unfortunately",
"vocal",
"retrieval",
"bronchoscopy",
"endotracheal"
] |
[
{
"code": "DA07.3",
"title": "Disturbances in tooth formation"
},
{
"code": "DA09.61",
"title": "Periapical abscess with sinus"
},
{
"code": "QA00.8",
"title": "Dental examination"
},
{
"code": "DA08.0",
"title": "Dental caries"
},
{
"code": "DA08.4",
"title": "Deposits on teeth"
},
{
"code": "8A43.Z",
"title": "Neuromyelitis optica, unspecified"
},
{
"code": "QB5Z",
"title": "Presence of unspecified device"
},
{
"code": "PL12.0",
"title": "Structural device failure, as mode of injury or harm"
},
{
"code": "PL12.2",
"title": "Perforation or protrusion by device, as mode of injury or harm"
},
{
"code": "PL12.1",
"title": "Functional device failure, as mode of injury or harm"
}
] |
=== ICD-11 CODES FOUND ===
|
[] |
DA07.3
|
Disturbances in tooth formation
|
We present a patient with autosomal recessive osteopetrosis 8. The clinical features of the proband are typical of the ARO phenotype such as increased bone density, large skull, bone-in-bone, thrombocytopenia, hepatosplenomegaly, loss of vision and “sandwich vertebrae”. We identified a deletion-insertion variant, NC_000007.14:g.26263639_26335651delinsCA in the homozygous state on chromosome 7 by genome sequencing. Exome sequencing failed to identify the large deletion because it encompasses a large noncoding region including the intergenic region upstream of SNX10 , there showing that exome sequencing is not an ideal method for identifying large deletions in noncoding regions. Genome sequencing identified the exact breakpoints of the 72,012 bp long region encompassing the regulatory region of SNX10 . The parents are healthy carriers of the same variant. Initially, we thought that due to the deletion of the 5′-UTR and exon 1 region of SNX10, there might be no SNX10 transcript, but a study by Ye et al., 2015 showed severe osteopetrosis but no rickets in the osteoclast-specific SNX10 knockout. However, our patient showed a moderate phenotype without rickets. Further mRNA analysis revealed the presence of a lower amount of SNX10 transcript. Therefore, we hypothesize that the decreased amount of SNX10 transcript results in abnormal endosome sorting and vesicular transport to osteoclast bone lacunae.
| 4.226563
| 0.488525
|
sec[3]/p[0]
|
en
| 0.999998
|
36526684
|
https://doi.org/10.1038/s10038-022-01104-2
|
[
"region",
"bone",
"large",
"sequencing",
"deletion",
"that",
"transcript",
"osteopetrosis",
"phenotype",
"variant"
] |
[
{
"code": "ND56.0",
"title": "Superficial injury of unspecified body region"
},
{
"code": "ND56.Z",
"title": "Unspecified injury to unspecified part of trunk, limb or body region"
},
{
"code": "CA44",
"title": "Pyothorax"
},
{
"code": "FB3Z",
"title": "Disorders of muscles, unspecified"
},
{
"code": "ND56.8",
"title": "Traumatic amputation of unspecified body region"
},
{
"code": "FC0Z",
"title": "Diseases of the musculoskeletal system or connective tissue, unspecified"
},
{
"code": "FB84.Z",
"title": "Osteomyelitis or osteitis, unspecified"
},
{
"code": "FB80.Z",
"title": "Disorder of bone density or structure, unspecified"
},
{
"code": "FB86.11",
"title": "Hypertrophy of bone"
},
{
"code": "FB86.1Z",
"title": "Bone hyperplasias, unspecified"
}
] |
=== ICD-11 CODES FOUND ===
|
[] |
ND56.0
|
Superficial injury of unspecified body region
|
Severe Parkinsonism and AM after VPS was diagnosed. The therapy of Madopar (levodopa/benserazide) and Pramipexole was initiated. Over the next 2 weeks, the dose of Madopar and Pramipexole slowly increased to 550/137.5 mg/day and 1.125 mg/day, respectively. Nearly a month after initiation of treatment, the patient let out a long-lost weak cry leading to an emotional moment for the family. After that, he began to pronounce monosyllabic words and was able to stand and walk with assistance. A complete recovery of speech and motor skills was observed 2 months later. The patient could not recall the events he had experienced during the period of severe illness. At a 6-month follow-up post-treatment, head MRI scan showed no ventricular dilation. The regions of interest (ROI) of cortex (frontal lobe, parietal lobe, temporal lobe, occipital lobe and cingulate gyrus) and subcortex (putamen, caudate nucleus, thalamus and midbrain) were delineated on 18 F-FDG PET/MR images. The uptake value of each ROI was normalized by cerebellum to obtain the standard uptake value ratio of FDG (FDG-SUVR). Compared with before levodopa treatment, FDG-SUVR of all ROIs increased significantly after 6 months of treatment ( Table 1 ). Over the next year, the doses of Madopar and Pramipexole were gradually reduced until they were discontinued. The patient's neurological symptoms have not recurred during the 10-month follow-up.
| 4.007813
| 0.950684
|
sec[1]/p[3]
|
en
| 0.999997
|
PMC10272721
|
https://doi.org/10.3389/fneur.2023.1184713
|
[
"lobe",
"madopar",
"pramipexole",
"levodopa",
"over",
"next",
"uptake",
"suvr",
"parkinsonism",
"diagnosed"
] |
[
{
"code": "CB40.2",
"title": "Pulmonary collapse"
},
{
"code": "LA75.0",
"title": "Accessory lobe of lung"
},
{
"code": "MD41",
"title": "Clinical findings on diagnostic imaging of lung"
},
{
"code": "LA75.1",
"title": "Agenesis of lung"
},
{
"code": "JA8A.1",
"title": "Malformation of placenta"
},
{
"code": "MB22.5",
"title": "Increased goal-directed activity"
},
{
"code": "DA0E.5Y",
"title": "Other specified malocclusion"
},
{
"code": "5B80.0Z",
"title": "Overweight, unspecified"
},
{
"code": "NE60",
"title": "Harmful effects of drugs, medicaments or biological substances, not elsewhere classified"
},
{
"code": "MG43.1",
"title": "Overeating"
}
] |
=== ICD-11 CODES FOUND ===
|
[] |
CB40.2
|
Pulmonary collapse
|
In March 2021, the patient complained of dizziness and headache, accompanied by nausea and vomiting, without numbness or disturbance of movement in the limbs. The patient did not pay attention to these and had not undergone further examination until June 2021. The patient was treated in Yunnan Cancer Hospital for liver cirrhosis; MRI examination found a space-occupying lesion in the left frontotemporal lobe, but further specific diagnosis and treatment were still not performed. Then, the patient developed blurred vision and double vision and went to the First Affiliated Hospital of Kunming Medical University for treatment. Head MRI examination revealed a shallow lobulated round mass (approximately 4.4×4.6×4.9 cm) with slightly long T2 and T1 signals under the left frontal cranial plate. FLAIR showed a slightly high signal, and the boundary was still clear. DWI showed high signal enhancement and obvious enhancement, and the adjacent dura mater was slightly thickened and enhanced. Moreover, the adjacent brain parenchyma was obviously compressed, and a large area of oedema and a right-skewed midline structure were observed. MRA showed that the compression of the bilateral anterior cerebral artery was shifted to the right, the A1 segment of the left anterior cerebral artery was slender, the distal part was well developed, and the M1 segment of the left middle cerebral artery was shifted downwards .
| 3.857422
| 0.983887
|
sec[1]/p[1]
|
en
| 0.999996
|
PMC10370419
|
https://doi.org/10.3389/fonc.2023.1209244
|
[
"slightly",
"cerebral",
"artery",
"still",
"vision",
"signal",
"enhancement",
"adjacent",
"shifted",
"segment"
] |
[
{
"code": "1D00.Z",
"title": "Infectious encephalitis, unspecified"
},
{
"code": "8E7Y",
"title": "Other specified diseases of the nervous system"
},
{
"code": "8B20",
"title": "Stroke not known if ischaemic or haemorrhagic"
},
{
"code": "8B1Z",
"title": "Cerebral ischaemia, unspecified"
},
{
"code": "8B11.5Z",
"title": "Cerebral ischaemic stroke, unspecified"
},
{
"code": "BD5Z",
"title": "Diseases of arteries or arterioles, unspecified"
},
{
"code": "BD52",
"title": "Certain specified disorders of arteries or arterioles"
},
{
"code": "BD52.3",
"title": "Rupture of artery"
},
{
"code": "BD52.2",
"title": "Stricture of artery"
},
{
"code": "BD40.Z",
"title": "Atherosclerotic chronic arterial occlusive disease, unspecified"
}
] |
=== ICD-11 CODES FOUND ===
|
[] |
1D00.Z
|
Infectious encephalitis, unspecified
|
Our patient, a 75-year-old male, had a left inguinoscrotal swelling for 12 years. He had no complaints as the hernia was reducible for 12 years. The patient had consistently declined to have surgery. He arrived at our emergency department complaining of terrible cramping, stomach discomfort, and distension, which had been going on for the previous five days. During this time, he did not have a bowel movement or passed any flatus. He restricted his intake to mere sips of water for the past five days. He had absolutely no urinary complaints. Physical examination revealed a moderately nourished man in severe distress from abdominal pain with a centrally distended abdomen. He was severely dehydrated and pale and gave the impression of being seriously ill. The patient’s blood pressure was 90/60 mmHg, pulse rate was 128/min, and temperature was 98.8 °F. Abdominal examination revealed a large, hard, and tender mass in the left inguinal region extending into the scrotum. The swelling measured 7 x 5 inches and was fixed and irreducible. There was guarding and rigidity as per abdominal examination. The left cord structures were palpable at the base of the penis, and the left testis was palpable vaguely at the tip of the swelling; right-sided cord structures and testis were normally palpable. No bowel sounds could be heard. The prostate gland was enlarged, and the rectum was vacant and free of any tumors.
| 3.587891
| 0.988281
|
sec[1]/p[0]
|
en
| 0.999997
|
37674969
|
https://doi.org/10.7759/cureus.43028
|
[
"swelling",
"abdominal",
"palpable",
"complaints",
"five",
"bowel",
"cord",
"structures",
"testis",
"inguinoscrotal"
] |
[
{
"code": "FA36.Z",
"title": "Effusion of joint, unspecified"
},
{
"code": "MA01.Z",
"title": "Enlarged lymph nodes, unspecified"
},
{
"code": "MD82",
"title": "Intra-abdominal or pelvic swelling, mass or lump"
},
{
"code": "ME61",
"title": "Subcutaneous swelling, mass or lump of uncertain or unspecified nature"
},
{
"code": "GB90.Y",
"title": "Other specified disorders of kidney or ureter"
},
{
"code": "MD81.3",
"title": "Acute abdomen"
},
{
"code": "JA01.0",
"title": "Abdominal pregnancy"
},
{
"code": "ME04.Z",
"title": "Ascites, unspecified"
},
{
"code": "NB51.0&XA3KX0",
"title": "Laceration without foreign body of abdominal wall"
},
{
"code": "NB9Y",
"title": "Other specified injuries to the abdomen, lower back, lumbar spine or pelvis"
}
] |
=== ICD-11 CODES FOUND ===
|
[] |
FA36.Z
|
Effusion of joint, unspecified
|
A 45-year-old man with chest discomfort on effort was referred to our hospital. He had been treated for hypertension, hyperlipidemia, hyperuricemia, and diabetes mellitus for 5 years. He had a current smoking habit and a body mass index of 35 indicating obesity, and no family history. Coronary angiography demonstrated severe coronary artery disease including severe stenosis of the left anterior descending artery, the diagonal branch, and the right coronary artery, as well as total occlusion of the left circumflex artery . Further examinations also revealed severe stenosis of the left internal carotid artery without any neurological symptoms . Because this was a marginal case for carotid artery revascularization, discussion was held to discern between the neurosurgery and cardiac surgery departments, with neurosurgeons suggesting that preemptive carotid artery stenting was not necessary. They recommended that blood pressure and hemoglobin levels be maintained during CABG, and that antiplatelet therapy be started as soon as possible following the procedure. Fig. 1 a Preoperative left coronary angiography showing severe stenosis of the left circumflex and left anterior descending arteries. b Preoperative right coronary angiography showing severe stenosis of the right coronary artery. c Preoperative computed tomography scan showing severe stenosis of the left common carotid artery (white arrowhead)
| 3.775391
| 0.982422
|
sec[1]/p[0]
|
en
| 0.999996
|
36650524
|
https://doi.org/10.1186/s13019-023-02135-2
|
[
"artery",
"coronary",
"stenosis",
"carotid",
"angiography",
"that",
"preoperative",
"descending",
"circumflex",
"chest"
] |
[
{
"code": "BD5Z",
"title": "Diseases of arteries or arterioles, unspecified"
},
{
"code": "BD52",
"title": "Certain specified disorders of arteries or arterioles"
},
{
"code": "BD52.3",
"title": "Rupture of artery"
},
{
"code": "BD52.2",
"title": "Stricture of artery"
},
{
"code": "BD40.Z",
"title": "Atherosclerotic chronic arterial occlusive disease, unspecified"
},
{
"code": "BA8Z",
"title": "Diseases of coronary artery, unspecified"
},
{
"code": "BA4Z",
"title": "Acute ischaemic heart disease, unspecified"
},
{
"code": "BA41.Z",
"title": "Acute myocardial infarction, unspecified"
},
{
"code": "BA5Z",
"title": "Chronic ischaemic heart disease, unspecified"
},
{
"code": "LA8C.2",
"title": "Congenital coronary arterial fistula"
}
] |
=== ICD-11 CODES FOUND ===
|
[] |
BD5Z
|
Diseases of arteries or arterioles, unspecified
|
A 77-year-old female patient experienced chronic cough and progressing dyspnea two months before the admission. Initial chest computed tomography (CT) showed no specific findings of infectious pneumonia, or interstitial pneumonia . Flowingly, transthoracic echocardiography exhibited right ventricular enlargement and significant PH (TRPG of 80 mmg). However, computed tomographic pulmonary angiography (CTPA) showed no specific findings of pulmonary embolism, instead of increased diffusive nodules and thickening interlobular septal . Accordingly, the blood coagulation tests showed a slightly hypercoagulative state (D-dimer of 19.38 μg/mL, and fibrin degradation products of 42.20 μg/mL). NT-proBNP level was also elevated . Additionally, no evidence of autoimmune disease was found. Fig. 1 Chest radiography and electrocardiography. A-C CT showed increased diffusive nodules and thickening interlobular septal. D CTPA showed no specific findings of pulmonary embolism. E Impaired of pulmonary perfusion detected by dual-energy CT. F PET-CT showed increased FDG uptake in the abdominal cavity near left pararenal with maximum SUV of 4.26, and the maximum cross-sectional area was about 28 × 18 mm. G TTE revealed enlargement of right ventricle, and compressed D-shaped left ventricle. H Echocardiography showed no evidence of D-shaped left ventricle after treatment. I Indicating improved chest CT after treatment
| 3.980469
| 0.976563
|
sec[1]/p[0]
|
en
| 0.999996
|
PMC10041787
|
https://doi.org/10.1186/s12959-023-00477-4
|
[
"pulmonary",
"chest",
"specific",
"ventricle",
"computed",
"pneumonia",
"echocardiography",
"enlargement",
"ctpa",
"embolism"
] |
[
{
"code": "CB40.Y",
"title": "Other specified diseases of the respiratory system"
},
{
"code": "LA75.1",
"title": "Agenesis of lung"
},
{
"code": "CA40.Z",
"title": "Pneumonia, organism unspecified"
},
{
"code": "CB41",
"title": "Respiratory failure"
},
{
"code": "NB32.3Y",
"title": "Other injury of lung"
},
{
"code": "CB7Z",
"title": "Diseases of the respiratory system, unspecified"
},
{
"code": "CB27",
"title": "Pleural effusion"
},
{
"code": "CA44",
"title": "Pyothorax"
},
{
"code": "MD30.Z",
"title": "Chest pain, unspecified"
},
{
"code": "NA80.Y&XJ1C6",
"title": "Thoracic haematoma"
}
] |
=== ICD-11 CODES FOUND ===
|
[] |
CB40.Y
|
Other specified diseases of the respiratory system
|
Discussion of the clinical case : The patient has metabolic, cardiorespiratory, and muscular alterations . Previous management was limited to a pharmacological approach, without considering lifestyle changes, such as nutritional and physical activity modifications. Basic evaluations were lacking at the time of patient admission to the cardiometabolic risk program, such as body composition, VO 2max , grip strength, and gait speed. An adequate follow-up and an early intervention would have improved the health of the patient before. Fig. 2 General evolution of cardiorespiratory fitness and muscle status during lifetime. Both conditions rapidly increase during youth, then stabilize during early adulthood, and finally decrease with slow, more or less linear kinetics. There is always variability in the values found across all ages, from which those in the lowest quartile (below percentile 25th, P25, i.e., below the white, discontinuous line) are at increased risk of cardiometabolic morbidity and mortality. The white dot illustrates the position of the patient in the clinical case regarding the distribution of the population of the same age. The routine evaluation of cardiorespiratory fitness and muscle status in adults allows to identify those below the P25 and take effective actions to help them soon cross the line over the P25 (white arrow in the insert), notably reducing their cardiometabolic risk
| 4.027344
| 0.911133
|
sec[5]/p[9]
|
en
| 0.999996
|
36683091
|
https://doi.org/10.1007/s00421-022-05114-y
|
[
"cardiorespiratory",
"cardiometabolic",
"risk",
"white",
"fitness",
"muscle",
"status",
"those",
"line",
"discussion"
] |
[
{
"code": "MC82.4",
"title": "Cardiopulmonary arrest"
},
{
"code": "CB00",
"title": "Acute respiratory distress syndrome"
},
{
"code": "MG40.0",
"title": "Cardiogenic shock"
},
{
"code": "BA01/BD1Z",
"title": "Hypertensive heart disease with heart failure"
},
{
"code": "KB23.0Z",
"title": "Respiratory distress syndrome of newborn, unspecified"
},
{
"code": "5D0Y",
"title": "Other specified metabolic disorders"
},
{
"code": "BC43.10",
"title": "Familial-genetic hypertrophic cardiomyopathy"
},
{
"code": "QC4Y",
"title": "Personal history of other specified health problems"
},
{
"code": "QA43.Z",
"title": "Supervision of high-risk pregnancy, unspecified"
},
{
"code": "QA43.Y",
"title": "Other specified supervision of high-risk pregnancy"
}
] |
=== ICD-11 CODES FOUND ===
|
[] |
MC82.4
|
Cardiopulmonary arrest
|
We present a case of a 50-year-old male with a longstanding history of ulcerative colitis who got diagnosed with advanced unresectable colon cancer at the hepatic flexure with extension into the peri-colonic fascia in the setting of hematochezia. The patient's medical history was significant for a 50-pound weight loss in the last five months preceding the diagnosis alongside worsening abdominal pain. On imaging, there were concerns for multiple enlarged abdominal lymph nodes as well as the potential involvement of the second and third part of the duodenum by the hepatic flexure carcinoma. The cancer was considered to be unresectable and potentially metastatic; therefore, systemic therapy was recommended. A biopsy of the hepatic flexure mass came back as adenocarcinoma with squamous differentiation. Given his otherwise young age and good performance status, the patient received triplet chemotherapy with targeted therapy (FOLFOXIRI with bevacizumab). He had an excellent clinical and radiographic response allowing for conversion surgery. Final pathology came back as ypT4bN0 (0 out of 30 lymph nodes), with no evidence of metastases and grade 3 tumor regression was noted. However, there were concerns about the margins being positive on the duodenal side as well as tumor deposits noted with the specimen. The patient underwent a subtotal colectomy given his prior history of inflammatory bowel disease.
| 3.886719
| 0.981445
|
sec[1]/p[0]
|
en
| 0.999997
|
PMC10428188
|
https://doi.org/10.7759/cureus.43391
|
[
"hepatic",
"flexure",
"unresectable",
"cancer",
"abdominal",
"concerns",
"lymph",
"nodes",
"well",
"came"
] |
[
{
"code": "DB9Z",
"title": "Diseases of liver, unspecified"
},
{
"code": "DB97.Z",
"title": "Inflammatory liver disease, unspecified"
},
{
"code": "DB99.7",
"title": "Hepatic failure without mention whether acute or chronic"
},
{
"code": "LB20.0Y",
"title": "Other specified structural developmental anomalies of liver"
},
{
"code": "LB20.0Z",
"title": "Structural developmental anomalies of liver, unspecified"
},
{
"code": "EA91.Y",
"title": "Other specified lichen planus"
},
{
"code": "EC23.0",
"title": "Non-syndromic genetically-determined hypermelanosis or lentiginosis"
},
{
"code": "EA90.52",
"title": "Flexural and intertriginous psoriasis"
},
{
"code": "2D85",
"title": "Malignant neoplasm metastasis in large intestine"
},
{
"code": "2B90.0Y&XA95L3",
"title": "Malignant neoplasm of hepatic flexure"
}
] |
=== ICD-11 CODES FOUND ===
|
[] |
DB9Z
|
Diseases of liver, unspecified
|
Before deciding to perform the islet alloautotransplantation, we discussed whether to treat the likely pancreas allograft rejection with a third round of methylprednisolone and a postponed second-line treatment for chronic (or mixed-type) ABMR, which consists of alemtuzumab, plasma exchange, and/or IVIg at our center. Viral complications (BK-viremia, COVID-19) had prohibited escalation before. However, we felt that efficacy of such therapy for longer-standing chronic active perhaps antibody-mediated pancreas rejection would be questionable and that, given the progressive complications (panniculitis) necessitating chronic use of opioids, a more definite solution was warranted. The persistent immunodominant HLA-DSA DQB1*05:02-DQA1*01:02 at 1 year post-islet alloautotransplantation suggests indeed that second-line treatment would not have made the DSA disappear. Although speculative, we also hypothesized that taking away the subsisting inflammatory stimulus (exocrine tissue rejection/pancreatitis) would likely increase the chance of a more sustained response to novel induction treatment and better preserve his kidney function considering his highly-immunized status . A limitation of this point of view is that an auto-immune etiology of his type-1 diabetes could not be established (no detectable auto-antibodies), which might have contributed to a more favorable outcome of the islet transplantation.
| 4.148438
| 0.517578
|
sec[2]/p[2]
|
en
| 0.999994
|
37692453
|
https://doi.org/10.3389/ti.2023.11505
|
[
"that",
"islet",
"rejection",
"alloautotransplantation",
"likely",
"pancreas",
"line",
"type",
"which",
"complications"
] |
[
{
"code": "8A80.Z",
"title": "Migraine, unspecified"
},
{
"code": "QA76",
"title": "Medication or substance that is known to be an allergen without injury or harm"
},
{
"code": "PL13.6",
"title": "Medication or substance that is known to be an allergen, as mode of injury or harm"
},
{
"code": "9C40.A0",
"title": "Papilloedema"
},
{
"code": "PA6Z",
"title": "Unintentional fall from unspecified height"
},
{
"code": "2E92.9",
"title": "Benign neoplasm of endocrine pancreas"
},
{
"code": "5A4Y",
"title": "Other specified disorders of glucose regulation or pancreatic internal secretion"
},
{
"code": "2C10.1",
"title": "Neuroendocrine neoplasms of pancreas"
},
{
"code": "2D81",
"title": "Malignant neoplasm metastasis in pancreas"
},
{
"code": "2C10.Y",
"title": "Other specified malignant neoplasms of pancreas"
}
] |
=== ICD-11 CODES FOUND ===
|
[] |
8A80.Z
|
Migraine, unspecified
|
This is an example. A 66-year-old man with 1.8 cm sporadic RET negative MTC underwent surgery. Pathology showed many microscopic lymph node metastases. Preoperative calcitonin at 3800 pg/ml decreased to 190 pg/ml postoperatively, indicating he had BPD. How do you predict the prognosis of the patient? Pathological node metastases and persistent disease. Poor prognosis? With the resected tumor weight, preoperative and postoperative calcitonin values, the residual tumor weight was calculated to be 0.18 g. The number ( β ) of doublings for this tumor to reach 1000 g was 12.4. Calcitonin DT was widely distributed in 45 patients with MTC treated at Kuma Hospital who had BPD postoperatively . Most of the patients had calcitonin DT of longer than 4 years. If his calcitonin DT was 4 years, the expected survival was calculated to be 49 years. If 1000 g seems too big, you can set the host-killing tumor size to 100 g. In this setting, β is 9.1 and the expected survival is 36 years. The actual calcitonin DT calculated 3 years after surgery was 5.6 years. I can say that this patient will not die of MTC although he has persistent disease. I said to the patient, “Don’t worry too much, you will live for long time.” Fig. 3 Distribution according to calcitonin doubling time of 45 patients with medullary thyroid carcinoma who underwent surgery at Kuma Hospital and had persistent hypercalcitoninemia postoperatively
| 3.869141
| 0.583984
|
sec[1]/p[3]
|
en
| 0.999997
|
36153411
|
https://doi.org/10.1007/s00268-022-06741-4
|
[
"calcitonin",
"this",
"tumor",
"postoperatively",
"persistent",
"calculated",
"patients",
"node",
"metastases",
"preoperative"
] |
[
{
"code": "5A04",
"title": "Hypersecretion of calcitonin"
},
{
"code": "4A01.03",
"title": "Transient hypogammaglobulinaemia of infancy"
},
{
"code": "2F9Z",
"title": "Neoplasms of unknown behaviour of unspecified site"
},
{
"code": "ME61",
"title": "Subcutaneous swelling, mass or lump of uncertain or unspecified nature"
},
{
"code": "2E6Z",
"title": "Carcinoma in situ of unspecified site"
},
{
"code": "2F91.1",
"title": "Neoplasms of unknown behaviour of trachea, bronchus or lung"
},
{
"code": "2F92",
"title": "Neoplasms of unknown behaviour of skin"
},
{
"code": "NE81.0Z",
"title": "Haemorrhage or haematoma of other or unspecified site complicating a procedure, not elsewhere classified"
},
{
"code": "NE81.3",
"title": "Postsurgical leak"
},
{
"code": "QF00",
"title": "Acquired absence of limb"
}
] |
=== ICD-11 CODES FOUND ===
|
[] |
5A04
|
Hypersecretion of calcitonin
|
Due to our high suspicion of HSV encephalitis in our patient, we analyze the current literature regarding the potential causes that could have yielded a negative PCR test. Some investigators believe that 1% to 5% of CSF specimens might contain inhibitors that interfere with PCR . For example, a high concentration of porphyrin compounds, which can be derived from the hemolysis of red blood cells, can interfere with PCR reactions . Other explanations for a negative PCR test include the PCR being sent during the first 4 days of neurological symptoms, the viral load being too low for detection, or the CSF sample being too low in volume . Low CSF cell counts, location of the infection in the CNS, sample dilution, and variation in primers used during PCR processing have also been suggested as the cause for false-negative results . Negative PCR tests tend to also be associated with low CSF protein and leukocyte counts . However, our case demonstrates an atypical presentation with high CSF glucose and protein with normal leukocyte counts. Additionally, our case is unique in the regard that the patient had reportedly been suffering from mild neurologic symptoms for ten days prior to the ED admission. We argue that the admission CSF analysis would’ve yielded a positive result for HSV based on the timeline that the CSF was collected after 72 hours since the onset of neurological symptoms due to infection.
| 4.128906
| 0.575684
|
sec[2]/p[1]
|
en
| 0.999995
|
PMC9980281
|
https://doi.org/10.7759/cureus.34448
|
[
"that",
"counts",
"yielded",
"interfere",
"neurological",
"sample",
"infection",
"protein",
"leukocyte",
"suspicion"
] |
[
{
"code": "8A80.Z",
"title": "Migraine, unspecified"
},
{
"code": "QA76",
"title": "Medication or substance that is known to be an allergen without injury or harm"
},
{
"code": "PL13.6",
"title": "Medication or substance that is known to be an allergen, as mode of injury or harm"
},
{
"code": "9C40.A0",
"title": "Papilloedema"
},
{
"code": "PA6Z",
"title": "Unintentional fall from unspecified height"
},
{
"code": "3B63.1Z",
"title": "Acquired thrombocytosis, unspecified"
},
{
"code": "3B64.Z",
"title": "Thrombocytopenia, unspecified"
},
{
"code": "4B0Z",
"title": "Immune system disorders involving white cell lineages, unspecified"
},
{
"code": "4B03.Z",
"title": "Eosinophilia, unspecified"
},
{
"code": "4B00.1Z",
"title": "Neutrophilia, unspecified"
}
] |
=== ICD-11 CODES FOUND ===
|
[] |
8A80.Z
|
Migraine, unspecified
|
A 66-year-old male patient with a history of prostatectomy due to prostate carcinoma and with a persistent single osteoblastic osseous metastasis (left os ilium) presented to our department with a first diagnosis of stage III AITL with exclusive lymph node manifestation. After five courses of CHOP, peripheral blood stem cell (PBSC) apheresis between each course of DHAP and DHAC was performed. At the timepoint of HD chemotherapy (BEAM) and ASCT, the patient was in complete remission. Within 6 months upon ASCT, early relapse occurred, and despite two courses of salvage treatment with gemcitabine and oxaliplatin, the disease progressed. The PSMA-PET result confirmed a stable disease (M1 oss.) of prostate carcinoma. Due to AITL progression, treatment was rotated to all-oral TEPIP (q4w, d1-7), and complete and durable remission (24 months) was achieved within two courses . Finally, treatment was terminated due to long-lasting CR after 2 years. Within the following 3 months, the patient relapsed with lymphoma infiltration of the skin, lymph nodes, and bone marrow (stage IV). At the same timepoint, a myelodysplastic neoplasm (MDS-RS-MLD) was incidentally diagnosed. TEPIP was re-initiated in five-weekly courses, resulting in a renewed very good PR within 3 months . Due to MDS-related cytopenia, TEPIP was replaced with 5-azacitidine. However, the patient died due to progression to AML at 5 months later.
| 3.982422
| 0.977051
|
sec[2]/sec[1]/sec[3]/p[0]
|
en
| 0.999996
|
PMC10250661
|
https://doi.org/10.3389/fonc.2023.1177330
|
[
"courses",
"within",
"tepip",
"prostate",
"carcinoma",
"stage",
"aitl",
"lymph",
"five",
"timepoint"
] |
[
{
"code": "LA8Y",
"title": "Other specified structural developmental anomaly of heart or great vessels"
},
{
"code": "8B10.Y",
"title": "Other specified transient ischaemic attack"
},
{
"code": "6A80",
"title": "Symptomatic and course presentations for mood episodes in mood disorders"
},
{
"code": "LA8C.1",
"title": "Anomalous aortic origin or course of coronary artery"
},
{
"code": "8B22.Y",
"title": "Other specified cerebrovascular disease"
},
{
"code": "QE70.Z",
"title": "Problems related to primary support group, including family circumstances, unspecified"
},
{
"code": "8D6Y",
"title": "Other specified disorders of cerebrospinal fluid pressure or flow"
},
{
"code": "LA8E.1",
"title": "Atrial septal defect within oval fossa"
},
{
"code": "GA10.CZ",
"title": "Endometriosis of unspecified site within the digestive system"
},
{
"code": "GA10.CY",
"title": "Endometriosis of other sites within the digestive system"
}
] |
=== ICD-11 CODES FOUND ===
|
[] |
LA8Y
|
Other specified structural developmental anomaly of heart or great vessels
|
The exact time length of reversible retinal hypoxic damage is unknown yet and it is estimated between 4.5 and 12 h . Intravenous tissue plasminogen activator (tPA) is an evidence-based therapy for AIS, when provided within 4.5 h . Treatment with tPA showed a strong effect even in patients with CRAO , however adequate randomized clinical trials are still ongoing . In centers where endovascular therapy is performed, intra-arterial tPA (Ia-tPA) may be considered an alternative treatment, and the time window for drug administration is potentially increased . Regretfully, although prompt management of acute CRAO is mandatory to maximize visual outcome and avoid any secondary ischemic event, a unanimous consensus on the topic has not been reached yet. Our child did not receive a thrombolytic treatment due to the late diagnosis, whereas anticoagulant therapy was soon administered. Several non-invasive treatments have been proposed and tried in some adult cases ; however these strategies are not uniformly supported by the current scientific literature, since there is no evidence of effectiveness . In order to improve retinal oedema, and bearing in mind the known inflammatory and vasculitis tendency of SARS-CoV-2 infection, iv and oral steroids were administered to our patient, nonetheless with poor visual outcome, in accordance to other adult cases in the literature and to the known prognosis of CRAO .
| 4.246094
| 0.86084
|
sec[2]/p[6]
|
en
| 0.999998
|
37704960
|
https://doi.org/10.1186/s12887-023-04276-8
|
[
"crao",
"time",
"retinal",
"evidence",
"however",
"visual",
"outcome",
"cases",
"literature",
"known"
] |
[
{
"code": "9B74.0",
"title": "Retinal artery occlusions"
},
{
"code": "PL13.52",
"title": "Incorrect timing of drug or medicament, as mode of injury"
},
{
"code": "QF2A",
"title": "Difficulty or need for assistance with community participation"
},
{
"code": "MF50.1",
"title": "Pollakiuria"
},
{
"code": "JA25.3",
"title": "Eclampsia, time period unspecified"
},
{
"code": "KD3B.Z",
"title": "Unspecified time of fetal death, cause not specified"
},
{
"code": "9B7Z",
"title": "Disorders of the retina, unspecified"
},
{
"code": "9B74.Z",
"title": "Retinal vascular occlusions, unspecified"
},
{
"code": "9B73.4",
"title": "Retinal breaks without detachment"
},
{
"code": "9B78.9",
"title": "Retinal atrophy"
}
] |
=== ICD-11 CODES FOUND ===
|
[] |
9B74.0
|
Retinal artery occlusions
|
New-onset therapy refractory status epilepticus (NORSE) can result from several conditions, of which the most common are autoimmune encephalitis and infectious diseases. 10 , 11 Mitochondrial diseases are listed as rare etiology of NORSE, and the current diagnostic recommendations do not contemplate a systematic screening for mitochondrial defects in adults, 10 , 12 , 13 which is still time and resource consuming outside of specialized centers. Since a normal CSF examination rules out the most common differential diagnoses of NORSE, the application of a rapid genetic test, as further diagnostic step, would have been a posteriori the approach of choice in the index patient. Rapid genetic testing strategies have been successfully applied in the setting of fulminant pediatric disorders, opening a window of opportunity when a potentially treatable disorder is untangled. 14 , 15 In our case, the time pressure because of unsuccessful status treatment and the concern of omitting a potentially effective drug, motivated, in the setting of missing guidelines, a difficult therapeutic choice. It remains speculative whether an earlier diagnosis, by means of rapid genetic testing, could have influenced the course of the disease in our patient. However, it would have prevented complications due to inappropriate application of first-line pharmacologic therapies (valproate in the setting of myoclonic epilepsy).
| 4.195313
| 0.489258
|
sec[1]/p[3]
|
en
| 0.999996
|
PMC10117698
|
https://doi.org/10.1212/NXG.0000000000200063
|
[
"norse",
"rapid",
"genetic",
"setting",
"status",
"which",
"common",
"diseases",
"mitochondrial",
"time"
] |
[
{
"code": "6A80.5",
"title": "Rapid cycling"
},
{
"code": "MC81.0",
"title": "Tachycardia, unspecified"
},
{
"code": "QA00.Y",
"title": "Other specified general examination or investigation of persons without complaint or reported diagnosis"
},
{
"code": "7B01.0",
"title": "REM sleep behaviour disorder"
},
{
"code": "BC81.3Z",
"title": "Atrial fibrillation, unspecified"
},
{
"code": "LD27.6Z",
"title": "Genetic lipodystrophy, unspecified"
},
{
"code": "8E02.Z",
"title": "Creutzfeldt-Jakob disease, unspecified"
},
{
"code": "EC20",
"title": "Genetic disorders of keratinisation"
},
{
"code": "AB56",
"title": "Hereditary hearing loss"
},
{
"code": "EC10",
"title": "Genetic syndromes with poikiloderma"
}
] |
=== ICD-11 CODES FOUND ===
|
[] |
6A80.5
|
Rapid cycling
|
A 27-year-old female, with underlying T1DM for 17 years, was recently admitted to the hospital in order to achieve better glycemic control. Vital signs at presentation were a temperature of 36.1 °C, a pulse rate of 100 beats/min, a respiratory rate of 28 breaths/min, and blood pressure of 120/85 mm Hg. Past medical history included T1DM managed with an insulin pump 15 years. In recent years, the dosage of insulin was 45 IU/d, and metformin 0.5 g (qid) was added to the therapeutic regimen 3 years ago due to weight gain. On admission, characteristics were noted as follows: her body weight was 75.0 kg; BMI was 28.2 kg/m 2 ; and waist circumference was 105.5 cm. Clinical laboratory analysis found that she was positive for urine ketones, her fasting blood glucose was 10.5 mmol/L, and her glycated hemoglobin (HbA1c) was 7.7%. Her was C-peptide <0.02 ng/mL, insulin antibodies (IAA) were 4.96 RU/mL, islet cell antibodies (ICA) were 6.09 IU/mL, and glutamic acid decarboxylase antibodies (GAD) were 18.44 IU/mL. An abdominal ultrasound indicated that she had a fatty liver. An electrocardiograph (ECG) and auscultation of the lungs showed no significant findings; her liver function, renal function, and thyroid function had no obvious abnormalities. The patient had no smoking and alcohol intake history, but she did have a history of fracture surgery due to a traffic accident and a family history of diabetes.
| 3.841797
| 0.983398
|
sec[1]/p[0]
|
en
| 0.999998
|
37241059
|
https://doi.org/10.3390/medicina59050827
|
[
"insulin",
"antibodies",
"function",
"blood",
"weight",
"that",
"liver",
"underlying",
"recently",
"order"
] |
[
{
"code": "5A44",
"title": "Insulin-resistance syndromes"
},
{
"code": "5A4Y",
"title": "Other specified disorders of glucose regulation or pancreatic internal secretion"
},
{
"code": "QB51.5",
"title": "Presence of endocrine implants"
},
{
"code": "EF02.0",
"title": "Fat hypertrophy"
},
{
"code": "PK9C.2",
"title": "Other or unspecified medical devices associated with injury or harm, prosthetic or other implants, materials or accessory devices"
},
{
"code": "JA86.Y",
"title": "Maternal care for other specified fetal problems"
},
{
"code": "MA14.14",
"title": "Anti-nuclear antibody positive"
},
{
"code": "MA14.13",
"title": "Anti-nuclear antibody negative"
},
{
"code": "JA86.0",
"title": "Maternal care for red cell antibodies"
},
{
"code": "MA14.1C",
"title": "Raised antibody titre"
}
] |
=== ICD-11 CODES FOUND ===
|
[] |
5A44
|
Insulin-resistance syndromes
|
A 20‐year‐old female patient was referred to our hospital with a 2‐year history of hypertrophic cardiomyopathy (HCM) and one occurrence of syncope. Two years ago, the patient was found to have a cardiac abnormality during an electrocardiograph (ECG) examination with sinus bradycardia and an irregular indoor conduction block. She denied having symptoms of palpitation, chest tightness, or shortness of breath. Echocardiography and CMR suggested HCM with a local narrow heart cavity. The patient was given appropriate HCM medication (details not available) but discontinued this 1 month later without the regular follow‐up. Her parents recalled that approximately 25 days before admission, the patient had sudden syncope with loss of consciousness while walking of 1‐min duration but no symptoms of incontinence, limb twitching, nausea, or vomiting were apparent. The parents recalled the ECG and head CT (Computed Tomography) taken at the time as being normal (results unavailable). The patient was admitted to our hospital and received a comprehensive examination, including somatoscopy (including muscle strength assessment by MRC grade and retinopathy by ophthalmoscope), evaluation of intellectual development by Wechsler Intelligence Scale, physical examination (including Holter, electrocardiogram, echocardiography, cardiac MRI), and laboratory tests (including myocardial enzymes and biochemical indicators).
| 3.945313
| 0.98291
|
sec[1]/sec[1]/p[0]
|
en
| 0.999996
|
37288668
|
https://doi.org/10.1002/mgg3.2216
|
[
"including",
"syncope",
"cardiac",
"echocardiography",
"parents",
"recalled",
"referred",
"hypertrophic",
"cardiomyopathy",
"occurrence"
] |
[
{
"code": "7A24",
"title": "Hypersomnia due to a medication or substance"
},
{
"code": "9D90",
"title": "Vision impairment including blindness"
},
{
"code": "NE61",
"title": "Harmful effects of or exposure to noxious substances, chiefly nonmedicinal as to source, not elsewhere classified"
},
{
"code": "3A50.02",
"title": "Haemoglobin H disease (– α/– – included)"
},
{
"code": "LA8B.Z",
"title": "Congenital anomaly of great arteries including arterial duct, unspecified"
},
{
"code": "MG45.Z",
"title": "Syncope or collapse, unspecified"
},
{
"code": "8D89.0",
"title": "Reflex syncope"
},
{
"code": "MH12.0",
"title": "Instantaneous death"
},
{
"code": "MD12",
"title": "Cough"
},
{
"code": "8D89.1",
"title": "Syncope due to autonomic failure"
}
] |
=== ICD-11 CODES FOUND ===
|
[] |
7A24
|
Hypersomnia due to a medication or substance
|
Prolonged afebrile neutropenia requiring growth factor support complicated the patient’s initial chemotherapy course and led to a delay in cycle two. A ctDNA level after the first cycle of FOLFOXIRI-Bev decreased to 0.04 MTM/mL . Interval imaging after two cycles of chemotherapy showed a response of all hepatic lesions, with ctDNA level cleared to undetectable (0.00 MTM/mL) . After completing three cycles of chemotherapy, in consultation with surgical oncology, the patient elected to have definitive microwave ablation of the three hepatic metastases. The patient’s bevacizumab was held during the third cycle of chemotherapy, pre-operatively, but was resumed during his fourth cycle, post-ablation. The patient’s ctDNA level remained undetectable (0.00 MTM/mL) throughout the remainder of his adjuvant treatment course. Considering the lack of detectable ctDNA and persistent side effects from chemotherapy experienced by the patient, a shared decision was made to defer the sixth cycle of chemotherapy and, overall, the patient completed five cycles of adjuvant chemotherapy. Colonoscopy one year after surgery was unremarkable, and no new metastatic lesions were noted on repeat CT scan after chemotherapy completion. Of note, the patient’s CEA levels remained within normal limits (≤3.0 ng/mL per institutional assay) throughout his disease course, from diagnosis to recurrence to post-treatment monitoring .
| 3.923828
| 0.962891
|
sec[1]/p[4]
|
en
| 0.999997
|
PMC9795219
|
https://doi.org/10.3389/fonc.2022.1074786
|
[
"chemotherapy",
"cycle",
"ctdna",
"course",
"cycles",
"hepatic",
"lesions",
"undetectable",
"three",
"ablation"
] |
[
{
"code": "QB97",
"title": "Contact with health services for chemotherapy session for neoplasm"
},
{
"code": "QC05.Y",
"title": "Other specified prophylactic measures"
},
{
"code": "QB9Y",
"title": "Other specified contact with health services for nonsurgical interventions not involving devices"
},
{
"code": "3B64.1Y",
"title": "Other specified acquired thrombocytopenia"
},
{
"code": "QC48.Y",
"title": "Other specified personal history of medical treatment"
},
{
"code": "6A80.5",
"title": "Rapid cycling"
},
{
"code": "PA03",
"title": "Unintentional land transport traffic event injuring a motor cyclist"
},
{
"code": "5C50.AZ",
"title": "Disorders of urea cycle metabolism, unspecified"
},
{
"code": "PA23",
"title": "Unintentional land transport event unknown whether traffic or nontraffic injuring a motor cyclist"
},
{
"code": "PA02",
"title": "Unintentional land transport traffic event injuring a pedal cyclist"
}
] |
=== ICD-11 CODES FOUND ===
|
[] |
QB97
|
Contact with health services for chemotherapy session for neoplasm
|
A healthy 12-year-old male with no significant past medical history or drug allergies was playing basketball at his aunt's house when he suddenly developed a severe headache accompanied by nausea and vomiting. He denied any head trauma to his aunt and mother. He was given an over-the-counter analgesic and went to bed, awakening in the early morning hours with another episode of a sudden severe headache that subsided without medication. He went back to bed, but a couple of hours later, his mother noticed that he had wet his bed. When awakened, he was drowsy and confused. His mother took him to the local hospital. He had two tonic-clonic seizure episodes immediately after his arrival and was intubated to protect his airway. A head computed tomographic (CT) scan without contrast showed a 6.0 x 3.2 x 3.0 cm right frontal intracerebral hematoma with rupture into the frontal ventricular horn extending into all the ventricles, causing a 1.2 cm shift of the midline . The patient was promptly transferred to our institution for neurosurgical evaluation. The patient arrived at our institution intubated without sedation. The physical examination demonstrated an acutely ill patient unresponsive to painful external stimuli. The head had no evidence of trauma. He did not open his eyes spontaneously or to pain. The pupils were midsize with sluggish pupillary reflexes. He had corneal and gag reflexes present.
| 3.6875
| 0.986328
|
sec[1]/p[0]
|
en
| 0.999997
|
PMC9814825
|
https://doi.org/10.7759/cureus.32251
|
[
"head",
"mother",
"without",
"aunt",
"headache",
"trauma",
"went",
"hours",
"that",
"intubated"
] |
[
{
"code": "NA63",
"title": "Traumatic amputation at neck level"
},
{
"code": "MB4D",
"title": "Headache, not elsewhere classified"
},
{
"code": "ME61",
"title": "Subcutaneous swelling, mass or lump of uncertain or unspecified nature"
},
{
"code": "MB48.3",
"title": "Light-headedness"
},
{
"code": "FB3Z",
"title": "Disorders of muscles, unspecified"
},
{
"code": "QA48.1",
"title": "Care or examination of lactating mother"
},
{
"code": "KB60.1",
"title": "Syndrome of infant of a diabetic mother, type 1 or 2, nongestational, insulin dependent"
},
{
"code": "1C1D.0",
"title": "Primary yaws"
},
{
"code": "KD35",
"title": "Neonatal withdrawal syndrome from maternal use of drugs of addiction"
},
{
"code": "KB60.0",
"title": "Syndrome of infant of mother with gestational diabetes"
}
] |
=== ICD-11 CODES FOUND ===
|
[] |
NA63
|
Traumatic amputation at neck level
|
The case is of a 29-year-old female of African descent from Haiti who was referred to hematology for severe neutropenia. Notably, she had a history of cardiovascular malformation (ventricular septal defect) surgically repaired at the age of 5 and recurrent skin and oto-sinopulmonary infections (sinusitis, otitis media, and pneumonia) since childhood resulting in progressive hearing loss and chronic bronchiectasis. She also reported anosmia and recurrent blisters of the philtrum, likely herpetic lesions, although no such lesions were present at the time of our examination. Of note, her HSV-1 antibodies were positive on serology. The patient reported no history of tobacco smoking, alcohol use, or substance abuse. Her family history was notable for a brother with recurrent infections but no formal diagnosis of primary immunodeficiency. There was no other known family history of hematologic disorders, immunodeficiency, or malignancy. Family members had been offered genetic testing but to date, no specimens have been sent in for evaluation. Six years prior to her current presentation, she underwent a bone marrow biopsy to evaluate incidentally-discovered neutropenia. The biopsy showed a largely normal marrow with trilineage hematopoiesis, erythroid hypoplasia, and plasmacytosis without light chain restriction. An aspirate could not be performed, and no cytogenetic or molecular testing was available.
| 3.931641
| 0.983887
|
sec[1]/p[0]
|
en
| 0.999997
|
PMC9925260
|
https://doi.org/10.1155/2023/3888680
|
[
"recurrent",
"family",
"neutropenia",
"infections",
"lesions",
"immunodeficiency",
"testing",
"marrow",
"biopsy",
"african"
] |
[
{
"code": "9A78.8",
"title": "Recurrent erosion of cornea"
},
{
"code": "7A22",
"title": "Kleine-Levin syndrome"
},
{
"code": "DC50.1Y",
"title": "Other specified secondary peritonitis"
},
{
"code": "GA33",
"title": "Recurrent pregnancy loss"
},
{
"code": "8C74.1Z",
"title": "Periodic paralysis, unspecified"
},
{
"code": "QE70.Z",
"title": "Problems related to primary support group, including family circumstances, unspecified"
},
{
"code": "2B90.Y",
"title": "Other specified malignant neoplasms of colon"
},
{
"code": "EE61",
"title": "Superficial fibromatoses"
},
{
"code": "9B70",
"title": "Inherited retinal dystrophies"
},
{
"code": "4B00.0Z",
"title": "Neutropaenia, unspecified"
}
] |
=== ICD-11 CODES FOUND ===
|
[] |
9A78.8
|
Recurrent erosion of cornea
|
A 26-year-old gravida 2 para 0, was referred to Princess Christian Maternity Hospital, Freetown, Sierra Leone, with amenorrhea of three months duration a three day history of worsening pelvic pain. She had a prior history of right salpingectomy performed 3 years before presentation as a result of ruptured right tubal EP. On examination, there was moderate tachycardia and lower abdominal tenderness; the pregnancy test was positive, and the transabdominal ultrasound scan revealed a 12-weeks left adnexal EP with a vital fetus and no free fluid collection in the pouch of Douglas . In this public academic hospital No other hematological diagnostic or imaging investigations were available, and they were not included in the free healthcare guaranteed by the national program. Blood tests and magnetic resonance imaging are carried out in other private institutions. A rapid test for hemoglobin was performed and the result was within the normal range. In the presence of the husband, the patient was offered detailed counselling for the management of her condition. Considering the vital fetus, the global consensus indicates the surgical removal of the pregnancy. As a symptom, pain could indicate impending or initial tubal rupture, and the consequent bleeding potentially threatens the patient’s survival. Therefore, emergency exploratory laparotomy was proposed for suspicion of the imminent rupture of the EP.
| 3.816406
| 0.983398
|
sec[2]/p[0]
|
en
| 0.999998
|
37685709
|
https://doi.org/10.3390/jcm12175642
|
[
"three",
"pain",
"tubal",
"pregnancy",
"vital",
"fetus",
"free",
"imaging",
"rupture",
"gravida"
] |
[
{
"code": "1D45",
"title": "Sandfly fever"
},
{
"code": "PA09",
"title": "Unintentional land transport traffic event injuring an occupant of a low powered passenger vehicle"
},
{
"code": "LD50.Y",
"title": "Other specified number anomalies of chromosome X"
},
{
"code": "PA19",
"title": "Unintentional land transport nontraffic event injuring an occupant of a low powered passenger vehicle"
},
{
"code": "NA82.4",
"title": "Multiple fractures of ribs"
},
{
"code": "MG3Z",
"title": "Pain, unspecified"
},
{
"code": "8E43.Z",
"title": "Pain disorders, unspecified"
},
{
"code": "MG31.Z",
"title": "Acute pain, unspecified"
},
{
"code": "MG30.Z",
"title": "Chronic pain, unspecified"
},
{
"code": "FB56.2",
"title": "Myalgia"
}
] |
=== ICD-11 CODES FOUND ===
|
[] |
1D45
|
Sandfly fever
|
Techniques of diagnostic imaging are available if there is a clinical suspicion of splenogonadal fusion. The most reliable investigation to confirm its diagnosis is preoperative imaging through 99 m Tc-sulfur colloid liver spleen scan, which detects accessory spleen. Recently, Doppler ultrasonography of the scrotum of patients with a palpable testicular mass has been used to diagnose preoperatively few cases of splenogonadal fusion by visualizing a hypervascular mass on the upper pole of testis and comparing it with patient's own normally located spleen . Such use of this modality again depends on high index of suspicion in cases of splenogonadal fusion presenting as cryptorchidism, but it may be of value in patients presenting with acute scrotal lump. Unaware of the nature of the left scrotal masses, many surgeons have sacrificed an intact testis because they lacked a proper preoperative diagnosis. Surgeons ignorant of the nature of the scrotal mass and fearing malignant degeneration have performed radical orchidectomy on salvageable testes . In our cases, as the patient had primary gonadal failure and laparoscopic assessment was suggestive of benign testicular mass due to splenogonadal fusion with atrophic testis, so simple excision of mass was done. This highlights the importance of laparoscopic assessment for the nature of the mass, which saves the patient from undergoing a radical surgery.
| 4.15625
| 0.577148
|
sec[2]/p[2]
|
en
| 0.999997
|
24963439
|
https://doi.org/10.1155/2014/796761
|
[
"splenogonadal",
"fusion",
"spleen",
"cases",
"testis",
"scrotal",
"nature",
"imaging",
"suspicion",
"preoperative"
] |
[
{
"code": "FA34.4",
"title": "Ankylosis of joint"
},
{
"code": "LB17.2",
"title": "Persistent cloaca"
},
{
"code": "FB00",
"title": "Ankylosis of spinal joint"
},
{
"code": "LD2G",
"title": "Conjoined twins"
},
{
"code": "LB79.0",
"title": "Fused fingers"
},
{
"code": "3B8Z",
"title": "Diseases of spleen, unspecified"
},
{
"code": "LA8Z",
"title": "Structural developmental anomaly of heart or great vessels, unspecified"
},
{
"code": "3B81.Y",
"title": "Other specified acquired disorders of spleen"
},
{
"code": "LB22.Y",
"title": "Other specified structural developmental anomalies of spleen"
},
{
"code": "3B81.2",
"title": "Atrophy of spleen"
}
] |
=== ICD-11 CODES FOUND ===
|
[] |
FA34.4
|
Ankylosis of joint
|
The masses seen at laparoscopy had the low power appearance of a well-differentiated carcinoma that was arranged primarily in solid nests. The tumor invaded through the muscularis propria and into the subserosal soft tissue . Multiple foci of tumor, the largest measuring three centimeters, were present in the lamina propria and submucosa as well as at the proximal and distal margins of resection. The tumor was composed of cells with abundant eosinophilic cytoplasm and round nuclei that contained finely stippled chromatin . Focal, mild anisokaryosis was present, but no necrosis was identified. The morphologic features were highly suspicious of a neuroendocrine tumor, so immunoperoxidase studies were performed for confirmation as well as ruling out an adenocarcinoma ( Table 1 ). The tumor was strongly and diffusely positive for synaptophysin and chromogranin, confirming neuroendocrine differentiation. To determine the grade of the tumor, a mitotic count and a Ki67 immunostain were also performed. The tumor had one mitosis per ten high power fields averaged over 50 high power fields, consistent with a G1. The Ki67 proliferation index was 3.5%, consistent with a G2. Since the proliferation index was of a higher grade, the tumor was graded as a G2. The tumor had metastasized to four lymph nodes , which, along with the tumor size and extent of invasion, was consistent with a more aggressive neoplasm.
| 4.242188
| 0.489746
|
sec[3]/p[0]
|
en
| 0.999997
|
25525544
|
https://doi.org/10.1155/2014/426161
|
[
"tumor",
"power",
"well",
"consistent",
"that",
"propria",
"present",
"neuroendocrine",
"grade",
"fields"
] |
[
{
"code": "2F9Z",
"title": "Neoplasms of unknown behaviour of unspecified site"
},
{
"code": "ME61",
"title": "Subcutaneous swelling, mass or lump of uncertain or unspecified nature"
},
{
"code": "2E6Z",
"title": "Carcinoma in situ of unspecified site"
},
{
"code": "2F91.1",
"title": "Neoplasms of unknown behaviour of trachea, bronchus or lung"
},
{
"code": "2F92",
"title": "Neoplasms of unknown behaviour of skin"
},
{
"code": "PB6Y",
"title": "Other unintentional cause of morbidity or mortality"
},
{
"code": "PA83.2&XE02T",
"title": "Accidental contact with powered knife"
},
{
"code": "MA80.1",
"title": "Dysphasia"
},
{
"code": "PA83.2",
"title": "Unintentionally cut or pierced by other or unspecified sharp object"
},
{
"code": "PA83.2&XE8MJ",
"title": "Accidental contact with powered chain-saw"
}
] |
=== ICD-11 CODES FOUND ===
|
[] |
2F9Z
|
Neoplasms of unknown behaviour of unspecified site
|
A 5-day-old male neonate was received in the nursery emergency of our hospital in a state of shock. The short history depicted that the neonate was a product of consanguineous marriage and was born in a private hospital through spontaneous vaginal delivery. The neonate had an ulcer over the vertex since birth. The ulcer was being managed with dressings. The neonate was going well when during change of dressing massive hemorrhage started. The medical staff of that hospital tightly packed the wound and referred the patient to our hospital for further management. On examination the patient had severe pallor, cold extremities, and impalpable pulses. Fortunately the blood was arranged in the meanwhile; the patient was transfused and then taken directly to the operation theatre. At operation the defect was identified on the vertex as ACC involving scalp, part of skull bone and meninges. It was found that the ulcer had eroded the superior sagittal sinus thus causing tremendous hemorrhage. The superior sagittal sinus was repaired by the neurosurgeon which resulted in hemostasis. The defect was covered with a transposition flap and donor area was covered with split thickness skin graft . Postoperative recovery was uneventful. The patient was started orally on the same day of the operation. At follow up both the flap and graft were healthy; the thigh wound was also healing. The patient is doing well now.
| 3.466797
| 0.989258
|
sec[1]/p[0]
|
en
| 0.999996
|
26023367
|
N/A
|
[
"neonate",
"that",
"ulcer",
"vertex",
"well",
"hemorrhage",
"wound",
"defect",
"sagittal",
"sinus"
] |
[
{
"code": "5A00.00",
"title": "Permanent congenital hypothyroidism with diffuse goitre"
},
{
"code": "KB45",
"title": "Neonatal hypertension"
},
{
"code": "KB2D",
"title": "Respiratory failure of newborn"
},
{
"code": "KB89.1",
"title": "Short bowel syndrome in neonate"
},
{
"code": "KC5Y",
"title": "Other specified postnatal iatrogenic skin injury"
},
{
"code": "8A80.Z",
"title": "Migraine, unspecified"
},
{
"code": "QA76",
"title": "Medication or substance that is known to be an allergen without injury or harm"
},
{
"code": "PL13.6",
"title": "Medication or substance that is known to be an allergen, as mode of injury or harm"
},
{
"code": "9C40.A0",
"title": "Papilloedema"
},
{
"code": "PA6Z",
"title": "Unintentional fall from unspecified height"
}
] |
=== ICD-11 CODES FOUND ===
|
[] |
5A00.00
|
Permanent congenital hypothyroidism with diffuse goitre
|
We present the case of a 23-year-old female who was diagnosed with GSD type Ia shortly after birth. She was hospitalized for the first time at the age of 6 months for hypoglycaemia and metabolic acidosis that occurred during a respiratory infection. She had a doll-like facial appearance, hepatomegaly and kidney enlargement. She had been repeatedly hospitalized for episodes of enormous hypoglycaemia during infancy and adolescence. The patient was treated out of hospital with frequent meals rich in carbohydrates during the day and every 2–3 h during the night and as a result she developed obesity. Besides nutritional therapy, she was also treated with allopurinol for hyperuricaemia. She came to our clinic at the age of 22 years for the diagnostic evaluation of arterial hypertension and dyslipidaemia. Her weight was initially 80 kg, height was 157 cm and BMI was 32.5. The levels of uric acid were 425 nmol/l, LDL 3 mmol/l, HDL 0.8 mmol/l, cholesterol 5.2 mmol/l and TG 2 mmol/l. Her in-office blood pressure measurement repeatedly showed raised values (160/90 mmHg in average). We carried out a 24-h ambulatory blood pressure monitoring, which revealed normal daily and average 24-h values of both systolic and diastolic pressure, but also the non-dipping pattern of systolic pressure during the night . The latter can be attributed to excessive nocturnal eating and consequently disturbed sleeping pattern.
| 4.035156
| 0.97998
|
sec[1]/p[0]
|
en
| 0.999997
|
24683476
|
https://doi.org/10.1530/EDM-13-0056
|
[
"mmol",
"pressure",
"hospitalized",
"hypoglycaemia",
"repeatedly",
"treated",
"night",
"blood",
"average",
"systolic"
] |
[
{
"code": "GB42.1",
"title": "Albuminuria, Grade A3"
},
{
"code": "GB42.0",
"title": "Albuminuria, Grade A2"
},
{
"code": "MA18.0Y",
"title": "Other specified elevated blood glucose level"
},
{
"code": "EH90.Z",
"title": "Pressure ulcer of unspecified grade"
},
{
"code": "MB23.L",
"title": "Pressured speech"
},
{
"code": "MD30.Z",
"title": "Chest pain, unspecified"
},
{
"code": "CB22.Y",
"title": "Other specified diseases of mediastinum, not elsewhere classified"
},
{
"code": "BA2Z",
"title": "Hypotension, unspecified"
},
{
"code": "5A41",
"title": "Hypoglycaemia without associated diabetes"
},
{
"code": "5A4Z",
"title": "Disorders of glucose regulation or pancreatic internal secretion, unspecified"
}
] |
=== ICD-11 CODES FOUND ===
|
[] |
GB42.1
|
Albuminuria, Grade A3
|
In the presented case, the patient complained of postprandial abdominal pain after starting peritoneal dialysis. Intestinal TAO has been rarely reported. Kobayashi et al . reported a case of TAO with intestinal ischemia and reviewed the literature. They summarized 26 cases of visceral TAO including their case. The mean age of patients was 39.1, and all but two patients were male. The predominant symptom was abdominal pain, and 20 of 26 patients underwent digestive organ resection. The perioperative mortality rate was 30%, and only three patients underwent conservative treatment. The present patient experienced improvement in postprandial abdominal pain after avoiding dehydration and switching from peritoneal dialysis to hemodialysis. It is known that hemodialysis is more susceptible to intestinal ischemia than peritoneal dialysis because of its more unstable hemodynamics . The patient in this case underwent peritoneal dialysis for this reason; however, even though he was not dehydrated and his blood pressure was not low while receiving peritoneal dialysis, he experienced severe abdominal pain that got worse after starting peritoneal dialysis. His pain was relieved after stopping peritoneal dialysis. We presumed that hyperglycemia of the peritoneal cavity induced several changes, including leukostasis, vasoconstriction, and a pro-inflammatory state that caused aggravation of intestinal hypoxia .
| 4.109375
| 0.812988
|
sec[2]/p[3]
|
en
| 0.999997
|
26286269
|
https://doi.org/10.1186/s13256-015-0659-8
|
[
"peritoneal",
"dialysis",
"pain",
"abdominal",
"intestinal",
"patients",
"that",
"postprandial",
"starting",
"ischemia"
] |
[
{
"code": "DC5Z",
"title": "Diseases of peritoneum, unspecified"
},
{
"code": "DC50.Z",
"title": "Peritonitis, unspecified"
},
{
"code": "NB91.Y&XA0KZ0",
"title": "Peritoneal laceration"
},
{
"code": "2F94",
"title": "Neoplasms of unknown behaviour of peritoneum"
},
{
"code": "DC51.Y",
"title": "Other specified disorders of peritoneum or retroperitoneum"
},
{
"code": "QB94.Z",
"title": "Care involving dialysis, unspecified"
},
{
"code": "QB94.1",
"title": "Care involving extracorporeal dialysis"
},
{
"code": "9B73.0",
"title": "Retinal detachment with retinal break"
},
{
"code": "QB94.2",
"title": "Care involving peritoneal dialysis"
},
{
"code": "QB94.Y",
"title": "Care involving other specified dialysis"
}
] |
=== ICD-11 CODES FOUND ===
|
[] |
DC5Z
|
Diseases of peritoneum, unspecified
|
A 68-year-old Japanese woman underwent barium follow-through examination for the upper gastrointestinal tract as part of a routine medical screening. A pedunculated polyp was detected in the duodenum . The patient was referred to our hospital for further investigation and treatment of the polyp. The patient had been taking medication for hyperlipidemia but had no history of gastrointestinal diseases. A physical examination revealed no abnormalities, and there was no evidence of peripheral lymphadenopathy. Laboratory findings revealed an elevated level of total cholesterol to 254 mg/dL, but levels of carcinoembryonic antigen and carbohydrate antigen 19-9 and blood cell counts were within normal ranges. Esophagogastroduodenoscopy showed a pedunculated polyp in the duodenal second portion. The head of the polyp was approximately 20 mm in size (Figures 2(a) and 2(b) ). There were no erosions or ulcers in the polyp. The tumor had long stalk; therefore it easily migrated to the stomach ( Figure 2(c) ). Magnifying observation with narrowband imaging confirmed that the surface was entirely covered with intact duodenal mucosa, suggesting a subepithelial origin of the tumor ( Figure 2(d) ). Endoscopic ultrasonography visualized at least three cystic structures in the polyp head (Figures 2(e) and 2(f) ). Based on the macroscopic and ultrasonography features, Brunner's gland hamartoma was highly suspected.
| 3.949219
| 0.974121
|
sec[1]/p[0]
|
en
| 0.999996
|
27579190
|
https://doi.org/10.1155/2016/6707235
|
[
"polyp",
"gastrointestinal",
"pedunculated",
"antigen",
"duodenal",
"head",
"figures",
"tumor",
"ultrasonography",
"japanese"
] |
[
{
"code": "CA0J.Y",
"title": "Other specified nasal polyp"
},
{
"code": "DB71.Z",
"title": "Anal polyp, unspecified"
},
{
"code": "DA09.0",
"title": "Pulpitis"
},
{
"code": "DB71.1",
"title": "Lymphoid polyp"
},
{
"code": "GA16.Y",
"title": "Other specified acquired abnormalities of uterus, except cervix"
},
{
"code": "DE2Z",
"title": "Diseases of the digestive system, unspecified"
},
{
"code": "DB30.Y",
"title": "Other specified obstruction of large intestine"
},
{
"code": "DA60.Y",
"title": "Other specified gastric ulcer"
},
{
"code": "NB91.Y&XA9607",
"title": "Gastrointestinal laceration"
},
{
"code": "DD90.3",
"title": "Functional dyspepsia"
}
] |
=== ICD-11 CODES FOUND ===
|
[] |
CA0J.Y
|
Other specified nasal polyp
|
A 66-year-old male was admitted to our hospital with an episode of chest pain during atrial fibrillation with rapid ventricular response following a month history of progressive exertional chest pain. He had been treated 15 years previously with two overlapping Duet 3.5 × 23 mm and 3.5 × 13 mm BMS to his proximal and mid right coronary artery. Electrocardiography did not show ischemic changes at rest, but a significant rise in cardiac biomarkers was noted, consistent with a diagnosis of non-ST-elevation myocardial infarction. Coronary angiography demonstrated a critical stenosis within the prior stent in the mid-RCA ( Figure 1(a) ) that was occlusive to flow when crossed with a Dragonfly Duo OCT catheter. Focal predilation of the severe stenosis was performed using a Trek 3.0 × 12 mm balloon to improve distal vessel flow. Subsequent OCT confirmed severe in-stent restenosis with a percent area stenosis of 78% and minimal luminal area of 2.3 mm 2 . OCT also revealed the presence of neoatherosclerosis with fibrocalcific plaque, lipid pools, macrophage accumulation, and neoangiogenesis (Figures 1(b) – 1(d) ) throughout the majority of the stent's length. A site of old plaque rupture was visualized within the stented segment neoatheroma ( Figure 1(e) ) with no evidence of acute thrombus. An incidental finding of malapposed uncovered struts at the proximal stent segment was also seen ( Figure 1(f) ).
| 4.050781
| 0.973633
|
sec[1]/p[0]
|
en
| 0.999996
|
27034852
|
https://doi.org/10.1155/2016/1652065
|
[
"stent",
"stenosis",
"chest",
"pain",
"coronary",
"within",
"flow",
"area",
"plaque",
"segment"
] |
[
{
"code": "PK93.2",
"title": "Gastroenterology or urology devices associated with injury or harm, prosthetic or other implants, materials or accessory devices"
},
{
"code": "QB51.1",
"title": "Presence of urogenital implants"
},
{
"code": "QB51.Y",
"title": "Presence of other specified devices other than cardiac or vascular implants"
},
{
"code": "QB51.3",
"title": "Presence of otological or audiological implants"
},
{
"code": "PL12.3",
"title": "Obstruction of device, as mode of injury or harm"
},
{
"code": "DB30.Y",
"title": "Other specified obstruction of large intestine"
},
{
"code": "BB60.Z",
"title": "Mitral valve stenosis, unspecified"
},
{
"code": "BD52.2",
"title": "Stricture of artery"
},
{
"code": "FA82",
"title": "Spinal stenosis"
},
{
"code": "DA40.0",
"title": "Gastric outlet obstruction"
}
] |
=== ICD-11 CODES FOUND ===
|
[] |
PK93.2
|
Gastroenterology or urology devices associated with injury or harm, prosthetic or other implants, materials or accessory devices
|
The patient is a 35-year-old man, who is a chronic snorer with a BMI of 32.4 kg/m 2 who is not sleepy (Epworth Sleepiness Scale = 1/24). The patient's wife is a very light sleeper and is highly sensitive to snoring sounds. Over the past 14 years, the patient's weight has gone as high as 129.5 kg (BMI = 39.7 kg/m 2 ) and as low as 88.2 kg (BMI = 27.1 kg/m 2 ). The patient is known to have heroic snoring confirmed by his spouse for several years, when his BMI is above 100 kg (BMI = 30.7 kg/m 2 ); however, at weights below 100 kg, there is no snoring noted. The patient's current weight has been relatively unchanged (±1 kg) for the past year. History and physical exam demonstrate that the patient has no nasal septal deviation and no inferior turbinate hypertrophy (grade 2 bilaterally ) and has not had any complaints of nasal obstruction (NOSE questionnaire score = 10/100). After reviewing the literature for the variety of published modalities to abate snoring, the patient selected to utilize a previously unpublished technique for snoring, which is the use of a standard nasopharyngeal airway tube. This modality was selected since the patient has no nasal obstruction, septal deviation, or turbinate hypertrophy; therefore, the anatomy should be conducive to placement and use of NPAT. The sizes of soft polyvinyl chloride nasopharyngeal airway tubes evaluated included 24, 26, 28, 30, and 36 French (Fr).
| 3.865234
| 0.978516
|
sec[1]/p[1]
|
en
| 0.999998
|
27795710
|
https://doi.org/10.1155/2016/3628716
|
[
"snoring",
"nasal",
"past",
"weight",
"septal",
"deviation",
"turbinate",
"hypertrophy",
"obstruction",
"selected"
] |
[
{
"code": "MD11.Y",
"title": "Other specified abnormalities of breathing"
},
{
"code": "MA82.2",
"title": "Nasality"
},
{
"code": "CA0Z",
"title": "Upper respiratory tract disorders, unspecified"
},
{
"code": "CA0Y",
"title": "Other specified upper respiratory tract disorders"
},
{
"code": "LA70.2",
"title": "Choanal atresia"
},
{
"code": "NA00.3&XJ1C6",
"title": "Haematoma of nose"
},
{
"code": "PL13.8",
"title": "Expired or deteriorated medication or substance, as mode of injury or harm"
},
{
"code": "BA50",
"title": "Old myocardial infarction"
},
{
"code": "8B25.4",
"title": "Late effects of stroke not known if ischaemic or haemorrhagic"
},
{
"code": "QE51.1",
"title": "History of spouse or partner violence"
}
] |
=== ICD-11 CODES FOUND ===
|
[] |
MD11.Y
|
Other specified abnormalities of breathing
|
A clinical examination revealed a first to second degree multinodular goiter, which was mobile, soft and not painful on palpation. There was no palpable lymphadenopathy in the neck. Thyroid function tests showed a hypothyroidism state: Increased thyrotropic-stimulating hormone [TSH; 6.12 μIU/ml; normal range (NR), 0.35–5.5 μIU/ml] and antithyroglobulin antibody levels (27.8 IU/ml; NR, ≤4.11 IU/ml); decreased free triiodothyronine (2.96 pmol/l; NR, 3.0–6.51 pmol/l) and triiodothyronine (0.9 nmol/l; NR, 0.92–2.79 nmol/l) concentrations; normal thyroxine and free thyroxine, thyroglobulin and thyroid-binding globulin levels; and a negative result for antithyroperoxidase antibody. Biochemical analyses only showed elevated C-reactive protein levels (31.44 mg/l; NR, ≤8 mg/l). Thyroid B-mode ultrasonographyrevealed a number of hypoechoic nodules in the bilateral lobe of the thyroid gland, with sizes varying between 5 and 11 mm. However, one of these nodules (7–8 mm in size) located near the top of the left thyroid lobe was a heterogeneous hypoechoic lesion, and infiltrative margins with microcalcifications were identified by ultrasonography . B-mode ultrasonography showed a malignant nodule and cervical lymphadenectasis. Computed tomography revealed that the cervical lymphonodule was enlarged on the left side, at levels III and IV . The chest X-ray was normal and other extrapulmonary TBs were excluded.
| 4.042969
| 0.969238
|
sec[1]/p[1]
|
en
| 0.999997
|
24765176
|
https://doi.org/10.3892/ol.2014.1901
|
[
"thyroid",
"antibody",
"free",
"triiodothyronine",
"pmol",
"nmol",
"thyroxine",
"mode",
"hypoechoic",
"nodules"
] |
[
{
"code": "5A03.Z",
"title": "Thyroiditis, unspecified"
},
{
"code": "5A0Z",
"title": "Disorders of the thyroid gland or thyroid hormones system, unspecified"
},
{
"code": "5A03.Y",
"title": "Other specified thyroiditis"
},
{
"code": "5A00.2Z",
"title": "Acquired hypothyroidism, unspecified"
},
{
"code": "5A03.0",
"title": "Acute thyroiditis"
},
{
"code": "JA86.Y",
"title": "Maternal care for other specified fetal problems"
},
{
"code": "MA14.14",
"title": "Anti-nuclear antibody positive"
},
{
"code": "MA14.13",
"title": "Anti-nuclear antibody negative"
},
{
"code": "JA86.0",
"title": "Maternal care for red cell antibodies"
},
{
"code": "MA14.1C",
"title": "Raised antibody titre"
}
] |
=== ICD-11 CODES FOUND ===
|
[] |
5A03.Z
|
Thyroiditis, unspecified
|
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